Zobrazeno 1 - 10
of 109
pro vyhledávání: '"M Saeed, Sheikh"'
Publikováno v:
International Journal of Molecular Sciences, Vol 25, Iss 11, p 5854 (2024)
Esophageal Cancer-Related Gene 2 (ECRG2), also known as Serine Peptidase Inhibitor Kazal type 7 (SPINK7), is a novel tumor suppressor gene from the SPINK family of genes that exhibits anticancer potential. ECRG2 was originally identified during effor
Externí odkaz:
https://doaj.org/article/516d92cb9573468c8ba6b2aea2866bd6
Autor:
M Saeed, Sheikh, Ying, Huang
Publikováno v:
Recent Patents on Anti-Cancer Drug Discovery. 18:108-113
Abstract: The use of antibody-drug conjugates is expected to transform the management of human malignancy. Antibody-drug conjugates for cancer treatment are designed to deliver anticancer drugs to tumor cells. The main components of such conjugates a
Publikováno v:
Journal of Experimental & Clinical Cancer Research, Vol 38, Iss 1, Pp 1-15 (2019)
Abstract Background Recently, we have reported the characterization of a novel protein named Coiled-coil Helix Tumor and Metabolism 1 (CHTM1). CHTM1 localizes to both cytosol and mitochondria. Sequence corresponding to CHTM1 is also annotated in the
Externí odkaz:
https://doaj.org/article/7ae0c11803214e45b8169237fd5387da
Autor:
Ying Huang, M. Saeed Sheikh, Jingxue Shi, Weixin Jin, JoAnne Montalbano, Xiuquan Luo, Hong Sun
Supplementary Data from DOC45, a Novel DNA Damage–Regulated Nucleocytoplasmic ATPase That Is Overexpressed in Multiple Human Malignancies
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::104f423307f1b327f7549db9b99c9946
https://doi.org/10.1158/1541-7786.22519056
https://doi.org/10.1158/1541-7786.22519056
Supplementary Tables S1-S4 from Neutral Sphingomyelinase-3 Is a DNA Damage and Nongenotoxic Stress-Regulated Gene That Is Deregulated in Human Malignancies
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b239ae76575c819f39ce831056cd87c3
https://doi.org/10.1158/1541-7786.22521711.v1
https://doi.org/10.1158/1541-7786.22521711.v1
Autor:
Ying Huang, Yingjie Hu, M. Saeed Sheikh, Lingyan Jiang, Weixin Jin, Xiaoling Shen, Kanglun Liu, Qing Sun
PDF - 430K, Supplementary Fig. S1 UV Spectrum of Lappaol F Supplementary Fig. S2 1H NMR Spectrum of Lappaol F Supplementary Fig. S3 13C NMR Spectrum of Lappaol F. Supplementary Fig. S4 ESI MS Spectrum of Lappaol F Supplentary Fig S5 Purity of Lappaol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6985b88d9cbce923cb0ca48b97dc453b
https://doi.org/10.1158/1535-7163.22503564.v1
https://doi.org/10.1158/1535-7163.22503564.v1
Autor:
Ying Huang, M. Saeed Sheikh, Jingxue Shi, Weixin Jin, JoAnne Montalbano, Xiuquan Luo, Hong Sun
In this article, we report the characterization of a novel DNA damage–regulated gene, named DNA damage–regulated overexpressed in cancer 45 (DOC45). Our results indicate that DNA damage–inducing agents, including doxorubicin (adriamycin), etopo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0027292916c072c426bc646568a509ce
https://doi.org/10.1158/1541-7786.c.6542271.v1
https://doi.org/10.1158/1541-7786.c.6542271.v1
We have identified a p53 and DNA damage–regulated gene that encodes a novel IQ motif protein, which we have named p53 and DNA damage–regulated IQ motif protein (PIQ). PIQ has two isoforms, long (PIQ-L) and short (PIQ-S), and both bind to calmodul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::403555589628dfc9d72fe15b5ddda52d
https://doi.org/10.1158/0008-5472.c.6494609.v1
https://doi.org/10.1158/0008-5472.c.6494609.v1
Autor:
M Saeed, Sheikh, Siem A, Satti
Publikováno v:
Mol Cell Pharmacol
The cyclin-dependent kinase (CDK) inhibitors have emerged as important cancer therapeutics. To date, three CDK4/6 inhibitors in combination with endocrine therapy have been approved by the U.S. Food and Drug Administration for the treatment of hormon
Publikováno v:
Cell Death Differ
We have previously reported that Monoglyceride Lipase (MGL) expression is absent or reduced in various human malignancies and MGL-deficient mice develop tumors in multiple organs. Evidence also suggests MGL to be a tumor suppressor, however, the mech