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Publikováno v:
British Journal of Biomedical Science. 72:201-203
(2015). Low serum creatine kinase activity in hospital patients. British Journal of Biomedical Science: Vol. 72, No. 4, pp. 201-203.
Autor:
M.A. Crook, N. Maghsoodi
Publikováno v:
Atherosclerosis Supplements. 28:e14-e15
Publikováno v:
Clinica Chimica Acta. 331:1-5
Background: Serum sialic acid (TSA) has been shown to be a cardiovascular risk factor and an acute phase reactant, with elevated concentrations associated with increased cardiovascular mortality and to precede the onset of type 2 diabetes. Aim: The p
Autor:
D. Evans, M.A. Crook
Publikováno v:
MRS Bulletin. 22:36-40
There have been many studies (see for instance Reference 1) directed at quantifying the effects of ionizing radiation on polymers. Mainly these studies have focused on assessing the degradation of mechanical properties. In addition to changes in mech
Publikováno v:
Diabetic Medicine. 13:238-242
Elevated serum sialic acid (SSA) predicts cardiovascular disease in the non-diabetic population and is also associated with the presence of microalbuminuria and clinical proteinuria in patients with insulin-dependent diabetes (IDDM). We have studied
Autor:
M.A. Crook, P.L. Walker
Publikováno v:
Clinica chimica acta; international journal of clinical chemistry. 418
The detection of lipaemia in a patient blood sample can be a clinical conundrum as well as an analytical nuisance. With a reported prevalence of 0.7% in all blood samples received for lipid studies its finding has been suggested to be an underappreci
Publikováno v:
European journal of internal medicine. 12(6)
Elevated serum total sialic acid (TSA) has been shown to be associated with increased cardiovascular mortality. It has been postulated that atherogenesis is a postprandial phenomenon. We tested the hypothesis that serum TSA and other acute phase prot
Autor:
M.A. Crook, C. Nelson-Piercy
Publikováno v:
Nutrition. 25:1098-1099
Autor:
N.J Miller, M.A Crook
Publikováno v:
Atherosclerosis. 131(1)
Hypobetalipoproteinaemia is a rare autosomal dominant condition, characterised by low serum cholesterol concentrations. The exact biochemical defect is not known although truncated forms of apolipoprotein B have been described [1]. Some of the compli