A PET-Surrogate Signature for the Interrogation of the Metabolic Status of Breast Cancers.

Autor: Confalonieri S; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Matoskova B; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Pennisi R; Department of Oncology, University of Torino Medical School, Candiolo, Turin, 10060, Italy.; Candiolo Cancer Institute, FPO-IRCCS, Str. Provinciale 142 km 3.95, Candiolo, Turin, 10060, Italy., Martino F; Department of Oncology, University of Torino Medical School, Candiolo, Turin, 10060, Italy.; Candiolo Cancer Institute, FPO-IRCCS, Str. Provinciale 142 km 3.95, Candiolo, Turin, 10060, Italy., De Mario A; Department of Biomedical Sciences, University of Padua, Via U. Bassi 58/B, Padua, 35131, Italy., Miloro G; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Montani F; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Rotta L; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Ferrari ME; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Gilardi L; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Ceci F; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy.; Department of Oncology and Haemato-Oncology, University of Milan, Milan, 20142, Italy., Grana CM; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy., Rizzuto R; Department of Biomedical Sciences, University of Padua, Via U. Bassi 58/B, Padua, 35131, Italy., Mammucari C; Department of Biomedical Sciences, University of Padua, Via U. Bassi 58/B, Padua, 35131, Italy., Di Fiore PP; IEO, European Institute of Oncology IRCCS, Via Ripamonti 435, Milan, 20141, Italy.; Department of Oncology and Haemato-Oncology, University of Milan, Milan, 20142, Italy., Lanzetti L; Department of Oncology, University of Torino Medical School, Candiolo, Turin, 10060, Italy.; Candiolo Cancer Institute, FPO-IRCCS, Str. Provinciale 142 km 3.95, Candiolo, Turin, 10060, Italy.

Publikováno v: Advanced science (Weinheim, Baden-Wurttemberg, Germany) [Adv Sci (Weinh)] 2024 Jul; Vol. 11 (28), pp. e2308255. Date of Electronic Publication: 2024 May 17.

UBPY: a growth regulated human ubiquitin isopeptidase

Autor: NAVIGLIO, Silvio, MATTEUCCI C, MATOSKOVA B, NAGASE T, NOMURA N, DI FIORE P. P. AND DRAETTA G. F.

The ubiquitin pathway has been implicated in the regulation of the abundance of proteins that control cell growth and proliferation. We have identified and characterized a novel human ubiquitin isopeptidase, UBPY, which both as a recombinant protein

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______3977::b366dae2f10826a54a4bf3e403c3f615
http://hdl.handle.net/11591/192053

Targeted analysis of Ubiquitin-Specific Peptidase (USP8) in a population of Iranian people with Cushing's disease and a systematic review of the literature.

Autor: Hashemi-Madani, Nahid¹, Cheraghi, Sara¹, Emami, Zahra¹, Mehrjardi, Ali Zare², Kaynama, Mahmoud Reza³, Khamseh, Mohammad E.¹ khamseh.m@iums.ac.ir

Publikováno v: BMC Endocrine Disorders. 6/11/2024, Vol. 24 Issue 1, p1-12. 12p.

Activation of RET as a dominant transforming gene by germline mutations of MEN2A and MEN2B

Autor: Santoro, M., Carlomagno, F., Romano, A., Bottaro, Dp P., Dathan, Na A., Grieco, M., Fusco, A., Vecchio, G., Matoskova, B., Kraus, Mh H., Di Fiore, P. P., Difiore, Pp, Et, A.

Multiple endocrine neoplasia types 2A and 2B (MEN2A and MEN2B) and familial medullary thyroid carcinoma are dominantly inherited cancer syndromes. All three syndromes are associated with mutations in RET, which encodes a receptor-like tyrosine kinase

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=od______2659::3a4763b8637d995fcbbebc531db77066
https://zenodo.org/record/894824

Ret-mediated mitogenesis requires Src kinase activity

Autor: Melillo, R. M., Barone, M. V., Lupoli, G., Cirafici, A. M., Carlomagno, F., Visconti, R., Matoskova, B., Di Fiore, P. P., Vecchio, G., Alfredo Fusco, Santoro, M.

Publikováno v: Scopus-Elsevier
Cancer research (Chic. Ill.) 59 (1999): 1120–1126.
info:cnr-pdr/source/autori:Melillo RM; Barone MV; Lupoli G; Cirafici AM; Carlomagno F; Visconti R; Matoskova B; Di Fiore PP; Vecchio G; Fusco A; Santoro M/titolo:Ret-mediated mitogenesis requires Src kinase activity/doi:/rivista:Cancer research (Chic. Ill.)/anno:1999/pagina_da:1120/pagina_a:1126/intervallo_pagine:1120–1126/volume:59
CIÊNCIAVITAE

The proto-oncogene RET encodes a transmembrane growth neurotrophic receptor with tyrosine kinase (TK) activity. RET mutations are associated with several human neoplastic and nonneoplastic diseases, including thyroid papillary carcinoma, multiple end

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::536396d0512d46d1e6d9862468ec43ce
http://www.scopus.com/inward/record.url?eid=2-s2.0-0033104338&partnerID=MN8TOARS