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Mitochondrial Dysfunction as a Potential Mechanism Mediating Cardiac Comorbidities in Parkinson's Disease.

Autor: Salis Torres, Agustina, Lee, Ji-Eun, Caporali, Andrea, Semple, Robert K., Horrocks, Mathew H., MacRae, Vicky E.

Publikováno v: International Journal of Molecular Sciences; Oct2024, Vol. 25 Issue 20, p10973, 30p

Hdac9 inhibits medial artery calcification through down-regulation of Osterix

Autor: He, Pengcheng, Yu, Hongjiao, Jiang, Lei, Chen, Ziying, Wang, Siying, Macrae, Vicky E., Fu, Xiaodong, Zhu, Dongxing

Publikováno v: In Vascular Pharmacology September 2020 132

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TGF-β induced PI3K/AKT/mTOR pathway controls myofibroblast differentiation and secretory phenotype of valvular interstitial cells through the modulation of cellular senescence in a naturally occurring in vitro canine model of myxomatous mitral valve disease

Autor: Tang, Paul, Markby, Greg, MacNair, Andrew J, Tang, Keyi, Tkacz, Michal, Parys, Maciej, Phadwal, Kanchan, MacRae, Vicky, Corcoran, Brendan

Publikováno v: Tang, P, Markby, G, MacNair, A J, Tang, K, Tkacz, M, Parys, M, Phadwal, K, MacRae, V & Corcoran, B 2023, ' TGF-β induced PI3K/AKT/mTOR pathway controls myofibroblast differentiation and secretory phenotype of valvular interstitial cells through the modulation of cellular senescence in a naturally occurring in vitro canine model of myxomatous mitral valve disease ', Cell proliferation, vol. 56, no. 6, e13435, pp. 1-23 . https://doi.org/10.1111/cpr.13435

PI3K/AKT/mTOR signalling contributes to several cardiovascular disorders. The aim of this study was to examine the PI3K/AKT/mTOR pathway in myxomatous mitral valve disease (MMVD). Double-immunofluorescence examined expression of PI3K and TGF-β1 in c

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______3094::289030044cd2e14bb9ce7bd81f344c14
https://hdl.handle.net/20.500.11820/10547549-51a1-453a-9430-e667835400c9