Zobrazeno 1 - 5
of 5
pro vyhledávání: '"MESH: Acid Anhydride Hydrolases"'
Autor:
Olivier Hermine, Vahid Asnafi, Izabela Bialuk, Marcia Bellon, Lee Ratner, Ambroise Marçais, Michael N. Petrus, Thomas A. Waldmann, Genoveffa Franchini, Toshiki Watanabe, Antoine Gessain, Masao Matsuoka, Christophe Nicot, Achiléa L. Bittencourt, Lourdes Farre, Veronica Galli, Xue Tao Bai
Publikováno v:
Dipòsit Digital de la UB
Universidad de Barcelona
Molecular Cancer
Molecular Cancer, 2021, 20 (1), pp.86. ⟨10.1186/s12943-021-01370-2⟩
Molecular Cancer, Vol 20, Iss 1, Pp 1-15 (2021)
Universidad de Barcelona
Molecular Cancer
Molecular Cancer, 2021, 20 (1), pp.86. ⟨10.1186/s12943-021-01370-2⟩
Molecular Cancer, Vol 20, Iss 1, Pp 1-15 (2021)
Background Human T cell Leukemia virus type 1 (HTLV-I) is etiologically linked to adult T cell leukemia/lymphoma (ATL) and an inflammatory neurodegenerative disease called HTLV-I-associated myelopathy or tropical spastic paraparesis (HAM/TSP). The ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6510393e031c514bb0b9c0e9b58acbdb
http://hdl.handle.net/2445/178671
http://hdl.handle.net/2445/178671
Autor:
Bruno Canard, Etienne Decroly
Publikováno v:
Current Opinion in Virology
Current Opinion in Virology, Elsevier, 2017, 24, pp.87-96. ⟨10.1016/j.coviro.2017.04.003⟩
Current Opinion in Virology, Elsevier, 2017, 24, pp.87-96. 〈10.1016/j.coviro.2017.04.003〉
Current Opinion in Virology, 2017, 24, pp.87-96. ⟨10.1016/j.coviro.2017.04.003⟩
Current Opinion in Virology, Elsevier, 2017, 24, pp.87-96. ⟨10.1016/j.coviro.2017.04.003⟩
Current Opinion in Virology, Elsevier, 2017, 24, pp.87-96. 〈10.1016/j.coviro.2017.04.003〉
Current Opinion in Virology, 2017, 24, pp.87-96. ⟨10.1016/j.coviro.2017.04.003⟩
Highlights • Many viruses cap their mRNAs with their own enzymes. • The latter have significantly different structures and mechanisms from cellular capping enzymes. • Unique active-site architecture and mechanisms should expedite inhibitor desi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a3072f39beb524ee33705cee046aafb7
https://pubmed.ncbi.nlm.nih.gov/28527860
https://pubmed.ncbi.nlm.nih.gov/28527860
Autor:
Yanick J. Crow, Catherine Morrissey, Leo A. H. Zeef, Emma M. Jenkinson, Gillian I. Rice, Valérie Briolat, Jean-Pierre Levraud, David Gent, Paul R. Kasher
Publikováno v:
Journal of Immunology
Journal of Immunology, Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists, 2015, 194 (6), pp.2819-2825. ⟨10.4049/jimmunol.1403157⟩
Journal of Immunology, 2015, 194 (6), pp.2819-2825. ⟨10.4049/jimmunol.1403157⟩
Journal of Immunology, Publisher : Baltimore : Williams & Wilkins, c1950-. Latest Publisher : Bethesda, MD : American Association of Immunologists, 2015, 194 (6), pp.2819-2825. ⟨10.4049/jimmunol.1403157⟩
Journal of Immunology, 2015, 194 (6), pp.2819-2825. ⟨10.4049/jimmunol.1403157⟩
In humans, loss of function mutations in the SAMHD1 (AGS5) gene cause a severe form of Aicardi-Goutières syndrome (AGS), an inherited inflammatory-mediated encephalopathy characterized by increased type I IFN activity and upregulation of IFN-stimula
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13597b059fe027c73b6fffeb22c26201
https://hal-pasteur.archives-ouvertes.fr/pasteur-02558343
https://hal-pasteur.archives-ouvertes.fr/pasteur-02558343
Autor:
Stéphane Koundrioukoff, Bernard Malfoy, Gaël A. Millot, R. Scott Hansen, Olivier Brison, Nicolas Vogt, Michelle Debatisse, Anne Letessier, Anne Marie Lachagès
Publikováno v:
Nature
Nature, Nature Publishing Group, 2011, 470 (7332), pp.120-123. ⟨10.1038/nature09745⟩
Nature, Nature Publishing Group, 2011, 470 (7332), pp.120-123. ⟨10.1038/nature09745⟩
Some chromosomal locations, known as common fragile sites, are predisposed to breakage. These sites have pathological relevance because they are often associated with chromosomal translocations. An analysis of the replication dynamics along a 1.6-Mb
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d2789e0443093b0c0f66251609257708
https://pubmed.ncbi.nlm.nih.gov/21293366
https://pubmed.ncbi.nlm.nih.gov/21293366
Autor:
Carla, Verri, Luca, Roz, Davide, Conte, Triantafillos, Liloglou, Anna, Livio, Aurelien, Vesin, Alessandra, Fabbri, Francesca, Andriani, Christian, Brambilla, Luca, Tavecchio, Giuseppe, Calarco, Elisa, Calabrò, Andrea, Mancini, Diego, Tosi, Paolo, Bossi, John K, Field, Elisabeth, Brambilla, Gabriella, Sozzi
Publikováno v:
American Journal of Respiratory and Critical Care Medicine
American Journal of Respiratory and Critical Care Medicine, American Thoracic Society, 2009, 179 (5), pp.396-401. ⟨10.1164/rccm.200807-1153OC⟩
Dadun. Depósito Académico Digital de la Universidad de Navarra
instname
American Journal of Respiratory and Critical Care Medicine, American Thoracic Society, 2009, 179 (5), pp.396-401. ⟨10.1164/rccm.200807-1153OC⟩
Dadun. Depósito Académico Digital de la Universidad de Navarra
instname
Rationale: Fragile histidine triad (FHIT) is a tumor suppressor gene involved in the pathogenesis of lung cancer. Objectives: The purpose of this study was to investigate the different molecular alterations leading to the inactivation of FHIT gene fu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f036564830bc7ef9c0095eafdb892f6
https://www.hal.inserm.fr/inserm-00359752
https://www.hal.inserm.fr/inserm-00359752