Zobrazeno 1 - 10
of 141
pro vyhledávání: '"M Memmi"'
Autor:
A Trancuccio, A Mazzanti, D Kukavica, G Giannini, M Memmi, P Gambelli, R Bloise, M Marino, M Morini, M Ortiz-Genga, C Napolitano, S G Priori
Publikováno v:
European Heart Journal. 43
Background Recently, a novel genetic-based classification for Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT) has been proposed to distinguish “typical” (RYR2 and CASQ2 genes) and “atypical” (TRDN, TECRL, CALM1–3, RYR2 loss-of-
Autor:
D Kukavica, A Mazzanti, A Trancuccio, G Giannini, M Marino, M Memmi, P Gambelli, R Bloise, M Morini, M Ortiz-Genga, C Napolitano, S G Priori
Publikováno v:
European Heart Journal. 43
Background Due to the catecholaminergic nature of arrhythmias that define the Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT), beta-blockers (BBs) represent the first line therapy for this severe arrhythmogenic syndrome. Despite optimal
Autor:
A Mazzanti, A Trancuccio, D Kukavica, M Marino, G Giannini, L Rossetti, M Memmi, R Bloise, M Morini, C Napolitano, S G Priori
Publikováno v:
European Heart Journal. 43
Background Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT) is a potentially fatal genetic arrhythmia syndrome and, despite optimal beta-blocker (BB) therapy, patients often experience life-threatening arrhythmic events (LAE). The role of
Publikováno v:
TP50. TP050 COVID: NONPULMONARY CRITICAL CARE, MECHANICAL VENTILATION, BEHAVIORAL SCIENCES, AND EPI.
Autor:
M Marino, A Trancuccio, Carlo Napolitano, Nicola Monteforte, M Memmi, Raffaella Bloise, Andrea Mazzanti, L Braghieri, D Kukavica, M Morini, S. G. Priori
Publikováno v:
European Heart Journal. 41
Background Type 1 Long QT Syndrome (LQT1) is an arrhythmogenic disorder, caused by loss-of-function mutations on KCNQ1 gene, coding for Kv7.1 potassium channel. Although LQT1 is described as the most benign form of LQTS, patients still experience arr
Autor:
Carlo Napolitano, R Bongianino, M Memmi, R Yanfei, Nicola Monteforte, Nian Liu, Raffaella Bloise, A Malovini, Andrea Mazzanti, M Morini, S. G. Priori, Valeria Novelli
Publikováno v:
European Heart Journal. 41
Background Inconsistent data support the role of CACNA1C as a disease-causing gene responsible for Brugada syndrome (BrS). As of today, the only gene consistently linked with BrS is SCN5A. Several CACNA1c genetic variants have been reported in associ
Background: Andersen-Tawil Syndrome type 1 (ATS1) is a rare arrhythmogenic disorder, caused by loss-of-function mutations in the KCNJ2 gene. We present here the largest cohort of patients with ATS1 with outcome data reported. Objectives: This study s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______2127::f55892104a5dfbf04862f5bd56760acd
https://pergamos.lib.uoa.gr/uoa/dl/object/uoadl:3104559
https://pergamos.lib.uoa.gr/uoa/dl/object/uoadl:3104559
Autor:
E Tenuta, Carlo Napolitano, Andrea Mazzanti, Antonio Curcio, M Morini, Eleonora Pagan, Nicola Monteforte, S. G. Priori, Raffaella Bloise, M Memmi, Vincenzo Bagnardi, M Marino
Publikováno v:
European Heart Journal. 40
Background Quinidine at high-dose is used in patients with Brugada Syndrome (BrS), but its efficacy to prevent life-threatening arrhythmic events (LAE) in BrS is unproven and its use is limited by side effects. Objective We assessed whether low-dose
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This chapter provides an overview of sexually transmitted infections (STIs) associated with bacterial agents, namely, syphilis, gonorrhea, chlamydia, and Mycoplasma genitalium infection, and a genital parasite responsible for trichomoniasis. Each yea
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::ce07883a7fb37a91847402648f472780
https://doi.org/10.1016/b978-0-12-800886-7.00013-3
https://doi.org/10.1016/b978-0-12-800886-7.00013-3