Zobrazeno 1 - 4
of 4
pro vyhledávání: '"M. I. Melhorn"'
Autor:
Mitsuko Yuzawa, Ryoichi Arita, Keijiro Ishikawa, Young-Bum Kim, Fumiyuki Sasaki, Hiroshi Enaida, M. I. Melhorn, Enoch Kim, Shigeo Yoshida, Paul Sweetnam, Andrea Vergani, Souska Zandi, Shintaro Nakao, Stewart Campbell, Tatsuro Ishibashi, Alexander Schering, Olivier Schueller, Fang Xie, Yasuaki Hata, Ruth Schmidt-Ullrich, Mahdi Taher, Dawei Sun, Kwang Hoon Chun, Muneo Yamaguchi, Sara Tezza, Francesca Gatti, Ming Zhao, Paolo Fiorina, Takehiko Yokomizo, Ali Hafezi-Moghadam
Publikováno v:
Cell Reports, Vol 10, Iss 7, Pp 1173-1186 (2015)
SummaryAge is a major risk factor in age-related macular degeneration (AMD), but the underlying cause is unknown. We find increased Rho-associated kinase (ROCK) signaling and M2 characteristics in eyes of aged mice, revealing immune changes in aging.
Autor:
Y. Ito, Nuria Lara-Castillo, Ali Hafezi-Moghadam, Ryoichi Arita, Yasuaki Hata, Dawei Sun, Souska Zandi, Tatsuro Ishibashi, Shuhei Kawahara, Shintaro Nakao, Alexander Schering, M. I. Melhorn
Publikováno v:
Blood. 117:1081-1090
Angio- and lymphangiogenesis are inherently related processes. However, how blood and lymphatic vessels regulate each other is unknown. This work introduces a novel mechanism explaining the temporal and spatial relation of blood and lymphatic vessels
Autor:
Sonja Frimmel, Toshio Hisatomi, Kenneth C. Hayes, Kousuke Noda, Faryan Tayyari, L. Almulki, Andrzej Pronczuk, Souska Zandi, M. I. Melhorn, Ali Hafezi-Moghadam
Publikováno v:
The FASEB Journal. 24:2443-2453
Metabolic syndrome (MetS) is a prevalent and complex disease, characterized by the variable coexistence of obesity, dyslipidemia, hyperinsulinaemia, and hypertension. The alarming rise in the prevalence of metabolic disorders makes it imperative to i
Autor:
Shintaro Nakao, Ali Hafezi-Moghadam, Mahdi Taher, M. I. Melhorn, Souska Zandi, Kenneth C. Hayes
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental Biology. 30(7)
The role of lymphatics in atherosclerosis is not yet understood. Here, we investigate lymphatic growth dynamics and marker expression in atherosclerosis in apolipoprotein E-deficient (apoE(-/-)) mice. The prolymphangiogenic growth factor, VEGF-C, was