Zobrazeno 1 - 10
of 19
pro vyhledávání: '"M. T. Gillespie"'
Publikováno v:
Applied Biochemistry and Biotechnology. :835-843
BioTreat, a commercially available nutrient-surfactant compound, was investigated for its ability to solubilize TCE. Potential mechanisms for enhancing biodegradation rates by the use of nutrient-surfactant mixtures are: increased solubilization of T
Autor:
T J, Martin, E H, Allan, P W M, Ho, J H, Gooi, J M W, Quinn, M T, Gillespie, V, Krasnoperov, N A, Sims
Publikováno v:
Advances in experimental medicine and biology. 658
Members of the ephrin and Eph family are local mediators of cell function through largely contact-dependent processes in development and in maturity. Production of ephrinB2 mRNA and protein are increased by PTH and PTHrP in osteoblasts. Both a synthe
Autor:
F P L, Lai, M, Cole-Sinclair, W-J, Cheng, J M W, Quinn, M T, Gillespie, J W, Sentry, H-G, Schneider
Publikováno v:
British journal of haematology. 126(2)
Summary The ratio of osteoprotegerin [OPG, tumour necrosis factor receptor superfamily, member 11b (TNFRSF11B)] to receptor activator of nuclear factor kappaB ligand [RANKL, tumour necrosis factor (ligand) superfamily, member 11 (TNFSF11)] in bone is
Autor:
J. M. W. Quinn, M. C. Lecomte, S. L. Asplund, K. Sugita, T. J. Huang, T. Matsushita, C. Rahuel, M. Nees, A. Takagi, J. E Compston, H. Okada, M. T. Canciani, S. Bord, M. A. Scott, J. P. Cartron, C. Capoferri, R. E. Lewis, Y. Colin, S. Breit, T. Kojima, W. El Nemer, D. P. Kontoyiannis, A. Yoshioka, J. Takamatsu, E. Frith, E. Gauthier, E. Lee, M. E. Reid, M. Muckenthaler, M. Cole-Sinclair, J. W. Sentry, P. Gane, Y. Kroviarski, A. E. Kulozik, K. Yamamoto, G. Burgess, J. I. O. Craig, M. Pfoersich, M. T. Gillespie, T. Yamazaki, T. Murate, S. H. Kroft, M. Sugimoto, T. Adachi, C. Hagemeier, U. Schaefer, W.-J. Cheng, C. Le Van Kim, F. P. L. Lai, G. R. Halverson, D. C. Ireland, N. J. Karandikar, H.-G. Schneider, P. M. Mannucci, H. Saito, R. W. McKenna
Publikováno v:
British Journal of Haematology. 126
Publikováno v:
Traffic (Copenhagen, Denmark). 2(11)
Parathyroid hormone-related protein is responsible for hypercalcemia induced by various tumors. The similarity of its N-terminus to that of parathyroid hormone enables parathyroid hormone-related protein to share parathyroid hormone's signaling prope
Autor:
E, Romas, O, Bakharevski, D K, Hards, V, Kartsogiannis, J M, Quinn, P F, Ryan, T J, Martin, M T, Gillespie
Publikováno v:
Arthritis and rheumatism. 43(4)
To investigate the cellular mechanism of bone destruction in collagen-induced arthritis (CIA).After induction of CIA in DA rats, a histologic study of the advanced arthritic lesion was carried out on whole, decalcified joints from the hindpaws of aff
Autor:
N, Udagawa, N, Takahashi, E, Jimi, K, Matsuzaki, T, Tsurukai, K, Itoh, N, Nakagawa, H, Yasuda, M, Goto, E, Tsuda, K, Higashio, M T, Gillespie, T J, Martin, T, Suda
Publikováno v:
Bone. 25(5)
We previously reported that osteoblasts/stromal cells are essentially involved in the activation as well as differentiation of osteoclasts through a mechanism involving cell-to-cell contact between osteoblasts/stromal cells and osteoclast precursors/
Publikováno v:
Endocrinology. 140(10)
Breast cancers commonly cause osteolytic metastases in bone, a process that is dependent upon osteoclast-mediated bone resorption. Recently the osteoclast differentiation factor (ODF), better termed RANKL (receptor activator of NF-kappaB ligand), exp
Autor:
F M, Collier, W H, Huang, W R, Holloway, J M, Hodge, M T, Gillespie, L L, Daniels, M H, Zheng, G C, Nicholson
Publikováno v:
Endocrinology. 139(3)
Although estrogen is important in human skeletal homeostasis, the major target cell in bone is unknown. Estrogen receptors (ER) have been demonstrated in osteoblasts and bone marrow stromal cells, but their presence in osteoclasts remains controversi
Publikováno v:
International journal of cancer. 73(6)
Several human breast cancer cell lines express the calcitonin receptor (CTR), but this has not been demonstrated previously in clinical breast cancers. We examined 18 primary breast cancers by reverse transcription-PCR, for expression of CTR and of t