Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Madhumathy Jeyaraman"'
Publikováno v:
Frontiers in Pharmacology, Vol 4 (2013)
The membrane and channel protein Connexin-43 (Cx43), as well as the cytokine transforming growth factor (TGF) beta, suppress proliferative growth in cardiomyocytes and other cell types. Previously we showed that the inhibitory effect of Cx43 is cance
Externí odkaz:
https://doaj.org/article/4a1110eabe4448feac595de783f252f8
Autor:
Barbara E. Nickel, Wattamon Srisakuldee, Stéphane Tanguy, Janna Makazan, David C. Spray, Xitong Dang, Madhumathy Jeyaraman, Dumitru A. Iacobas, Elissavet Kardami
Publikováno v:
Progress in Biophysics and Molecular Biology
Progress in Biophysics and Molecular Biology, Elsevier, 2007, 94 (1-2), pp.245-64. ⟨10.1016/j.pbiomolbio.2007.03.009⟩
Progress in Biophysics and Molecular Biology, Elsevier, 2007, 94 (1-2), pp.245-64. ⟨10.1016/j.pbiomolbio.2007.03.009⟩
International audience; The purpose of this paper is to provide a brief overview of current thinking on the role of connexins, in particular Cx43, in growth regulation, and a more detailed discussion as to potential mechanisms involved with an emphas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7cdf80dee50764904b1f384c529e2494
https://doi.org/10.1016/j.pbiomolbio.2007.03.009
https://doi.org/10.1016/j.pbiomolbio.2007.03.009
Autor:
Elissavet Kardami, Zhi-Sheng Jiang, Robert R. Fandrich, Madhumathy Jeyaraman, Ian M.C. Dixon, Peter A. Cattini, Ge-Bo Wen
Publikováno v:
Journal of molecular and cellular cardiology. 42(1)
The heart expresses high and low molecular weight (hmw, lmw) fibroblast growth factor 2 (FGF-2) isoforms. While the injury-repair-related activities of lmw-FGF-2 have been studied extensively, those of hmw-FGF-2 have not. Thus, we investigated the ef
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::30c510726e9c52350e9efbd43299b64a
https://pubmed.ncbi.nlm.nih.gov/17045289
https://pubmed.ncbi.nlm.nih.gov/17045289
Publikováno v:
Molecular and cellular biochemistry. 289(1-2)
The ability of the gap junction phosphoprotein connexin-43 (Cx43) to inhibit DNA synthesis in primary cardiomyocytes is regulated by serine (S) 262, a protein kinase C phosphorylation site that also affects metabolic coupling. We have now examined if
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e97b94dda65ef1151963564fc7bfc20
https://pubmed.ncbi.nlm.nih.gov/16718370
https://pubmed.ncbi.nlm.nih.gov/16718370
Autor:
Stéphane Tanguy, Zhi-Sheng Jiang, Bradley W. Doble, Madhumathy Jeyaraman, Robert R. Fandrich, Elissavet Kardami
Publikováno v:
Pathophysiology of Cardiovascular Disease ISBN: 9781461350842
The gap junction protein connexin-43 (Cx43) exists mainly in the phosphory-lated state in the normal heart. We have investigated short-term effects of ischemia on cardiac Cx43 phosphorylation and distribution, in four models: global ischemia of the e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::84df4607e9dc69b93466be919d79e616
https://doi.org/10.1007/978-1-4615-0453-5_19
https://doi.org/10.1007/978-1-4615-0453-5_19
Autor:
Madhumathy, Jeyaraman, Stéphane, Tanguy, Robert R, Fandrich, Anton, Lukas, Elissavet, Kardami
Publikováno v:
Molecular and cellular biochemistry. 242(1-2)
The gap junction protein connexin-43 (Cx43) exists mainly in the phosphorylated state in the normal heart, while ischemia induces dephosphorylation. Phosphatase(s) involved in cardiac Cx43 dephosphorylation have not as yet been identified. We examine
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d4a0c2ea46bb067dc0dcec3068e16a46
https://pubmed.ncbi.nlm.nih.gov/12619875
https://pubmed.ncbi.nlm.nih.gov/12619875
Publikováno v:
Cardiac Cell Biology ISBN: 9781441953247
The gap junction protein connexin-43 (Cx43) exists mainly in the phosphorylated state in the normal heart, while ischemia induces dephosphorylation. Phosphatase(s) involved in cardiac Cx43 dephosphorylation have not as yet been identified. We examine
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::546e0d9f8d7648143fad27056aaad042
https://doi.org/10.1007/978-1-4757-4712-6_17
https://doi.org/10.1007/978-1-4757-4712-6_17
Publikováno v:
Journal of Molecular and Cellular Cardiology. 33:A53
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::29186980ae2a2319e9387909de1d276e
https://doi.org/10.1016/s0022-2828(01)90208-1
https://doi.org/10.1016/s0022-2828(01)90208-1