Zobrazeno 1 - 10 of 17 pro vyhledávání: '"M R, Yelich"'
1
Role of somatostatin in glucose regulation during endotoxicosis in the rat
Autor: Beth A. Drolet, D. M. Umporowicz, M. R. Yelich
Publikováno v: American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 264:R254-R261
This study assessed the role of somatostatin in regulating glucose homeostasis during endotoxicosis in the rat. Plasma levels of somatostatin, glucose, lactate, insulin, and glucagon were measured in control and endotoxin-treated rats. Cysteamine was
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e85200d921829e765f0ea84d41cc2a11
https://doi.org/10.1152/ajpregu.1993.264.2.r254
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2
In vivo endotoxin and IL-1 potentiate insulin secretion in pancreatic islets
Autor: M. R. Yelich
Publikováno v: American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 258:R1070-R1077
This study evaluated the in vivo effects of endotoxin and interleukin 1 (IL-1) on in vitro insulin secretion from perfused rat pancreases and isolated pancreatic islets. Glucose-induced insulin secretion was potentiated in pancreases obtained from ra
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::49ac6d5a0387083a87ce5ddc623179b6
https://doi.org/10.1152/ajpregu.1990.258.4.r1070
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3
Role of the adrenal cortex and medulla in the young rats' glucoregulatory response to endotoxin
Autor: L, Witek-Janusek, M R, Yelich
Publikováno v: Shock (Augusta, Ga.). 3(6)
The importance of the adrenal gland in the 10 day old (10 d) rats' glucoregulatory response to endotoxin (ETX) was investigated. Plasma glucose, lactate, and liver glycogen were measured at 0, 2, 3, and 4 h after ETX to 10 d adrenalectomized (ADRNX),
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::f9733f693439ad83a723b5df629a0493
https://pubmed.ncbi.nlm.nih.gov/7656068
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4
The effect of epinephrine on insulin and glucagon secretion from the endotoxic rat pancreas
Autor: M R, Yelich
Publikováno v: Pancreas. 8(4)
This study determined whether in vivo endotoxin treatment alters the ability of epinephrine to inhibit immunoreactive insulin (IRI) secretion or to stimulate immunoreactive glucagon (IRG) secretion from the in vitro perfused rat pancreas preparation.
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::7c7d17b37f748dc9f4c30a9a5596b18c
https://pubmed.ncbi.nlm.nih.gov/8361965
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5
Polymyxin-B suppresses endotoxin-induced insulin hypersecretion in pancreatic islets
Autor: M R, Yelich, C K, Schieber, D M, Umporowicz, J P, Filkins
Publikováno v: Circulatory shock. 38(2)
Endotoxin induces insulin hypersecretion in vivo, which results in hyperinsulinemia and glucose dyshomeostasis. Polymyxin-B (PMX-B), an inhibitor of protein kinase C (PKC), has been shown to ameliorate the consequences of endotoxin-induced hyperinsul
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::aba80f4ac86ac2bd87c47d9f3bc80793
https://pubmed.ncbi.nlm.nih.gov/1423925
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6
Insulin-inhibiting effects of epinephrine are blunted during endotoxicosis in the rat
Autor: M R, Yelich, D M, Umporowicz, M, Qi, S B, Jones
Publikováno v: Circulatory shock. 35(3)
This study was conducted to examine the conditions under which epinephrine (EPI) blocks glucose-induced hyperinsulinemia in normal and endotoxic rats. Nutritional state (fasting vs. feeding) and anesthetic state (conscious vs. sodium pentobarbital an
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::03b09322952098483178f45fcb878981
https://pubmed.ncbi.nlm.nih.gov/1777952
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7
Glucoregulatory, hormonal, and metabolic responses to endotoxicosis or cecal ligation and puncture sepsis in the rat: a direct comparison
Autor: M R, Yelich
Publikováno v: Circulatory shock. 31(3)
This study characterized the cecal ligation and puncture (CLP) model of sepsis and the bolus endotoxin model of sepsis in rats with regard to specific hormonal, metabolic, and glucoregulatory changes which occur during the early, compensatory phases
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::6156c6d21127461dd00e6061eb1ed7e2
https://pubmed.ncbi.nlm.nih.gov/2192817
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8
Mechanism of hyperinsulinemia in endotoxicosis
Autor: J. P. Filkins, M. R. Yelich
Publikováno v: American Journal of Physiology-Endocrinology and Metabolism. 239:E156-E161
Hyperinsulinemia subsequent to endotoxemia is a key factor leading to the disturbance of glucose homeostasis in endotoxin shock. This study investigated the mechanism(s) of hyperinsulinemia during endotoxicosis in the rat. Two primary mechanisms for
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5014e2bb949c72231bf8d75d39ed7525
https://doi.org/10.1152/ajpendo.1980.239.2.e156
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10
Mechanism of hyperinsulinemia after reticuloendothelial system phagocytosis
Autor: J. P. Filkins, M. R. Yelich
Publikováno v: The American journal of physiology. 242(2)
Endocytic loading of the reticuloendothelial system (RES) results in acute hyperinsulinemia and functional hyperinsulinism. Colloidal carbon blockade of the RES in rats resulted in elevations of both portal vein and systemic serum immunoreactive insu
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9dcbbf7e406c384007a295a8ed62c920
https://pubmed.ncbi.nlm.nih.gov/7039336
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