Zobrazeno 1 - 10
of 11
pro vyhledávání: '"M. A. Esparza-Flores"'
Autor:
M. A. Esparza-Flores, Beatriz López-Guido, Lilia Beatriz Aguilar-López, Ana Rebeca Jaloma-Cruz, Hilda Luna-Záizar, C. R. Cortés Álvarez
Publikováno v:
International Journal of Laboratory Hematology. 31:673-682
Severe hemophilia A (HA) patients develop inhibitory alloantibodies to factor VIII:C and therefore require bypass agents that are scarce, expensive and may provoke secondary effects. Twenty-three severe HA patients who were high-responders to FVIII i
Autor:
J N Márquez-Reyes, Z C De Alba-López, J E Herrera-Ruvalcaba, M C Terrones-Saldívar, M Juárez-Ávila, M I Esparza-Flores, A Rosas-Cabral, J Del Real-Cajero, N P Malo-Martínez, R A García-González
Publikováno v:
Lux Médica. 4:15-20
Objetivo: Conocer la prevalencia de sobrepeso y obesidad en escolares preadolescentes del municipio de Aguascalientes en el periodo comprendido entre abril y mayo de 2008, mediante la medición del índice de masa corporal (IMC). Material y Métodos:
Autor:
Alberto Tlacuilo-Parra, Javier Orozco-Alcala, Nadia Tostado-Rabago, Beatriz López-Guido, Ramses Morales-Zambrano, M. A. Esparza-Flores
Publikováno v:
British Journal of Haematology. 140:562-567
Reduced bone mineral density (BMD) in childhood is a risk factor for osteoporosis in later life. This case-control study determined the prevalence of low BMD, calcium intake and physical activity in 62 haemophilic children and 62 sex-, race- and age-
Autor:
Johanna Milena Mantilla-Capacho, Hilda Luna-Záizar, Claudia Patricia Beltrán-Miranda, Lilia Beatriz Aguilar-López, Ana Rebeca Jaloma-Cruz, Rogelio Troyo-Sanromán, Beatriz López-Guido, M. A. Esparza-Flores
Publikováno v:
American Journal of Hematology. 82:283-287
Hemophilia A (HA) is one of the most common inherited bleeding disorders caused by FVIII gene mutations. Inversion of intron 22 (inv22) originates 50% of cases of severe HA and is a major risk factor for inhibitor development. Inversion of intron 1 (
Autor:
E. Leal, Lilia Beatriz Aguilar-López, M. A. Esparza-Flores, Patricio Barros-Núñez, C. Aguilar-Luna, Beatriz López-Guido, C. Medina, A. R. Jaloma-Cruz
Publikováno v:
Hematological Oncology. 21:25-31
Bone marrow (BM) is accepted as the tissue of choice for the detection of monoclonal populations in leukemias and lymphomas; however, obtaining BM can be painful and traumatic for the patients. Although it is possible to detect clonality in periphera
Autor:
M. D. C. Rodríguez-Zepeda, M. A. Esparza-Flores, Janet Soto-Padilla, Hilda Luna-Záizar, A. R. Jaloma-Cruz, A. Berges-Garcia, M. T. Pompa-Garza, Claudia Patricia Beltrán-Miranda
Publikováno v:
Haemophilia : the official journal of the World Federation of Hemophilia. 20(1)
Summary In Mexico, 15% of haemophilia A (HA) patients develop inhibitory alloantibodies in response to replacement therapy with factor VIII (FVIII), requiring bypass therapy such as activated prothrombin complex concentrate (APCC). Because bypass the
Autor:
J. P. Meza-Espinoza, Melva Gutiérrez-Angulo, Juan Ramón González-García, B. Lopez-Guido, Verónica Judith Picos-Cárdenas, Horacio Rivera, M. A. Esparza-Flores
Publikováno v:
Hematological oncology. 22(3)
A semi-quantitative expression analysis of both AML1-a and AML1-total was performed by RT-PCR in 19 children with acute lymphoblastic leukemia (ALL) at diagnosis. AML1-a expression was assessed in 16 bone marrow (BM) and 13 peripheral blood (PB) samp
Autor:
E, Leal, M A, Esparza-Flores, B, López-Guido, C, Aguilar-Luna, L, Aguilar-López, A R, Jaloma-Cruz, C, Medina, P, Barros-Núñez
Publikováno v:
Hematological oncology. 21(1)
Bone marrow (BM) is accepted as the tissue of choice for the detection of monoclonal populations in leukemias and lymphomas; however, obtaining BM can be painful and traumatic for the patients. Although it is possible to detect clonality in periphera
Autor:
M. A. Esparza-Flores, Melva Gutiérrez-Angulo, Ingo Hansmann, G.J.R. González, Verónica Judith Picos-Cárdenas, María de la Luz Ayala-Madrigal, J. P. Meza-Espinoza
Publikováno v:
Cancer genetics and cytogenetics. 134(2)
We report a boy with Down syndrome and leukemia who acquired uniparental isodisomy of chromosome 7q as a secondary chromosomal change during recurrence of the disease. His karyotype before therapy was 46,XY,der(1)t(1;1)(p36;q32),−7,+21c[17]/46,idem
Autor:
H, Benítez-Aranda, J M, Farfán Canto, M A, Esparza-Flores, R, Bernaldez-Ríos, C, Alvarez-Amaya
Publikováno v:
Boletin medico del Hospital Infantil de Mexico. 42(11)