Zobrazeno 1 - 5
of 5
pro vyhledávání: '"MESH: Receptors, Glucagon"'
Autor:
Pierre Cattan, Julien Castel, Rémy Burcelin, Aurélie Waget, Daniel J. Drucker, Mattieu Armanet, Melis Karaca, Christophe Magnan, Jens J. Holst, Céline Garret, Thierry Sulpice, Gaëlle Payros, Myriam Masseboeuf, Adriano Maida, Cendrine Cabou
Publikováno v:
Endocrinology
Endocrinology, Endocrine Society, 2011, 152 (8), pp.3018-29. 〈10.1210/en.2011-0286〉
Endocrinology, 2011, 152 (8), pp.3018-29. ⟨10.1210/en.2011-0286⟩
Endocrinology, Endocrine Society, 2011, 152 (8), pp.3018-29. ⟨10.1210/en.2011-0286⟩
Endocrinology, Endocrine Society, 2011, 152 (8), pp.3018-29. 〈10.1210/en.2011-0286〉
Endocrinology, 2011, 152 (8), pp.3018-29. ⟨10.1210/en.2011-0286⟩
Endocrinology, Endocrine Society, 2011, 152 (8), pp.3018-29. ⟨10.1210/en.2011-0286⟩
International audience; Inhibition of dipeptidyl peptidase-4 (DPP-4) activity improves glucose homeostasis through a mode of action related to the stabilization of the active forms of DPP-4-sensitive hormones such as the incretins that enhance glucos
Publikováno v:
FEBS Letters
FEBS Letters, Wiley, 2006, 580 (24), pp.5697-704. ⟨10.1016/j.febslet.2006.09.021⟩
FEBS Letters, 2006, 580 (24), pp.5697-704. ⟨10.1016/j.febslet.2006.09.021⟩
FEBS Letters, Wiley, 2006, 580 (24), pp.5697-704. 〈10.1016/j.febslet.2006.09.021〉
FEBS Letters, Wiley, 2006, 580 (24), pp.5697-704. ⟨10.1016/j.febslet.2006.09.021⟩
FEBS Letters, 2006, 580 (24), pp.5697-704. ⟨10.1016/j.febslet.2006.09.021⟩
FEBS Letters, Wiley, 2006, 580 (24), pp.5697-704. 〈10.1016/j.febslet.2006.09.021〉
To assess glucagon receptor compartmentalization and signal transduction in liver parenchyma, we have studied the functional relationship between glucagon receptor endocytosis, phosphorylation and coupling to the adenylate cyclase system. Following a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fadde96ea9bd99d4f1bfa6d69df9344c
https://www.hal.inserm.fr/inserm-00175470
https://www.hal.inserm.fr/inserm-00175470
Publikováno v:
Cellular Signalling
Cellular Signalling, Elsevier, 2011, 23 (3), pp.522-8. ⟨10.1016/j.cellsig.2010.09.014⟩
Cellular Signalling, Elsevier, 2011, 23 (3), pp.522-8. ⟨10.1016/j.cellsig.2010.09.014⟩
Defective insulin secretion is a feature of type 2 diabetes that results from inadequate compensatory increase in β-cell mass, decreased β-cell survival and impaired glucose-dependent insulin release. Pancreatic β-cell proliferation, survival and
Autor:
Daniel J. Drucker, Rémy Burcelin, Miguel A. Iglesias, Dong Hoon Kim, Aurélie Waget, Patrice D. Cani, Sophie Rastrelli, André Colom, Claude Knauf, Nathalie M. Delzenne, Randy J. Seeley, Chantal Chabo
Publikováno v:
Diabetes
Diabetes, American Diabetes Association, 2008, 57 (10), pp.2603-12. ⟨10.2337/db07-1788⟩
Diabetes, Vol. 57, no. 10, p. 2603-2612 (2008)
Diabetes, 2008, 57 (10), pp.2603-12. ⟨10.2337/db07-1788⟩
Diabetes, American Diabetes Association, 2008, 57 (10), pp.2603-12. ⟨10.2337/db07-1788⟩
Diabetes, Vol. 57, no. 10, p. 2603-2612 (2008)
Diabetes, 2008, 57 (10), pp.2603-12. ⟨10.2337/db07-1788⟩
OBJECTIVE—Ingested glucose is detected by specialized sensors in the enteric/hepatoportal vein, which send neural signals to the brain, which in turn regulates key peripheral tissues. Hence, impairment in the control of enteric-neural glucose sensi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::496c5fb67a3a03624c467d1aed041663
https://www.hal.inserm.fr/inserm-00409097
https://www.hal.inserm.fr/inserm-00409097
Autor:
Corinne Leloup, Cendrine Cabou, Rémy Burcelin, Nicolas Marsollier, Luc Pénicaud, Gérard Campistron, Daniel J. Drucker, Christophe Magnan, Céline Cruciani-Guglielmacci
Publikováno v:
Diabetes
Diabetes, American Diabetes Association, 2008, 57 (10), pp.2577-87. ⟨10.2337/db08-0121⟩
Diabetes, 2008, 57 (10), pp.2577-87. ⟨10.2337/db08-0121⟩
Diabetes, American Diabetes Association, 2008, 57 (10), pp.2577-87. ⟨10.2337/db08-0121⟩
Diabetes, 2008, 57 (10), pp.2577-87. ⟨10.2337/db08-0121⟩
OBJECTIVE— To ascertain the importance and mechanisms underlying the role of brain glucagon-like peptide (GLP)-1 in the control of metabolic and cardiovascular function. GLP-1 is a gut hormone secreted in response to oral glucose absorption that re
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::220714480e0936bb59aa0aeb72dc8102
https://www.hal.inserm.fr/inserm-00408890
https://www.hal.inserm.fr/inserm-00408890