Zobrazeno 1 - 3
of 3
pro vyhledávání: '"MESH: Cytosol/metabolism"'
Publikováno v:
Cell Host and Microbe
Cell Host and Microbe, 2014, 15 (6), pp.792--798. ⟨10.1016/j.chom.2014.05.003⟩
Cell Host and Microbe, Elsevier, 2014, 15 (6), pp.792--798. ⟨10.1016/j.chom.2014.05.003⟩
Cell Host and Microbe, 2014, 15 (6), pp.792--798. ⟨10.1016/j.chom.2014.05.003⟩
Cell Host and Microbe, Elsevier, 2014, 15 (6), pp.792--798. ⟨10.1016/j.chom.2014.05.003⟩
Comment in : Nod-like receptors have a grip on stem cells. [Cell Host Microbe. 2014]; International audience; The intestinal crypt is a site of potential interactions between microbiota products, stem cells, and other cell types found in this niche,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d4bcec5076405c467b8a1349f9b45056
https://doi.org/10.1016/j.chom.2014.05.003
https://doi.org/10.1016/j.chom.2014.05.003
Autor:
Patrick England, Julien Barbier, Sylvain Pichard, Anne Araye, Javier Pérez, Alexandre Chenal, Bruno Baron, Amélie Goudet, Daniel Gillet, Sophie Zinn-Justin
Publikováno v:
PLoS ONE
PLoS ONE, 2016, 11 (4), pp.e0153401. ⟨10.1371/journal.pone.0153401⟩
PLoS ONE, Vol 11, Iss 4, p e0153401 (2016)
PLoS ONE, Public Library of Science, 2016, 11 (4), pp.e0153401. ⟨10.1371/journal.pone.0153401⟩
PLoS ONE, 2016, 11 (4), pp.e0153401. ⟨10.1371/journal.pone.0153401⟩
PLoS ONE, Vol 11, Iss 4, p e0153401 (2016)
PLoS ONE, Public Library of Science, 2016, 11 (4), pp.e0153401. ⟨10.1371/journal.pone.0153401⟩
International audience; Botulinum neurotoxin A (BoNT/A) is composed of three domains: a catalytic domain (LC), a translocation domain (H N) and a receptor-binding domain (H C). Like most bacterial toxins BoNT/A is an amphitropic protein, produced in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::54fbe07a82d025638b54118c5f0f0e50
https://hal-pasteur.archives-ouvertes.fr/pasteur-01378747
https://hal-pasteur.archives-ouvertes.fr/pasteur-01378747
Critical role of cytosolic phospholipase A2 in bronchial mucus hypersecretion in CFTR-deficient mice
Autor:
Lhousseine Touqui, Aarbiou J, Bob J. Scholte, Yongzheng Wu, Y. Kita, Pierre-Régis Burgel, Borot F, Michel Chignard, Ollero M, Clémence Martin, Fariel Dif, Ignacio Garcia-Verdugo, D. Israel-Biet, Dimitri Leduc
Publikováno v:
European Respiratory Journal
European Respiratory Journal, 2010, 36 (5), pp.1120-1130. ⟨10.1183/09031936.00183409⟩
European Respiratory Journal, 36(5), 1120-1130. European Respiratory Society
European Respiratory Journal, European Respiratory Society, 2010, 36 (5), pp.1120-1130. ⟨10.1183/09031936.00183409⟩
European Respiratory Journal, 2010, 36 (5), pp.1120-1130. ⟨10.1183/09031936.00183409⟩
European Respiratory Journal, 36(5), 1120-1130. European Respiratory Society
European Respiratory Journal, European Respiratory Society, 2010, 36 (5), pp.1120-1130. ⟨10.1183/09031936.00183409⟩
Cystic fibrosis (CF) is due to mutations in the CF transmembrane conductance regulator gene CFTR. CF is characterised by mucus dehydration, chronic bacterial infection and inflammation, and increased levels of cytosolic phospholipase A2 alpha (cPLA2
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b7ca3cac07312cba69eba555c3cbf17d
https://hal-pasteur.archives-ouvertes.fr/pasteur-02863044
https://hal-pasteur.archives-ouvertes.fr/pasteur-02863044