Cynomolgus macaque IL37 polymorphism and control of SIV infection

Autor: Henri-Jean Garchon, Nicolas Tchitchek, Delphine Desjardins, Roger Le Grand, Bruno Vaslin, Takashi Shiina, Nicolas Congy-Jolivet, Nathalie Dereuddre-Bosquet, Brigitte Autran, Ioannis Theodorou, Olivier Lambotte, Antoine Blancher, Alice Aarnink, Shingo Suzuki

Publikováno v: Scientific Reports, Vol 9, Iss 1, Pp 1-15 (2019)
Scientific Reports
Scientific Reports, Nature Publishing Group, 2019, 9 (1), ⟨10.1038/s41598-019-44235-x⟩
Scientific Reports, Nature Publishing Group, 2019, 9 (1), pp.7981. ⟨10.1038/s41598-019-44235-x⟩
Scientific Reports, 2019, 9 (1), pp.7981. ⟨10.1038/s41598-019-44235-x⟩

The association between gene polymorphisms and plasma virus load at the set point (SP-PVL) was investigated in Mauritian macaques inoculated with SIV. Among 44 macaques inoculated with 50 AID50, six individuals were selected: three with SP-PVL among

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7f10cd764ad05ff00d00a2e63ad53fe4
http://link.springer.com/article/10.1038/s41598-019-44235-x

Microglial Priming as Trained Immunity in the Brain

Autor: David Brough, Jessica Quintin, Michael J. Haley, Stuart M. Allan

Publikováno v: Haley, M, Brough, D, Quintin, J & Allan, S 2018, ' Microglial priming as trained immunity in the brain ', Neuroscience . https://doi.org/10.1016/j.neuroscience.2017.12.039
Neuroscience
Neuroscience, 2019, 405, pp.47-54. ⟨10.1016/j.neuroscience.2017.12.039⟩
Neuroscience, Elsevier-International Brain Research Organization, 2019, 405, pp.47-54. ⟨10.1016/j.neuroscience.2017.12.039⟩

International audience; In this review we discuss the possibility that the phenomenon of microglial priming can be explained by the mechanisms that underlie trained immunity. The latter involves the enhancement of inflammatory responses by epigenetic

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::41305ec16a2e389b365efae9eb25fbd2
https://doi.org/10.1016/j.neuroscience.2017.12.039

Selective predisposition to bacterial infections in IRAK-4–deficient children: IRAK-4–dependent TLRs are otherwise redundant in protective immunity

Autor: Sami Al-Hajjar, Carlos Rodríguez-Gallego, John I. Gallin, Toshiro Hara, Maya Chrabieh, Andrew C. Issekutz, Huey Hsuan Chang, Horst von Bernuth, Joanne Smart, Helen Chapel, Eric Vivier, László Maródi, David P. Speert, Mimi L.K. Tang, Jean-Laurent Casanova, Capucine Picard, Xiaoxia Li, Steven M. Holland, Shen-Ying Zhang, Franck J. Barrat, Stephan Ehl, Abdulaziz Al-Ghonaium, Ben Zion Garty, Chaim M. Roifman, Xavier Bossuyt, Douglas R. McDonald, Anne Puel, Hidetoshi Takada, Ofer Levy, Damien Sanlaville, Laurent Abel, Frederic Geissmann, Richard L. Miller, Cheng-Lung Ku, Kun Yang, Noorbibi K. Day-Good, Coleen K. Cunningham

Publikováno v: The Journal of Experimental Medecine
The Journal of Experimental Medecine, The Rockefeller University Press, 2007, 204 (10), pp.2407-22. ⟨10.1084/jem.20070628⟩
The Journal of Experimental Medecine, The Rockefeller University Press, 2007, 204 (10), pp.2407-22. 〈10.1084/jem.20070628〉
Journal of Experimental Medicine
Journal of Experimental Medicine, Rockefeller University Press, 2007, 204 (10), pp.2407-22. ⟨10.1084/jem.20070628⟩
Journal of Experimental Medicine, 2007, 204 (10), pp.2407-22. ⟨10.1084/jem.20070628⟩
The Journal of Experimental Medicine
Journal of Experimental Medicine, 204(10), 2407-2422. ROCKEFELLER UNIV PRESS

Human interleukin ( IL) 1 receptor - associated kinase 4 ( IRAK- 4) deficiency is a recently discovered primary immunodefi ciency that impairs Toll/ IL- 1R immunity, except for the Toll- like receptor ( TLR) 3 - and TLR4 - interferon ( IFN)-alpha/bet

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7099915ba0c10ec616712048d096eeb1
https://doi.org/10.1084/jem.20070628

Interleukin-33 overexpression is associated with liver fibrosis in mice and humans

Autor: Annie L'Helgoualc'h, Mariette Lisbonne, Hugues Gascan, Michel Rauch, Katia Bourd-Boittin, Nathalie Théret, Bruno Turlin, Claire Piquet-Pellorce, Michel Samson, Pierrick Marvie, Laurence Preisser

Publikováno v: Journal of Cellular and Molecular Medicine
Journal of Cellular and Molecular Medicine, Wiley Open Access, 2010, 14 (6B), pp.1726-39. ⟨10.1111/j.1582-4934.2009.00801.x⟩
Journal of Cellular and Molecular Medicine, 2010, 14 (6B), pp.1726-39. ⟨10.1111/j.1582-4934.2009.00801.x⟩

International audience; Interleukin-33 (IL-33), the most recently identified member of the IL-1 family, induces synthesis of T Helper 2 (Th2)-type cytokines via its heterodimeric ST2/IL-1RAcP receptor. Th2-type cytokines play an important role in fib

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9086df8539c7c19aa7e9779d113e42dd
https://doi.org/10.1111/j.1582-4934.2009.00801.x

The Inflammatory Response in Acyl-CoA Oxidase 1 Deficiency (Pseudoneonatal Adrenoleukodystrophy)

Autor: Pierre Andreoletti, Stéphane Mandard, Aurore Vluggens, Gérard Lizard, Sander Kersten, Janardan K. Reddy, H. I. El Hajj, Kévin Ragot, Ronald J.A. Wanders, H. R. Waterham, Mustapha Cherkaoui-Malki

Publikováno v: Endocrinology, 153(6), 2568-2575. The Endocrine Society
Endocrinology
Endocrinology, Endocrine Society, 2012, 153 (6), pp.2568-75. 〈10.1210/en.2012-1137〉
Endocrinology, Endocrine Society, 2012, 153 (6), pp.2568-75. ⟨10.1210/en.2012-1137⟩
Endocrinology, 153(6), 2568-2575
Endocrinology 153 (2012) 6

Among several peroxisomal neurodegenerative disorders, the pseudoneonatal adrenoleukodystrophy (P-NALD) is characterized by the acyl-coenzyme A oxidase 1 (ACOX1) deficiency, which leads to the accumulation of very-long-chain fatty acids ( VLCFA) and

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b25c8f0010ebbc23f73a7fe631931ebf
https://pure.amc.nl/en/publications/the-inflammatory-response-in-acylcoa-oxidase-1-deficiency-pseudoneonatal-adrenoleukodystrophy(7bd25523-b244-427f-ad1d-aaed82029c73).html