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Relative expression of epidermal growth factor receptor in placental cytotrophoblasts and choriocarcinoma cell lines

Autor: Karen L. Kaul, M. S. Filla

Publikováno v: Placenta. 18:17-27

The role of transforming growth factor-alpha (TGF-alpha)-epidermal growth factor receptor (EGFR) interactions in regulating benign and malignant trophoblast proliferation were examined. Benign cytotrophoblast (CT) demonstrated mitogenic stimulation i

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::29f85c2914e194f05e74067739f72d09
https://doi.org/10.1016/s0143-4004(97)90067-9

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The cell surface proteoglycan syndecan-1 mediates fibroblast growth factor-2 binding and activity

Autor: M S, Filla, P, Dam, A C, Rapraeger

Publikováno v: Journal of cellular physiology. 174(3)

Binding of fibroblast growth factors (FGFs) to receptor tyrosine kinases (FGFRs) and signaling is facilitated by binding of FGF to heparan sulfate proteoglycans (HSPGs). There are multiple families of HSPGs, including extracellular and cell surface f

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::7600d7ba35c12308f6cf9db4333fe8be
https://pubmed.ncbi.nlm.nih.gov/9462693

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A potential transforming growth factor alpha/epidermal growth factor receptor autocrine circuit in placental cytotrophoblasts

Autor: M S, Filla, C X, Zhang, K L, Kaul

Publikováno v: Cell growthdifferentiation : the molecular biology journal of the American Association for Cancer Research. 4(5)

A potential autocrine loop involving transforming growth factor alpha (TGF-alpha) and the epidermal growth factor (EGF) receptor (EGFR) in benign placental cytotrophoblasts was examined. EGFR were localized to villous cytotrophoblasts and syncytiotro

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::74356b2f614c7b249eb947a3ba24e496
https://pubmed.ncbi.nlm.nih.gov/8518231

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Distribution and metabolism of the retinoid, N-(4-methoxyphenyl)-all-trans-retinamide, the major metabolite of N-(4-hydroxyphenyl)-all-trans-retinamide, in female mice

Autor: T A, Hultin, M S, Filla, D L, McCormick

Publikováno v: Drug metabolism and disposition: the biological fate of chemicals. 18(2)

The metabolism and disposition of N-(4-methoxyphenyl)-all-trans-retinamide (MPR), the major metabolite of N-(4-hydroxyphenyl)-all-trans-retinamide (4-HPR), were investigated in female B6D2F1 (BDF) mice. Following a single oral dose of 10 mg/kg, MPR d

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::5a9330b2b2421666a7f596153892aa31
https://pubmed.ncbi.nlm.nih.gov/1971569

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