Zobrazeno 1 - 10
of 50
pro vyhledávání: '"Mabel Buelna-Chontal"'
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 10 (2022)
Externí odkaz:
https://doaj.org/article/f063a16f20fb4bdb82664b93c265d2d3
Publikováno v:
Antioxidants, Vol 11, Iss 3, p 507 (2022)
The transcription factor Nrf2 is a master regulator of multiple cytoprotective genes that maintain redox homeostasis and exert anti-inflammatory functions. The Nrf2-Keap1 signaling pathway is a paramount target of many cardioprotective strategies, be
Externí odkaz:
https://doaj.org/article/ba2d3156f6df474eba78f69f28506ff1
Autor:
Edmundo Chávez, Mabel Buelna-Chontal, Arturo Macías-López, Luz Hernández-Esquivel, Francisco Correa, Natalia Pavón
Publikováno v:
Biochemistry Research International, Vol 2020 (2020)
Mitochondrial permeability transition is characterized by the opening of a transmembranal pore that switches membrane permeability from specific to nonspecific. This structure allows the free traffic of ions, metabolites, and water across the mitocho
Externí odkaz:
https://doaj.org/article/83bd155652804f31ab5769c6d808e572
Mitochondrial Quality Control in Cardiac-Conditioning Strategies against Ischemia-Reperfusion Injury
Publikováno v:
Life, Vol 11, Iss 11, p 1123 (2021)
Mitochondria are the central target of ischemic preconditioning and postconditioning cardioprotective strategies, which consist of either the application of brief intermittent ischemia/reperfusion (I/R) cycles or the administration of pharmacological
Externí odkaz:
https://doaj.org/article/dbb21420bd6d4696aa2fe7b1a70bb9fe
Autor:
Mabel Buelna-Chontal, Wylly R. García-Niño, Alejandro Silva-Palacios, Cristina Enríquez-Cortina, Cecilia Zazueta
Publikováno v:
Antioxidants, Vol 10, Iss 5, p 749 (2021)
Post-translational modifications based on redox reactions “switch on-off” the biological activity of different downstream targets, modifying a myriad of processes and providing an efficient mechanism for signaling regulation in physiological and
Externí odkaz:
https://doaj.org/article/dcdc720a462741dfba6f138d069026dd
Publikováno v:
International Journal of Molecular Sciences, Vol 20, Iss 15, p 3657 (2019)
Obesity is considered to significantly increase the risk of the development of a vast range of metabolic diseases. However, adipogenesis is a complex physiological process, necessary to sequester lipids effectively to avoid lipotoxicity in other tiss
Externí odkaz:
https://doaj.org/article/ff6ddfa873e84f89a093fe5669fb97db
Mitochondrial Quality Control in Cardiac-Conditioning Strategies against Ischemia-Reperfusion Injury
Publikováno v:
Life, Vol 11, Iss 1123, p 1123 (2021)
Life
Life
Mitochondria are the central target of ischemic preconditioning and postconditioning cardioprotective strategies, which consist of either the application of brief intermittent ischemia/reperfusion (I/R) cycles or the administration of pharmacological
Autor:
Julio Sandoval, Horacio Osorio-Alonso, Rosa Luz de la Fuente-León, Fausto Sánchez, Ivan Rubio-Gayosso, Mabel Buelna-Chontal, José L. Sánchez-Gloria, Fausto Sánchez-Muñoz, Adrián Henández-Díazcouder, Roxana Carbó
Publikováno v:
Life sciences. 287
Background Cold temperatures can aggravate pulmonary diseases and promote pulmonary arterial hypertension (PAH); however, the underlying mechanism has not been fully explored. Aim To explore the effect of chronic cold exposure on the production of in
Autor:
Jonatan Barrera-Chimal, Cesar Cortés-González, Isabel Amador-Martínez, Felipe Massó, Mabel Buelna-Chontal, Frederic Jaisser, Johannes García-Ballhaus
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental BiologyREFERENCES. 35(8)
Uremic cardiomyopathy is a common complication in chronic kidney disease (CKD) patients, accounting for a high mortality rate. Several mechanisms have been proposed to link CKD and cardiac alterations; however, the early cardiac modifications that oc
Autor:
Edmundo Chávez, Mabel Buelna-Chontal, Francisco Correa, Luz Hernández-Esquivel, Cristina Uribe-Alvarez, Arturo Macías-López, Natalia Pavón
Publikováno v:
Biochemistry and Cell Biology. 97:187-192
In the kidney, the accumulation of heavy metals such as Cd2+produces mitochondrial dysfunctions, i.e., uncoupling of the oxidative phosphorylation, inhibition of the electron transport through the respiratory chain, and collapse of the transmembrane