Zobrazeno 1 - 10
of 15
pro vyhledávání: '"M. K. Waldor"'
Publikováno v:
mSphere, Vol 6, Iss 4 (2021)
Vibrio parahaemolyticus
Externí odkaz:
https://doaj.org/article/0a228fe28b094ce1974f5844bdcdecf2
Publikováno v:
Virulence, Vol 9, Iss 1, Pp 981-993 (2018)
Vibrio vulnificus can be a highly invasive pathogen capable of spreading from an infection site to the bloodstream, causing sepsis and death. To survive and proliferate in blood, the pathogen requires mechanisms to overcome the innate immune defenses
Externí odkaz:
https://doaj.org/article/69af55aa62cc454b81ff5bef9c7f0151
Publikováno v:
Journal of Medical Entomology. 43:288-295
Publikováno v:
Journal of medical entomology. 43(2)
Whether the house fly, Musca domestica L., gut is a permissive environment for horizontal transfer of antibiotic resistance and virulence genes between strains of Escherichia coli is not known. House flies were immobilized and force fed suspensions o
Publikováno v:
Molecular microbiology. 41(2)
The genes encoding cholera toxin, the principal virulence factor of Vibrio cholerae, are part of the circular single-stranded DNA genome of CTXphi. In toxigenic V. cholerae strains, the CTXphi genome is typically found in integrated arrays of tandeml
Autor:
M K, Waldor, J J, Mekalanos
Publikováno v:
The Journal of infectious diseases. 170(2)
In October 1992, a non-O1 strain of Vibrio cholerae emerged as a cause of epidemic cholera in India and Bangladesh. This antigenically novel clone has been designated serogroup O139 synonym Bengal. Since its emergence, V. cholerae O139 has caused a m
Autor:
M K, Waldor, J J, Mekalanos
Publikováno v:
Lancet (London, England). 343(8909)
Publikováno v:
Infectious agents and disease. 2(4)
Publikováno v:
The Journal of Immunology. 138:3737-3741
Central nervous system toxoplasmosis causes disability and death in up to 30% of patients with acquired immune deficiency syndrome (AIDS). The source of the toxoplasma infection in these patients and the specific immune deficit that allows for this v
Publikováno v:
The Journal of Immunology. 139:3660-3664
Experimental allergic encephalomyelitis (EAE) is an autoimmune disease mediated by CD4+ T cells. Prior studies have established that monoclonal anti-CD4 antibodies can reverse EAE. To determine whether immunoglobulin isotype plays a role in the thera