Výsledky vyhledávání - "M van der Paardt"

Increased disease activity is associated with a deteriorated lipid profile in patients with ankylosing spondylitis

Autor: Mike J L Peters, I E van der Horst-Bruinsma, M H M T de Koning, Ben A. C. Dijkmans, R J van de Stadt, Michael T. Nurmohamed, M van der Paardt, J.C. van Denderen, Jos W. R. Twisk, V P van Halm

Publikováno v: Annals of the Rheumatic Diseases, 65(11), 1473-7. BMJ Publishing Group
van Halm, V P, van Denderen, J C, Peters, M J, Twisk, J W, van der Paardt, M, van der Horst-Bruinsma, I E, van de Stadt, RJ, Koning, M H M T, Dijkmans, B A C & Nurmohamed, M T 2006, ' Increased disease activity is associated with a deteriorated lipid profile in patients with ankylosing spondylitis ', Annals of the Rheumatic Diseases, vol. 65, no. 11, pp. 1473-1477 . https://doi.org/10.1136/ard.2005.050443
van Halm, V P, van Denderen, J C, Peters, M J L, Twisk, J W R, van der Paardt, M, van der Horst-Bruinsma, I E, van de Stadt, R J, de Koning, M H M T, Dijkmans, B A C & Nurmohamed, M T 2006, ' Increased disease activity is associated with a deteriorated lipid profile in patients with ankylosing spondylitis ', Annals of the Rheumatic Diseases, vol. 65, no. 11, pp. 1473-7 . https://doi.org/10.1136/ard.2005.050443
Annals of the Rheumatic Diseases, 65(11), 1473-1477. BMJ Publishing Group

BACKGROUND: Cardiovascular mortality is increased in patients with ankylosing spondylitis. A possible explanation might be a more prevalent atherogenic lipid profile in patients with ankylosing spondylitis than in the general population. It has been

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ea1234d7c23030a17c456eb45323a573
https://doi.org/10.1136/ard.2005.050443

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Double blind, randomised, placebo controlled study of leflunomide in the treatment of active ankylosing spondylitis

Autor: J.C. van Denderen, B A C Dijkmans, I E van der Horst-Bruinsma, Michael T. Nurmohamed, M van der Paardt, Y M M A de Ryck

Publikováno v: Annals of the Rheumatic Diseases, 64(12), 1761-1764. BMJ Publishing Group
van Denderen, J C, van der Paardt, M, Nurmohamed, M T, de Ryck, Y M, Dijkmans, B A C & van der Horst-Bruinsma, I E 2005, ' Double blind, randomised, placebo controlled study of leflunomide in the treatment of active ankylosing spondylitis ', Annals of the Rheumatic Diseases, vol. 64, no. 12, pp. 1761-1764 . https://doi.org/10.1136/ard.2005.036491

Objective: To assess the efficacy and safety of leflunomide in active ankylosing spondylitis (AS) compared with placebo in a 24 week pilot study. Methods: In a single centre randomised, double blind, placebo controlled study, 45 patients with active

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c82d27f99178a1fc4bffff3583b31eb0
https://doi.org/10.1136/ard.2005.036491

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Susceptibility to ankylosing spondylitis: no evidence for the involvement of transforming growth factor beta 1 (TGFB1) gene polymorphisms

Autor: M van der Paardt, B A C Dijkmans, J. B. A. Crusius, Amado Salvador Peña, Maria Asuncion Garcia-Gonzalez, I E van der Horst-Bruinsma

Publikováno v: Annals of the Rheumatic Diseases, 64(4), 616-619. BMJ Publishing Group
van der Paardt, M, Crusius, J B A, Garcia-Gonzalez, MA, Dijkmans, B A C, Pena, A S & van der Horst-Bruinsma, I E 2005, ' Susceptibility to ankylosing spondylitis: no evidence for the involvement of transforming growth factor beta 1 (TGFB1) gene polymorphisms ', Annals of the Rheumatic Diseases, vol. 64, no. 4, pp. 616-619 . https://doi.org/10.1136/ard.2004.027698

Background: Genetic factors are thought to be crucial in the pathogenesis of ankylosing spondylitis. Transforming growth factor β1 (TGFβ1) is a multifunctional cytokine that plays a key role in inflammation. Two functional single nucleotide polymor

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cff59a4d3dc5558f169a0780343a38b0
https://doi.org/10.1136/ard.2004.027698

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NHG-Standaard Artritis

Autor: M. van der Paardt, H. Woutersen-Koch, Hjem Janssens, K.J. Gorter, P. van der Pas, P. G. Van Peet, Hahm Lagro

Publikováno v: NHG-Standaarden voor de huisarts 2011 ISBN: 9789031382781
NHG-Standaarden voor de huisarts 2011

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::7a4f480ba40374da06d96f341639bdf9
https://doi.org/10.1007/978-90-313-8279-8_30

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Prevalence of Chlamydia trachomatis in urine of male patients with ankylosing spondylitis is not increased

Autor: B A C Dijkmans, Servaas A. Morré, J.C. van Denderen, I E van der Horst-Bruinsma, M van der Paardt, A J C van den Brule, P D Bezemer

Publikováno v: Annals of the Rheumatic Diseases. 59:300-302

OBJECTIVE To compare the prevalence of Chlamydia trachomatis infections in ankylosing spondylitis (AS) patients with controls, using DNA amplification assays in urine specimens. METHODS The prevalence of C trachomatis infections was assessed in 32 ma

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be04f2b74ab726865fa66a4878b27db1
https://doi.org/10.1136/ard.59.4.300

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No evidence for involvement of the Toll-like receptor 4 (TLR4) A896G and CD14-C260T polymorphisms in susceptibility to ankylosing spondylitis

Autor: M H M T de Koning, Amado Salvador Peña, I E van der Horst-Bruinsma, R J van de Stadt, B A C Dijkmans, M van der Paardt, J. B. A. Crusius, Servaas A. Morré

Publikováno v: van der Paardt, M, Crusius, J B A, Koning, M H M T, Morre, S A, van de Stadt, RJ, Dijkmans, B A C, Pena, A S & van der Horst-Bruinsma, I E 2005, ' No evidence for involvement of the Toll-like receptor 4 (TLR4) A896G and CD14-C260T polymorphisms in susceptibility to ankylosing spondylitis ', Annals of the Rheumatic Diseases, vol. 64, no. 2, pp. 235-238 . https://doi.org/10.1136/ard.2004.021105
Annals of the Rheumatic Diseases, 64(2), 235-238. BMJ Publishing Group

Objectives: Ankylosing spondylitis (AS) is a multifactorial and polygenic disease. Apart from HLA, other genes very probably play a role in disease susceptibility. Indigenous bacteria of the gastrointestinal flora appear to play a role in the pathoge

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9fdd7ca952d5f5167c767b9b4e6ae0d3
https://research.vumc.nl/en/publications/f6d3caf7-ca52-4159-8bc8-ac5fc06a6f2d

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Interleukin-1β and interleukin-1 receptor antagonist gene polymorphisms in ankylosing spondylitis

Autor: Pieter J. Kostense, Behrooz Z. Alizadeh, B A C Dijkmans, I E van der Horst-Bruinsma, Maria Asuncion Garcia-Gonzalez, J B A Crusius, P. Baudoin, Amado Salvador Peña, M van der Paardt

Publikováno v: Rheumatology. Oxford University Press
Rheumatology, 41(12), 1419-1423. Oxford University Press

OBJECTIVE: Since ulcerative colitis and Crohn's disease, which are associated with ankylosing spondylitis (AS), have been found to be variably associated with the IL-1B and the IL-1RN genes encoding interleukin-1beta (IL-1beta) and the interleukin-1

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5095b97a3ec938ad118e12289aa2e278
https://research.rug.nl/en/publications/684ba2de-a833-4c42-9eab-d833db0e43ba

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CARD15 gene mutations are not associated with ankylosing spondylitis

Autor: Laura Murillo, R J van de Stadt, M van der Paardt, M H M T de Koning, Ben A. C. Dijkmans, J B A Crusius, Amado Salvador Peña, I E van der Horst-Bruinsma

Publikováno v: van der Paardt, M, Crusius, J B A, Koning, M H, Murillo, L S, van de Stadt, RJ, Dijkmans, B A C, Pena, A S & van der Horst-Bruinsma, I E 2003, ' CARD15 gene mutations are not associated with ankylosing spondylitis. ', Genes and Immunity, vol. 4, no. 1, pp. 77-8 . https://doi.org/10.1038/sj.gene.6363914
Genes and Immunity, 4(1), 77-8. Nature Publishing Group

An insertion mutation at nucleotide 3020 (3020insC) and a missense mutation G2722C in the CARD15 gene on chromosome 16p have been reported to be associated with Crohn's disease (CD). The protein encoded by the CARD15 gene is expressed in peripheral m

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd266b90d9a1230bf146d86c08eb5497
https://doi.org/10.1038/sj.gene.6363914

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