Zobrazeno 1 - 10
of 32
pro vyhledávání: '"M. J. Holtzman"'
Publikováno v:
The Journal of Immunology. 155:1117-1131
To define the relationship between T cell phenotype and adhesiveness, we examined T cell adhesion to endothelial cell, fibroblast, and epithelial cell monolayers as well as extracellular matrix proteins (collagen and fibronectin) using a three-color
Autor:
M. J. Holtzman, Walter R. Cook, R. A. Mewaldt, M. Mazed, Thomas J. Cunningham, J. J. Rosenberg, Eric R. Fossum, A. C. Cummings
Publikováno v:
Remote Sensing Reviews. 8:245-253
The development of a new two‐dimensional position‐sensitive detector for use in space‐borne energetic particle spectrometers is described. The novel feature of this device is the use of interdigitated pixels to provide both dimensions of positi
Autor:
E. Agapov, J. T. Battaile, R. Tidwell, R. Hachem, G. A. Patterson, R. A. Pierce, J. J. Atkinson, M. J. Holtzman
Publikováno v:
American Journal of Respiratory Cell and Molecular Biology.
Publikováno v:
Apoptosis : an international journal on programmed cell death. 5(5)
Proliferative expansion of lymphoid cells is required for effective immune responses against invading microorganisms, but after the infection is controlled, the expanded effector cells must be eliminated to prevent non-adaptive accumulation of cells.
Publikováno v:
The Biochemical journal.
Interleukin 1beta (IL-1beta) suppresses the IL-6-dependent induction of type II acute-phase response genes, but the underlying mechanism for this suppression remains uncertain. Here we report that treatment of human hepatocullular carcinoma HepG2 cel
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 162(3)
Over activation of CD4+ T cells in the peripheral blood and airway tissues is characteristic of asthma; therefore, we investigated whether activated T cells from asthmatic subjects have altered apoptotic potential through the Fas death receptor. We f
Publikováno v:
Proceedings of the Association of American Physicians. 110(1)
A major goal of our research is to understand how immune cells (especially T cells) infiltrate the pulmonary airway during host defense and inflammatory disease (especially asthma). In that context, we have proposed that epithelial cells lining the a
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 155(3)
To define the relationship between T cell phenotype and adhesiveness, we examined T cell adhesion to endothelial cell, fibroblast, and epithelial cell monolayers as well as extracellular matrix proteins (collagen and fibronectin) using a three-color