Zobrazeno 1 - 10
of 76
pro vyhledávání: '"M Stamatovic"'
Autor:
Chelsea M. Phillips, Allison M. Johnson, Svetlana M. Stamatovic, Richard F. Keep, Anuska V. Andjelkovic
Publikováno v:
Neurobiology of Disease, Vol 186, Iss , Pp 106277- (2023)
Cerebral cavernous malformation type-3 (CCM3) is a type of brain vascular malformation caused by mutations in programmed cell death protein-10 (PDCD10). It is characterized by early life occurrence of hemorrhagic stroke and profound blood-brain barri
Externí odkaz:
https://doaj.org/article/3789c476f5bf4676a0ee02db6fe36697
Publikováno v:
Fluids and Barriers of the CNS, Vol 20, Iss 1, Pp 1-23 (2023)
Abstract Incomplete recovery of blood–brain barrier (BBB) function contributes to stroke outcomes. How the BBB recovers after stroke remains largely unknown. Emerging evidence suggests that epigenetic factors play a significant role in regulating p
Externí odkaz:
https://doaj.org/article/97350e4e10d947fdb76c9e0fe6dca07b
Autor:
Muyu Situ, Ali Francisco Citalan-Madrid, Svetlana M. Stamatovic, Richard F. Keep, Anuska V. Andjelkovic
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 16 (2022)
Cerebral amyloid angiopathy (CAA) is a small vessel disease characterized by amyloid β (Aβ) peptide deposition within the walls of medium to small-caliber blood vessels, cerebral microhemorrhage, and blood–brain barrier (BBB) leakage. It is commo
Externí odkaz:
https://doaj.org/article/bd1a6beb278a42199fc3104646bfb26b
Autor:
Anuska V. Andjelkovic, Svetlana M. Stamatovic, Chelsea M. Phillips, Gabriela Martinez-Revollar, Richard F. Keep
Publikováno v:
Fluids and Barriers of the CNS, Vol 17, Iss 1, Pp 1-21 (2020)
Abstract The complexity of the blood–brain barrier (BBB) and neurovascular unit (NVU) was and still is a challenge to bridge. A highly selective, restrictive and dynamic barrier, formed at the interface of blood and brain, the BBB is a “gatekeepe
Externí odkaz:
https://doaj.org/article/967802406b744d6b897da3f4c2399b98
Autor:
Svetlana M. Stamatovic, Gabriela Martinez-Revollar, Anna Hu, Jennifer Choi, Richard F. Keep, Anuska V. Andjelkovic
Publikováno v:
Neurobiology of Disease, Vol 126, Iss , Pp 105-116 (2019)
Accumulating evidence suggest that cerebral microvascular disease increases with advancing age and is associated with lacunar stroke, leukoaraiosis, vascular dementia and Alzheimer disease. Increased blood brain barrier (BBB) permeability/leakage tak
Externí odkaz:
https://doaj.org/article/02925d2696024f288d605ee6568b35f9
Autor:
Anuska V. Andjelkovic, Muyu Situ, Ali Francisco Citalan-Madrid, Svetlana M. Stamatovic, Jianming Xiang, Richard F. Keep
Publikováno v:
Stroke. 54:661-672
Cerebral endothelial cells and their linking tight junctions form a unique, dynamic and multi-functional interface, the blood-brain barrier (BBB). The endothelium is regulated by perivascular cells and components forming the neurovascular unit. This
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a3280370df080b45d7328aee801886a7
https://doi.org/10.1161/strokeaha.122.040578
https://doi.org/10.1161/strokeaha.122.040578
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 9, p 5000 (2022)
Cerebral cavernous malformation (CCM) is a cerebromicrovascular disease that affects up to 0.5% of the population. Vessel dilation, decreased endothelial cell–cell contact, and loss of junctional complexes lead to loss of brain endothelial barrier
Externí odkaz:
https://doaj.org/article/7c763f8626d9435bb3948d7d8ef95aab
Autor:
Svetlana M. Stamatovic, Chelsea M. Phillips, Gabriela Martinez-Revollar, Richard F. Keep, Anuska V. Andjelkovic
Publikováno v:
Frontiers in Neuroscience, Vol 13 (2019)
Cessation of blood flow leads to a complex cascade of pathophysiological events at the blood-vascular-parenchymal interface which evolves over time and space, and results in damage to neural cells and edema formation. Cerebral ischemic injury evokes
Externí odkaz:
https://doaj.org/article/1166721c363946ddad3bdaaee43d74f4
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 2492
Cerebral ischemic injury evokes a complex cascade of pathophysiological events at the blood-vascular-parenchymal interface. These evolve over time and space and result in progressive neurological damage. Emerging evidence suggests that blood-brain ba
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::5d77972d1592ea1f3df3dc509b922e14
https://pubmed.ncbi.nlm.nih.gov/35733052
https://pubmed.ncbi.nlm.nih.gov/35733052
Autor:
Nikola Sladojevic, Svetlana M. Stamatovic, Richard F. Keep, Jamison J. Grailer, J. Vidya Sarma, Peter A. Ward, Anuska V. Andjelkovic
Publikováno v:
Neurobiology of Disease, Vol 67, Iss , Pp 57-70 (2014)
Proinflammatory mediators trigger intensive postischemic inflammatory remodeling of the blood–brain barrier (BBB) including extensive brain endothelial cell surface and junctional complex changes. Junctional adhesion molecule-A (JAM-A) is a compone
Externí odkaz:
https://doaj.org/article/be26f4c1339244e68041ab1d400ff618