Zobrazeno 1 - 5
of 5
pro vyhledávání: '"M. Amany Fawzy"'
Autor:
Amira Shoukry, Nagwa S. Said, M.N. Ayman Abd-Elrahman, Tamer Saber, M. Amany Fawzy, Sally Shalaby
Publikováno v:
Egyptian Journal of Chest Disease and Tuberculosis, Vol 62, Iss 4, Pp 567-574 (2013)
Background: Thyroid dysfunction or non-thyroidal illness syndrome (NTIS) is frequently detected in chronic, systemic diseases. The systemic manifestations of chronic obstructive pulmonary disease (COPD) include a number of endocrine disorders. The se
Externí odkaz:
https://doaj.org/article/76f2e88e0918403f903da9fca4f4a923
Publikováno v:
Egyptian Journal of Chest Disease and Tuberculosis, Vol 62, Iss 3, Pp 501-511 (2013)
Background: Significant portions of central airway stenosis patients present with unresectable disease. Using bronchotherapeutic procedures to maintain a patent airway and improve clinical symptoms and quality of life is a well-known armamentarium te
Externí odkaz:
https://doaj.org/article/a0600b8e34d543d681ad249f12507327
Publikováno v:
Egyptian Journal of Chest Disease and Tuberculosis, Vol 62, Iss 1, Pp 45-50 (2013)
Background: Adiponectin (APN) is a secretory protein synthesized by adipocytes and plays a potential role in regulating the inflammatory response by many of autocrine or paracrine. The mechanism of APN participation in the inflammation of COPD remain
Externí odkaz:
https://doaj.org/article/43f0c8c7744d4f359aa76c304e913d7b
Autor:
Sally M. Shalaby, Nagwa S. Said, M. Amany Fawzy, M.N. Ayman Abd-Elrahman, Amira Shoukry, Tamer Saber
Publikováno v:
Egyptian Journal of Chest Disease and Tuberculosis, Vol 62, Iss 4, Pp 567-574 (2013)
Background: Thyroid dysfunction or non-thyroidal illness syndrome (NTIS) is frequently detected in chronic, systemic diseases. The systemic manifestations of chronic obstructive pulmonary disease (COPD) include a number of endocrine disorders. The se
Publikováno v:
Egyptian Journal of Chest Disease and Tuberculosis, Vol 62, Iss 1, Pp 45-50 (2013)
Background: Adiponectin (APN) is a secretory protein synthesized by adipocytes and plays a potential role in regulating the inflammatory response by many of autocrine or paracrine. The mechanism of APN participation in the inflammation of COPD remain