Zobrazeno 1 - 5
of 5
pro vyhledávání: '"M. Dolors Sans"'
Autor:
Mark J. Hoenerhoff, Bradley Nelson, Guojing Zhu, Brooke McKnight, David Ginsburg, Bin Zhang, M. Dolors Sans, John A. Williams, Lesley Everett, Rami Khoriaty, Nancy L. Vogel, Stephen A. Ernst, Haritha Durairaj
Publikováno v:
Molecular Biology of the Cell
Inactivation of Sec23b exclusively in the pancreatic acinar cells of adult mice results in loss of pancreatic mass, with evidence of cell loss, degeneration of exocrine cells (with smaller-than-normal zymogen granules and ER dilation), ER stress, and
Autor:
Stephen J. Crozier, John A. Williams, Jackie Y. Wang, Stephen I. Lentz, Stephen A. Ernst, M. Dolors Sans
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 299:G1154-G1163
Dietary protein can stimulate pancreatic growth in the absence of CCK release, but there is little data on the regulation of CCK-independent growth. To identify mechanisms whereby protein stimulates pancreatic growth in the absence of CCK release, C5
Publikováno v:
The Journal of Physiology. 573:775-786
Cholecystokinin (CCK)-induced pancreatic growth in mice involves parallel increases in DNA and protein. The mammalian target of rapamycin (mTOR) signalling pathway regulates mRNA translation and its activation is implicated in growth of various tissu
Autor:
John A. Williams, Mitsuo Tashiro, M. Julia Bragado, M. Dolors Sans, Andrzej Dabrowski, Claus Schäfer
Publikováno v:
Pharmacology & Toxicology. 91:297-303
Cholecystokinin (CCK) acting through its G protein-coupled receptor is now known to activate a variety of intracellular signaling mechanisms and thereby regulate a complex array of cellular functions in pancreatic acinar cells. The best studied mecha
Autor:
John A, Williams, M Dolors, Sans, Mitsuo, Tashiro, Claus, Schäfer, M Julia, Bragado, Andrzej, Dabrowski
Publikováno v:
Pharmacologytoxicology. 91(6)
Cholecystokinin (CCK) acting through its G protein-coupled receptor is now known to activate a variety of intracellular signaling mechanisms and thereby regulate a complex array of cellular functions in pancreatic acinar cells. The best studied mecha