Zobrazeno 1 - 10
of 18
pro vyhledávání: '"MESH: Retinoblastoma Protein"'
Autor:
Amira Zine El Abidine, Ikram Guizani, Samir Boubaker, Paola Massimi, Vjekoslav Tomaić, Lawrence Banks, Emna Ennaifer, Rahima Bel Haj Rhouma
Publikováno v:
Virology
Virology, Elsevier, 2017, 500, pp.218-225. ⟨10.1016/j.virol.2016.10.023⟩
Virology, Elsevier, 2017, 500, pp.218-225. ⟨10.1016/j.virol.2016.10.023⟩
International audience; Human Papillomavirus E6 and E7 play critical roles in cancer development, although not all isolates of the viral oncoproteins are identical. A common E7 variant encodes an amino acid change at N29S. We show that this change in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c1d8ea5745965979ea627a17d6d8726
https://hal.archives-ouvertes.fr/hal-01513648
https://hal.archives-ouvertes.fr/hal-01513648
Autor:
Michel Fontes, Cécile Mignon-Ravix, Carole Couillault, Anne Millet, Nathalie Pujol, Jonathan J. Ewbank, Carlos Cardoso
Publikováno v:
Developmental Biology
Developmental Biology, Elsevier, 2005, 278 (1), pp.49-59. ⟨10.1016/j.ydbio.2004.10.014⟩
Developmental Biology, 2005, 278 (1), pp.49-59. ⟨10.1016/j.ydbio.2004.10.014⟩
Developmental Biology, Elsevier, 2005, 278 (1), pp.49-59. ⟨10.1016/j.ydbio.2004.10.014⟩
Developmental Biology, 2005, 278 (1), pp.49-59. ⟨10.1016/j.ydbio.2004.10.014⟩
Mutations in the XNP/ATR-X gene cause several X-linked mental retardation syndromes in humans. The XNP/ATR-X gene encodes a DNA-helicase belonging to the SNF2 family. It has been proposed that XNP/ATR-X might be involved in chromatin remodelling. The
Autor:
Philippe Leboulch, Masayuki Yamamoto, Osamu Ohneda, Ritsuko Shimizu, Leila Maouche-Chretien, Sylvie Gisselbrecht, Zahra Kadri, Paul-Henri Romeo, Stany Chrétien
Publikováno v:
PLoS Biology
PLoS Biology, Public Library of Science, 2009, 7 (6), pp.e1000123. 〈10.1371/journal.pbio.1000123〉
PLoS Biology, Vol 7, Iss 6, p e1000123 (2009)
PLoS Biology, Public Library of Science, 2009, 7 (6), pp.e1000123. ⟨10.1371/journal.pbio.1000123⟩
PLoS Biology, 2009, 7 (6), pp.e1000123. ⟨10.1371/journal.pbio.1000123⟩
PLoS Biology, Public Library of Science, 2009, 7 (6), pp.e1000123. 〈10.1371/journal.pbio.1000123〉
PLoS Biology, Vol 7, Iss 6, p e1000123 (2009)
PLoS Biology, Public Library of Science, 2009, 7 (6), pp.e1000123. ⟨10.1371/journal.pbio.1000123⟩
PLoS Biology, 2009, 7 (6), pp.e1000123. ⟨10.1371/journal.pbio.1000123⟩
Cell differentiation is often coupled with cell cycle arrest. Here, we show that direct binding of the erythroid transcription factor GATA-1 to the retinoblastoma protein and the pRb/E2F transcription factor complex is critical for red blood cell for
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d3382409bf32982f51303968cb1fb922
http://www.hal.inserm.fr/inserm-00707647/document
http://www.hal.inserm.fr/inserm-00707647/document
Autor:
Stéphane Dalle, Lluis Fajas, Safia Costes, Emilie Blanchet, Said Assou, Jean-Sébastien Annicotte, Irena Iankova, Claude Sardet, Carine Chavey, Jacques Teyssier
Publikováno v:
Nature Cell Biology
Nature Cell Biology, Nature Publishing Group, 2009, 11 (8), pp.1017-1023. ⟨10.1038/ncb1915⟩
Nature Cell Biology, Nature Publishing Group, 2009, 11 (8), pp.1017-23. 〈10.1038/ncb1915〉
Nature Cell Biology, 2009, 11 (8), pp.1017-23. ⟨10.1038/ncb1915⟩
Nature Cell Biology, Nature Publishing Group, 2009, 11 (8), pp.1017-23. ⟨10.1038/ncb1915⟩
Nature Cell Biology, Nature Publishing Group, 2009, 11 (8), pp.1017-1023. ⟨10.1038/ncb1915⟩
Nature Cell Biology, Nature Publishing Group, 2009, 11 (8), pp.1017-23. 〈10.1038/ncb1915〉
Nature Cell Biology, 2009, 11 (8), pp.1017-23. ⟨10.1038/ncb1915⟩
Nature Cell Biology, Nature Publishing Group, 2009, 11 (8), pp.1017-23. ⟨10.1038/ncb1915⟩
International audience; CDK4-pRB-E2F1 cell-cycle regulators are robustly expressed in non-proliferating beta cells, suggesting that besides the control of beta-cell number the CDK4-pRB-E2F1 pathway has a role in beta-cell function. We show here that
Publikováno v:
Nature Cell Biology
Nature Cell Biology, Nature Publishing Group, 2012, 14 (3), pp.266-275. ⟨10.1038/ncb2443⟩
Nature Cell Biology, 2012, 14 (3), pp.266-275. ⟨10.1038/ncb2443⟩
Nature Cell Biology, Nature Publishing Group, 2012, 14 (3), pp.266-275. ⟨10.1038/ncb2443⟩
Nature Cell Biology, 2012, 14 (3), pp.266-275. ⟨10.1038/ncb2443⟩
International audience; Cellular senescence is a tumour-suppressor mechanism that is triggered by cancer-initiating or promoting events in mammalian cells. The molecular underpinnings for this stable arrest involve transcriptional repression of proli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::79e48cf5c16a63cc4b12e576a4f9b023
https://hal-pasteur.archives-ouvertes.fr/pasteur-03236097
https://hal-pasteur.archives-ouvertes.fr/pasteur-03236097
Autor:
Lars Zender, Mukesh Kumar, Satyanarayana Ande, Hüseyin Sirma, Julia M. Weise, Britta Witt, Christiane Guguen-Guillouzo, André Lechel, Jitendra K. Meena, Cagatay Günes, Vadim Sakk, Karl-Lenhard Rudolph
Publikováno v:
Gastroenterology
Gastroenterology, 2011, 141 (1), pp.326-37, 337.e1-3. ⟨10.1053/j.gastro.2011.03.047⟩
Gastroenterology, WB Saunders, 2011, 141 (1), pp.326-37, 337.e1-3. ⟨10.1053/j.gastro.2011.03.047⟩
Gastroenterology, 2011, 141 (1), pp.326-37, 337.e1-3. ⟨10.1053/j.gastro.2011.03.047⟩
Gastroenterology, WB Saunders, 2011, 141 (1), pp.326-37, 337.e1-3. ⟨10.1053/j.gastro.2011.03.047⟩
International audience; BACKGROUND & AIMS: Telomerase activity has not been detected in healthy human liver biopsy samples, but it is up-regulated in most human liver tumors. It is not clear whether telomerase is activated in response to acute or chr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::14191aab7bca96c9edf1dbb1ed55d49b
https://hdl.handle.net/10033/210669
https://hdl.handle.net/10033/210669
Autor:
Slavica Krantic, Vjekoslav Dulic
Publikováno v:
Cell Cycle
Cell Cycle, Taylor & Francis, 2010, 9 (16), pp.3150. ⟨10.4161/cc.9.16.12887⟩
Cell Cycle, Taylor & Francis, 2010, 9 (16), pp.3150. ⟨10.4161/cc.9.16.12887⟩
Comment on "S-phase lengthening induced by p16(INK4a) overexpression in malignant cells with wild-type pRb and p53." Chien WW, Domenech C, Catallo R, Salles G, Ffrench M. Cell Cycle. 2010 Aug 15;9(16):3286-96. doi: 10.4161/cc.9.16.12600. Epub 2010 Au
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ed6b078577ae113b5006e64ae7fc035
https://hal.archives-ouvertes.fr/hal-03233696/file/p16Ink4A_Dulic_Cell.pdf
https://hal.archives-ouvertes.fr/hal-03233696/file/p16Ink4A_Dulic_Cell.pdf
Autor:
Mario Noti, Dominic Leiser, Atanas G. Atanasov, Kristina Schoonjans, Nadia Corazza, Corinne Roesselet, Thomas Brunner
Publikováno v:
FASEB Journal
FASEB Journal, Federation of American Society of Experimental Biology, 2008, 22 (12), pp.4117-25. ⟨10.1096/fj.08-114157⟩
FASEB Journal, 2008, 22 (12), pp.4117-25. ⟨10.1096/fj.08-114157⟩
FASEB Journal, Federation of American Society of Experimental Biology, 2008, 22 (12), pp.4117-25. ⟨10.1096/fj.08-114157⟩
FASEB Journal, 2008, 22 (12), pp.4117-25. ⟨10.1096/fj.08-114157⟩
International audience; Glucocorticoids are anti-inflammatory steroids with important applications in the treatment of inflammatory diseases. Endogenous glucocorticoids are mainly produced by the adrenal glands, although there is increasing evidence
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::071f5992861bd5cbe6e4d26c383f2215
https://www.hal.inserm.fr/inserm-00350850
https://www.hal.inserm.fr/inserm-00350850
Publikováno v:
Molecular Cancer Research
Molecular Cancer Research, American Association for Cancer Research, 2008, 6 (3), pp.418-25. ⟨10.1158/1541-7786.MCR-07-0381⟩
Molecular Cancer Research, American Association for Cancer Research, 2008, 6 (3), pp.418-25. ⟨10.1158/1541-7786.MCR-07-0381⟩
Permanent silencing of E2F-dependent genes is a hallmark of the irreversible cell cycle exit that characterizes terminally differentiated and senescent cells. The determinant of this silencing during senescence has been proposed to be the binding of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a85a99ec396c1715720cf3ddd794d44a
https://hal.archives-ouvertes.fr/hal-00317872
https://hal.archives-ouvertes.fr/hal-00317872
Autor:
Kristell Bastide, Jean-François Bernaudin, Christophe Joubert, Bernard Malfoy, Sylvie Chevillard, Bruno Lectard, Marie-Noëlle Guilly, Céline Levalois
Publikováno v:
Lung Cancer
Lung Cancer, 2009, 63 (3), pp.348-53. ⟨10.1016/j.lungcan.2008.06.007⟩
Lung Cancer, Elsevier, 2009, 63 (3), pp.348-53. ⟨10.1016/j.lungcan.2008.06.007⟩
Lung Cancer, 2009, 63 (3), pp.348-53. ⟨10.1016/j.lungcan.2008.06.007⟩
Lung Cancer, Elsevier, 2009, 63 (3), pp.348-53. ⟨10.1016/j.lungcan.2008.06.007⟩
International audience; Inhalation of radon is closely associated with an increased risk of lung cancers. While the involvement of Ink4a in lung tumor development has been widely described, the tumor suppressor gene has not been studied in radon-indu