Zobrazeno 1 - 10
of 18
pro vyhledávání: '"M. A. Petroff"'
Autor:
P. A. R. Ade, Z. Ahmed, M. Amiri, D. Barkats, R. Basu Thakur, C. A. Bischoff, D. Beck, J. J. Bock, H. Boenish, E. Bullock, V. Buza, J. R. Cheshire IV, S. E. Clark, J. Connors, J. Cornelison, M. Crumrine, A. Cukierman, E. V. Denison, M. Dierickx, L. Duband, M. Eiben, S. Fatigoni, J. P. Filippini, S. Fliescher, C. Giannakopoulos, N. Goeckner-Wald, D. C. Goldfinger, J. Grayson, P. Grimes, G. Hall, G. Halal, M. Halpern, E. Hand, S. Harrison, S. Henderson, S. R. Hildebrandt, J. Hubmayr, H. Hui, K. D. Irwin, J. Kang, K. S. Karkare, E. Karpel, S. Kefeli, S. A. Kernasovskiy, J. M. Kovac, C. L. Kuo, K. Lau, E. M. Leitch, A. Lennox, K. G. Megerian, L. Minutolo, L. Moncelsi, Y. Nakato, T. Namikawa, H. T. Nguyen, R. O’Brient, R. W. Ogburn IV, S. Palladino, M. A. Petroff, T. Prouve, C. Pryke, B. Racine, C. D. Reintsema, S. Richter, A. Schillaci, R. Schwarz, B. L. Schmitt, C. D. Sheehy, B. Singari, A. Soliman, T. St. Germaine, B. Steinbach, R. V. Sudiwala, G. P. Teply, K. L. Thompson, J. E. Tolan, C. Tucker, A. D. Turner, C. Umiltà, C. Vergès, A. G. Vieregg, A. Wandui, A. C. Weber, D. V. Wiebe, J. Willmert, C. L. Wong, W. L. K. Wu, H. Yang, K. W. Yoon, E. Young, C. Yu, L. Zeng, C. Zhang, S. Zhang, BICEP/Keck Collaboration
Publikováno v:
The Astrophysical Journal, Vol 945, Iss 1, p 72 (2023)
We characterize Galactic dust filaments by correlating BICEP/Keck and Planck data with polarization templates based on neutral hydrogen (H i ) observations. Dust polarization is important for both our understanding of astrophysical processes in the i
Externí odkaz:
https://doaj.org/article/29fae8a42e284f7da0b1856a856fd13b
Autor:
Tristan W. Harding, L.M.D. Delbridge, Claire L. Curl, M. Vila Petroff, Jimmy D. Bell, Stephen B. Harrap
Cardiac hypertrophy is the most potent cardiovascular risk factor after age, with relative mortality risk greater in women. The cognate issue of whether ischaemia coincident with hypertrophic co-morbidity has differing gender aetiology/outcome has no
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::02a43b395d57aeaac561d5e44edcccc4
http://sedici.unlp.edu.ar/handle/10915/140257
http://sedici.unlp.edu.ar/handle/10915/140257
Publikováno v:
Reproduction. :753-760
Tumour necrosis factor alpha (TNF-alpha) and gamma-interferon (IFN-gamma) are cytotoxic to bovine luteal cells in vitro and may contribute to cell death during luteolysis in vivo. In this study, the mechanism by which luteal cells are killed by TNF-a
Publikováno v:
Biology of Reproduction. 62:879-885
Macrophage migration inhibitory factor (MIF) is a pro-inflammatory cytokine produced by T cells and macrophages. A number of tissues also produce MIF during states of active differentiation and/or proliferation. The purpose of this study was to deter
Publikováno v:
Endocrinology. 140:1018-1021
There is considerable evidence that luteolysis in the cow and other species involves components of the immune system. In this study, we examined the expression of the mRNAs for TNF-alpha, IFN-gamma, IL-1beta, IL-2, and IL-2 receptor (IL-2R) by revers
Bovine Luteal Cells Elicit Major Histocompatibility Complex Class II-Dependent T-Cell Proliferation1
Publikováno v:
Biology of Reproduction. 57:887-893
Major histocompatibility complex (MHC) class II molecules are expressed in the bovine corpus luteum (CL) in a manner correlating with luteolysis. Whether bovine luteal cells can stimulate T-cell proliferation in a class II-restricted manner was inves
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 267:H1222-H1226
In rabbits chronically pretreated with nifedipine (20 mg/day for 25 days), we demonstrate upregulation of Ca2+ channels in mammalian heart (maximal binding capacity = 222 +/- 14 and 421 +/- 55 fmol/mg protein for control and treated, respectively; P
Publikováno v:
Reproduction (Cambridge, England). 121(5)
Tumour necrosis factor alpha (TNF-alpha) and gamma-interferon (IFN-gamma) are cytotoxic to bovine luteal cells in vitro and may contribute to cell death during luteolysis in vivo. In this study, the mechanism by which luteal cells are killed by TNF-a
Publikováno v:
Biology of reproduction. 62(4)
Macrophage migration inhibitory factor (MIF) is a pro-inflammatory cytokine produced by T cells and macrophages. A number of tissues also produce MIF during states of active differentiation and/or proliferation. The purpose of this study was to deter
Autor:
M K, Petroff
Publikováno v:
Journal of nursing staff development : JNSD. 10(5)