Výsledky vyhledávání - "M L Alvarez Gonzalez"

Neuroendocrine mechanism of onset of puberty. Sequential reduction in activity of inhibitory and facilitatory N-methyl-D-aspartate receptors

Autor: M L Alvarez Gonzalez, Paul Franchimont, Arlette Gerard, J P Bourguignon

Publikováno v: Journal of Clinical Investigation. 90:1736-1744

In humans and in several animal species, puberty results from changes in pulsatile gonadotropin-releasing hormone (GnRH) secretion in the hypothalamus. In particular, the frequency of pulsatile GnRH secretion increases at the onset of puberty, as can

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::98cda2f595f7b3e056a6296d17bb0838
https://doi.org/10.1172/jci116047

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Mixed origin of neovascularization of human endometrial grafts in immunodeficient mouse models

Autor: Christine Galant, Etienne Marbaix, Michelle Nisolle, M. L. Alvarez Gonzalez, Aude Beliard, Francis Frankenne, Jean-Michel Foidart

Publikováno v: Human reproduction (Oxford, England). 24(9)

In vivo mouse models have been developed to study the physiology of normal and pathologic endometrium. Although angiogenesis is known to play an important role in endometrial physiology and pathology, the origin of neovasculature in xenografts remain

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b9a5b96ea57ea4f5ce31779e0b83489e
https://pubmed.ncbi.nlm.nih.gov/19508999

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Development of an animal experimental model to study the effects of levonorgestrel on the human endometrium

Autor: Jean-Michel Foidart, Michelle Nisolle, Christine Galant, Etienne Marbaix, Francis Frankenne, Soraya Labied, Aude Beliard, M. L. Alvarez Gonzalez

Publikováno v: Human Reproduction, Vol. 24, no. 3, p. 697-704 (2009)
Human Reproduction (Oxford, England)

BACKGROUND This study was designed to develop an animal model to test the response of endometrium to local progestin delivery. METHODS Proliferative human endometrium was subcutaneously grafted in two groups of SCID mice that received, 2 days before,

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc49110dfe87b16cd051f46c1fa02391
https://hdl.handle.net/2078.1/35769

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Endogenous glutamate involvement in pulsatile secretion of gonadotropin-releasing hormone: evidence from effect of glutamine and developmental changes

Autor: Arlette Gerard, Gentiane Purnelle, Paul Franchimont, J P Bourguignon, M L Alvarez Gonzalez

Publikováno v: Endocrinology. 136(3)

The secretion of GnRH can be stimulated by glutamate (GLU) and GLU agonists, whereas GLU receptor antagonists inhibit GnRH. Using 6-diazo-5-oxo-L-norleucine (DON), an inhibitor of glutaminase, we aimed to study the involvement of endogenous GLU in Gn

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a4fbe6a4ae4f5c6211c71862216e0cd6
https://pubmed.ncbi.nlm.nih.gov/7867599

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Effects of changes in nutritional conditions on timing of puberty: clinical evidence from adopted children and experimental studies in the male rat

Autor: L. Fawe, Paul Franchimont, Arlette Gerard, M L Alvarez Gonzalez, J P Bourguignon

Publikováno v: Hormone research. 38

Among 32 patients with idiopathic central precocious puberty seen during a 3-year period, 1/4 were adopted children from developing countries who showed early sexual maturation during the catch-up process following their arrival in Belgium. To study

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::97af819f9c25d55232936c0b9cd0d54f
https://pubmed.ncbi.nlm.nih.gov/1295822

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INHIBITION OF GnRH SECRETION BY IGF-I DEGRADATION INTO A SUB-PRODUCT ANTAGONIST ATNMDA RECEPTORS: AN EFFECT DEVELOPING AFTER ONSET OF PUBERTY

Autor: Arlette Gerard, J P Bourguignon, Paul Franchimont, M L Alvarez Gonzalez

Publikováno v: Pediatric Research. 33:S29-S29

INHIBITION OF GnRH SECRETION BY IGF-I DEGRADATION INTO A SUB-PRODUCT ANTAGONIST ATNMDA RECEPTORS: AN EFFECT DEVELOPING AFTER ONSET OF PUBERTY

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::e56095d74a0a41c23181b8fb622ffb8d
https://doi.org/10.1203/00006450-199305001-00154

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