Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Madhuri Ramanathan"'
Autor:
Diego C Reino, Vadim Pisarenko, David Palange, Danielle Doucet, Robert P Bonitz, Qi Lu, Iriana Colorado, Sharvil U Sheth, Benjamin Chandler, Kolenkode B Kannan, Madhuri Ramanathan, Da Zhong Xu, Edwin A Deitch, Rena Feinman
Publikováno v:
PLoS ONE, Vol 6, Iss 8, p e14829 (2011)
BackgroundInjurious non-microbial factors released from the stressed gut during shocked states contribute to the development of acute lung injury (ALI) and multiple organ dysfunction syndrome (MODS). Since Toll-like receptors (TLR) act as sensors of
Externí odkaz:
https://doaj.org/article/14c935ccf42d4031b16d9051dee8602b
Autor:
Qi Lu, Iriana Colorado, Marta Bosch-Marce, Kolenkode B. Kannan, Madhuri Ramanathan, Sharvil U. Sheth, Rena Feinman, Francis J. Caputo, Edwin A. Deitch, Chirag D. Badami, Gregg L. Semenza, Billy Abungu, Anthony C. Watkins, Danielle Doucet, Da Zhong Xu, Dimitrios Barlos, Shirhan Attan
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 299:G833-G843
Acute lung injury (ALI) and the development of the multiple organ dysfunction syndrome (MODS) are major causes of death in trauma patients. Gut inflammation and loss of gut barrier function as a consequence of splanchnic ischemia-reperfusion (I/R) ha
Autor:
Dmitry Lukashev, Samuel Leibovich, Madhuri Ramanathan, Wenting Luo, Michail V. Sitkovsky, Balázs Csóka, György Haskó
Publikováno v:
Journal of Leukocyte Biology. 86:681-689
Up-regulation of adenosine A2A receptors (A2ARs) and the HIF-1αl. 1 isoform plays an important role in the switch of macrophages from an inflammatory (M1) to an angiogenic (M2-like) phenotype. Adenosine A2AR and TLR agonists synergize to induce an
Autor:
Geert-Jan Boons, György Haskó, Madhuri Ramanathan, Grace Pinhal-Enfield, S. Joseph Leibovich, Stefanie N. Vogel, Andrew L. Salzman
Publikováno v:
The American Journal of Pathology. 163:711-721
Adenosine A(2A) receptor (A(2A)R) agonists synergize with Escherichia coli (E. coli) LPS [toll-like receptor (TLR)4 agonist] to up-regulate vascular endothelial growth factor (VEGF) expression in murine macrophages. Here, we demonstrate that TLR2, TL
Publikováno v:
Journal of Neuroscience Methods. 96:19-23
Synaptic terminals are sites of high metabolic activity and thus are particularly vulnerable to oxidative stress. Oxidative damage to proteins can be toxic to neurons and may cause irreversible cell damage and neurodegeneration. A neuroprotective mec
Autor:
Benjamin Chandler, Qi Lu, Sharvil U. Sheth, Iriana Colorado, Da Zhong Xu, Madhuri Ramanathan, Kolenkode B. Kannan, Edwin A. Deitch, Vadim Pisarenko, David Palange, Diego Reino, Robert P. Bonitz, Rena Feinman, Danielle Doucet
Publikováno v:
PLoS ONE, Vol 6, Iss 8, p e14829 (2011)
PLoS ONE
PLoS ONE
BackgroundInjurious non-microbial factors released from the stressed gut during shocked states contribute to the development of acute lung injury (ALI) and multiple organ dysfunction syndrome (MODS). Since Toll-like receptors (TLR) act as sensors of
Publikováno v:
Journal of clinical microbiology. 48(7)
HIV-1 viral load testing is essential to the management of HIV-1-infected patients, and proper specimen handling ensures accurate viral load (VL) results. This study was performed to (i) evaluate the effect of freezing plasma in situ in BD Vacutainer
Macrophages are an important source of vascular endothelial growth factor (VEGF). Adenosine A2Areceptor (A2AR) agonists with Toll-like receptor (TLR) 2, 4, 7, and 9 agonists synergistically induce macrophage VEGF expression. We show here using VEGF p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::766754a32f3f349db95c78583b057fb4
https://europepmc.org/articles/PMC1751314/
https://europepmc.org/articles/PMC1751314/
Publikováno v:
Scopus-Elsevier
Vascular endothelial growth factor (VEGF) expression in murine peritoneal macrophages is strongly upregulated by hypoxia via transcriptional and posttranscriptional mechanisms. Interferon-γ (IFN-γ) with Escherichia coll lipopolysaccharide (LPS) als
Autor:
Grace Pinhal-Enfield, Michael A. Schwarzschild, Anthony Rosania, Samuel Leibovich, Genie Elson, Madhuri Ramanathan, Jiang-Fan Chen, Bruce N. Cronstein, Marlene A. Jacobson, Carmen Montesinos, Paula C. Belem, J. Stephen Fink
Publikováno v:
The American journal of pathology. 160(6)
Under normoxic conditions, macrophages from C57BL mice produce low levels of vascular endothelial growth factor (VEGF). Hypoxia stimulates VEGF expression by approximately 500%; interferon-gamma (IFN-gamma) with endotoxin [lipopolysaccharide (LPS)] a