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Autor:
Frances V. Fuller-Pace, Samer Abdallah, Pierre Roux, Christelle Anguille, Marie P. Khoury, Alexandra Diot, Nikola Arsic, Valerie Meuray, Colin A. Purdie, Philip R. Quinlan, Judit Remenyi, St�phanie Vinot, Marion de Toledo, S�bastien M. Joruiz, Ma�lys Cren, Gilles Gadea, Lee B. Jordan, Kenneth Fernandes, Alastair M. Thompson, Jean-Christophe Bourdon
Publikováno v:
eLife
eLife, eLife Sciences Publication, 2016, 5, 〈10.7554/eLife.14734〉
eLife, Vol 5 (2016)
eLife, eLife Sciences Publication, 2016, 5, pp.e14734. ⟨10.7554/eLife.14734⟩
eLife, 2016, 5, pp.e14734. ⟨10.7554/eLife.14734⟩
eLife, eLife Sciences Publication, 2016, 5, 〈10.7554/eLife.14734〉
eLife, Vol 5 (2016)
eLife, eLife Sciences Publication, 2016, 5, pp.e14734. ⟨10.7554/eLife.14734⟩
eLife, 2016, 5, pp.e14734. ⟨10.7554/eLife.14734⟩
TP53 is conventionally thought to prevent cancer formation and progression to metastasis, while mutant TP53 has transforming activities. However, in the clinic, TP53 mutation status does not accurately predict cancer progression. Here we report, base
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd6e5e3665203f26a5977193bbf1fdd8
http://hal.univ-reunion.fr/hal-01703040
http://hal.univ-reunion.fr/hal-01703040