Zobrazeno 1 - 10
of 16
pro vyhledávání: '"M. Carla Cabrera"'
Autor:
Priscilla A. Furth, Donald D. Muccio, Clinton J. Grubbs, Michael J. Pishvaian, Bhaskar V.S. Kallakury, Edgar S. Díaz-Cruz, M. Carla Cabrera
PDF file - 643K, Relative expression levels of Cdk1, Rbl1 and Cdc25a in 4- and 7-month-old mice before and after TAg down-regulation by doxycycline treatment for 14 days and in conjunction with treatment with PD0332991 for 10 days, UAB30 for 14 days,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::18fab4c28919e1a5c101a4fde8c129ac
https://doi.org/10.1158/1940-6207.22525134
https://doi.org/10.1158/1940-6207.22525134
Autor:
Priscilla A. Furth, Donald D. Muccio, Clinton J. Grubbs, Michael J. Pishvaian, Bhaskar V.S. Kallakury, Edgar S. Díaz-Cruz, M. Carla Cabrera
PDF file - 688K, Retinyl palmitate levels were decreased in mice treated with UAB30 for 28 days
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7b303750564e275daa1f4140c016fee4
https://doi.org/10.1158/1940-6207.22525128
https://doi.org/10.1158/1940-6207.22525128
Autor:
Priscilla A. Furth, Donald D. Muccio, Clinton J. Grubbs, Michael J. Pishvaian, Bhaskar V.S. Kallakury, Edgar S. Díaz-Cruz, M. Carla Cabrera
Loss of normal growth control is a hallmark of cancer progression. Therefore, understanding the early mechanisms of normal growth regulation and the changes that occur during preneoplasia may provide insights of both diagnostic and therapeutic import
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2d0637b647844371086cfc433845fdc2
https://doi.org/10.1158/1940-6207.c.6544590
https://doi.org/10.1158/1940-6207.c.6544590
Autor:
Priscilla A. Furth, Donald D. Muccio, Clinton J. Grubbs, Michael J. Pishvaian, Bhaskar V.S. Kallakury, Edgar S. Díaz-Cruz, M. Carla Cabrera
PDF file - 830K, TAg was effectively downregulated by doxycycline in submandibular salivary tissue of mice treated with pharmacological agents UAB30, rosiglitazone (ROSI) or PD0332991
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::200e3d804fa38e0706d075940771886f
https://doi.org/10.1158/1940-6207.22525131.v1
https://doi.org/10.1158/1940-6207.22525131.v1
Autor:
Priscilla A. Furth, Donald D. Muccio, Clinton J. Grubbs, Michael J. Pishvaian, Bhaskar V.S. Kallakury, Edgar S. Díaz-Cruz, M. Carla Cabrera
PDF file - 7K
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1948af1debf33e8dfe55509141da5b0f
https://doi.org/10.1158/1940-6207.22525125
https://doi.org/10.1158/1940-6207.22525125
Publikováno v:
Annals of the New York Academy of Sciences. 1229:147-155
Aberrations in estrogen signaling increase breast cancer risk. Molecular mechanisms that impact breast cancer initiation, promotion, and progression can be investigated using genetically engineered mouse models. Increasing estrogen receptor alpha (ER
Autor:
Richard F. Helm, Malcolm Potts, Stephen M. Slaughter, M. Carla Cabrera, Michael L. Manning, Graham D. Jack
Publikováno v:
Cellular Signalling. 19:772-781
Multicellular aggregates (spheroids) of primary human foreskin fibroblasts (HFF-2) and a glioblastoma cell line (T98G) entered and exited from long term (2 weeks) metabolic arrest utilizing an autocrine response. Cytokine production (specifically IFN
Autor:
Timm Schroeder, Priscilla A. Furth, M Carla Cabrera, Sarah Millman, Rebecca E Nakles, Susette C. Mueller, Peter Johnson, Philipp S. Hoppe
Publikováno v:
BMC Proceedings
Background Loss of BRCA1 has been linked to increased cell proliferation in human mammary epithelial cells. To determine if this phenotype is mirrored in the normalappearing mammary epithelial cells from mouse models of BRCA1 deficiency, time-lapse i
Autor:
Sarah Millman, Peter Johnson, Priscilla A. Furth, Susette C. Mueller, Timm Schroeder, M. Carla Cabrera, Philipp S. Hoppe, Rebecca E. Nakles
Publikováno v:
J. Vis. Exp. 72:e50198 (2013)
Time-lapse imaging can be used to compare behavior of cultured primary preneoplastic mammary epithelial cells derived from different genetically engineered mouse models of breast cancer. For example, time between cell divisions (cell lifetimes), apop
Publikováno v:
Breast Cancer Research : BCR
STAT5 consists of two proteins, STAT5A/B, that impact mammary cell differentiation, proliferation, and survival. In normal development, STAT5 expression and activity are regulated by prolactin signaling with JAK2/ELF5, EGF signaling networks that inc