Zobrazeno 1 - 10
of 13
pro vyhledávání: '"MARINA, MARTINA"'
Autor:
MARINA, MARTINA, SARAGIH, GOLDA RANI, KUSTIATI, ULAYATUL, BUDIPITOJO, TEGUH, WIJAYANTO, HERY, PANGESTININGSIH, TRI WAHYU, ARIANA, WENDO, WORO DANUR, BUDIARIATI, VISTA, KUSINDARTA, DWI LILIEK
Publikováno v:
Biodiversitas: Journal of Biological Diversity; May2024, Vol. 25 Issue 5, p2121-2129, 9p
Publikováno v:
Neurology International; Sep2022, Vol. 14 Issue 3, p561-573, 13p
Autor:
Finnern, Marina Martina
The dissertation investigated the differentiation of subsyndromes in a spectrum from regional to widespread chronic musculoskeletal pain on the basis of mechanism-related somatosensory and clinical phenotypes within the framework of the multidimensio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2c43fee383396ef1f581e64e10ddce04
http://archiv.ub.uni-heidelberg.de/volltextserver/28198/
http://archiv.ub.uni-heidelberg.de/volltextserver/28198/
Autor:
Diego Bonetti, Michela Clerici, Savani Anbalagan, Marina Martina, Giovanna Lucchini, Maria Pia Longhese
Publikováno v:
PLoS Genetics, Vol 6, Iss 5, p e1000966 (2010)
Eukaryotic cells distinguish their chromosome ends from accidental DNA double-strand breaks (DSBs) by packaging them into protective structures called telomeres that prevent DNA repair/recombination activities. Here we investigate the role of key tel
Externí odkaz:
https://doaj.org/article/d0124054b0434487909e343d30b0dbea
Autor:
Nicola Manfrini, Daniele Cesena, Maxime Wery, Antonin Morillon, Marina Martina, Maria Pia Longhese, Fabrizio d'Adda di Fagagna, Marc Descrimes, Camilla Trovesi
Publikováno v:
EMBO reports
EMBO Reports
EMBO Reports, EMBO Press, 2014, 16 (2), pp.221-231. ⟨10.15252/embr.201439458⟩
EMBO Reports, 2014, 16 (2), pp.221-231. ⟨10.15252/embr.201439458⟩
EMBO Reports
EMBO Reports, EMBO Press, 2014, 16 (2), pp.221-231. ⟨10.15252/embr.201439458⟩
EMBO Reports, 2014, 16 (2), pp.221-231. ⟨10.15252/embr.201439458⟩
Eukaryotic cells respond to DNA double-strand breaks (DSBs) by activating a checkpoint that depends on the protein kinases Tel1/ATM and Mec1/ATR. Mec1/ATR is activated by RPA-coated single-stranded DNA (ssDNA), which arises upon nucleolytic degradati
Diverse roles in DNA metabolism have been envisaged for budding yeast and mammalian Rif1. In particular, yeast Rif1 is involved in telomere homeostasis, while its mammalian counterpart participates in the cellular response to DNA double-strand breaks
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be4929c52e93c961d307250e997c6659
http://hdl.handle.net/10281/80585
http://hdl.handle.net/10281/80585
Publikováno v:
Chromosoma.
Telomeres are specialized nucleoprotein complexes that provide protection to the ends of eukaryotic chromosomes. Telomeric DNA consists of tandemly repeated G-rich sequences that terminate with a 3′ single-stranded overhang, which is important for
Autor:
Marina Martina, Michela Clerici, Giovanna Lucchini, Veronica Baldo, Maria Pia Longhese, Diego Bonetti
Publikováno v:
Molecular and cellular biology. 32(9)
Generation of G-strand overhangs at Saccharomyces cerevisiae yeast telomeres depends primarily on the MRX (Mre11-Rad50-Xrs2) complex, which is also necessary to maintain telomere length by recruiting the Tel1 kinase. MRX physically interacts with Rif
The ends of eukaryotic chromosomes need to be protected from detection as DNA double strand breaks by the DNA damage response pathways. Failure to do so would have devastating consequences for genome integrity. Packaging of chromosome ends into prote
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a8de72388ef008c486a3067eab48ca0d
http://hdl.handle.net/10281/28914
http://hdl.handle.net/10281/28914
Shelterin-like proteins and Yku inhibit nucleolytic processing of Saccharomyces cerevisiae telomeres
Autor:
Savani Anbalagan, Giovanna Lucchini, Marina Martina, Michela Clerici, Diego Bonetti, Maria Pia Longhese
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 6, Iss 5, p e1000966 (2010)
PLoS Genetics, Vol 6, Iss 5, p e1000966 (2010)
Eukaryotic cells distinguish their chromosome ends from accidental DNA double-strand breaks (DSBs) by packaging them into protective structures called telomeres that prevent DNA repair/recombination activities. Here we investigate the role of key tel