Zobrazeno 1 - 10
of 24
pro vyhledávání: '"MICHELLE P. MARASSI"'
Autor:
Daniel C. Trindade, Raquel C. Trindade, Michelle P. Marassi, Ornélia P.P.R. Martins, Ricardo H. Costa-e-Sousa, Elisabete C. Mattos, Alcides Marinho-Jr., Luís C. Reis, Emerson L. Olivares
Publikováno v:
Anais da Academia Brasileira de Ciências, Vol 79, Iss 2, Pp 250-259 (2007)
We investigated the morphologic and functional changes of infarcted rat hearts under a paradigm of angiotensinconverting enzyme inhibition. Myocardial infarction was induced by left coronary artery ligation and a control group (SHAM) underwent sham-o
Externí odkaz:
https://doaj.org/article/cd83df8d0d484385bc2456359f3a07c6
Autor:
DANILO LUSTRINO, ALBA C.M. SILVA, IRACEMA G. ARAUJO, VICTOR M. TUNHOLI, VINÍCIUS M. TUNHOLI-ALVES, ROSANE N. CASTRO, DENISE P. CARVALHO, JAIRO PINHEIRO, MICHELLE P. MARASSI
Publikováno v:
Anais da Academia Brasileira de Ciências, Iss 0
ABSTRACT The objective of this study was to identify thyroid hormones and to examine their putative site of synthesis in Achatina fulica snails. For this purpose, radioimmunoassays were performed for T3 and T4 before and after long starvation with or
Externí odkaz:
https://doaj.org/article/4cc29aafa0324aeaaf1f24764eae8b0b
Autor:
Fernando A C Seara, Leonardo Maciel, Raiana A Q Barbosa, Nayana C Rodrigues, Anderson L B Silveira, Michelle P Marassi, Adriana B Carvalho, José Hamilton M Nascimento, Emerson L Olivares
Publikováno v:
PLoS ONE, Vol 13, Iss 1, p e0190355 (2018)
Thyroid dysfunctions can increase the risk of myocardial ischemia and infarction. However, the repercussions on cardiac ischemia/reperfusion (IR) injury remain unclear so far. We report here the effects of hypothyroidism and thyrotoxicosis in the sus
Externí odkaz:
https://doaj.org/article/707fb4df842e438a816a3add076f6948
Autor:
Vinícius Menezes Tunholi-Alves, Alba C.M. Silva, Jairo Pinheiro, Denise P. Carvalho, Rosane Nora Castro, Danilo Lustrino, Victor Menezes Tunholi, Iracema G. Araujo, Michelle P. Marassi
Publikováno v:
Anais da Academia Brasileira de Ciências v.89 n.3 suppl.0 2017
Anais da Academia Brasileira de Ciências
Academia Brasileira de Ciências (ABC)
instacron:ABC
Anais da Academia Brasileira de Ciências, Iss 0
Anais da Academia Brasileira de Ciências, Volume: 89, Issue: 3 Supplement, Pages: 2181-2188, Published: 24 JUL 2017
Anais da Academia Brasileira de Ciências
Academia Brasileira de Ciências (ABC)
instacron:ABC
Anais da Academia Brasileira de Ciências, Iss 0
Anais da Academia Brasileira de Ciências, Volume: 89, Issue: 3 Supplement, Pages: 2181-2188, Published: 24 JUL 2017
The objective of this study was to identify thyroid hormones and to examine their putative site of synthesis in Achatina fulica snails. For this purpose, radioimmunoassays were performed for T3 and T4 before and after long starvation with or without
Autor:
Michelle P. Marassi, Luís Carlos Reis, Emerson L. Olivares, André S. Mecawi, Alba C.M. Silva, G.E. Império, Iracema G. Araujo, Fernando A.C. Seara
Publikováno v:
Brazilian Journal of Medical and Biological Research
Brazilian Journal of Medical and Biological Research v.52 n.10 2019
Associação Brasileira de Divulgação Científica (ABDC)
instacron:ABDC
Brazilian Journal of Medical and Biological Research v.52 n.10 2019
Associação Brasileira de Divulgação Científica (ABDC)
instacron:ABDC
Considering the recognized role of thyroid hormones on the cardiovascular system during health and disease, we hypothesized that type 2 deiodinase (D2) activity, the main activation pathway of thyroxine (T4)-to-triiodothyronine (T3), could be an impo
Autor:
Iracema G. Araujo, Luís Carlos Reis, Emerson L. Olivares, Alba C.M. Silva, Fernando A.C. Seara, André S. Mecawi, Michelle P. Marassi, Guinever E. Imperio
Considering the recognized role of thyroid hormones on the cardiovascular system during health and disease, we hypothesized that type 2 deiodinase (D2) activity, the main activation pathway of thyroxine (T4)-to-triiodothyronine (T3), could be an impo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::688e2a81617c792bdcd9e940be862ffe
https://doi.org/10.1101/353052
https://doi.org/10.1101/353052
Autor:
Manuela França de Souza Miranda, Anderson Luiz Bezerra da Silveira, André S. Mecawi, Emerson L. Olivares, Michelle P. Marassi, Roberto Laureano Melo, Alba C.M. Silva, José Antunes-Rodrigues
Publikováno v:
Experimental Physiology. 99:868-880
Based on the relevance of the renin-angiotensin system and the ongoing controversy regarding the role of the sympathetic nervous system in thyroid hormone-induced cardiac hypertrophy, the aim of the present study was to establish whether the putative
Autor:
Nayana Coutinho Rodrigues, Michelle P. Marassi, Wellington da Silva Côrtes, Luís Carlos Reis, André S. Mecawi, Rafael Sonoda-Côrtes, Fábio Fagundes da Rocha, Claudio Silva-Almeida, Iracema G. Araujo, Fernanda Marques Pestana, Emerson L. Olivares
Publikováno v:
Neuropharmacology. 62:446-456
Although serotonergic system has been classically implicated in mood modulation, there has been relatively little study on the relationship between this system and thyroid hormones (TH) economy in stress models. When TH are studied, the effects of st
Autor:
Denise P. Carvalho, Alba C. M. Da Silva, Michelle P. Marassi, Mônica Mühlbauer, Andrea Claudia Freitas Ferreira, Alexandre Lopes Lourenço
Publikováno v:
Journal of Endocrinology. 205:271-277
The sodium–iodide symporter (NIS) mediates iodide uptake into the thyrocytes, which is important for the diagnosis and therapy of thyroid disorders. Decreased ability to uptake iodide in thyroid carcinomas reduces the efficacy of radioiodine therap
Autor:
Adriana Bastos Carvalho, Leonardo Maciel, Raiana A. Q. Barbosa, José Nascimento, Anderson Luiz Bezerra da Silveira, Emerson L. Olivares, Nayana Coutinho Rodrigues, Fernando A.C. Seara, Michelle P. Marassi
Publikováno v:
PLoS ONE, Vol 13, Iss 1, p e0190355 (2018)
PLoS ONE
PLoS ONE
Aim Thyroid dysfunctions can increase the risk of myocardial ischemia and infarction. However, the repercussions on cardiac ischemia/reperfusion (IR) injury remain unclear so far. We report here the effects of hypothyroidism and thyrotoxicosis in the