Zobrazeno 1 - 10
of 107
pro vyhledávání: '"M G, Shubich"'
Autor:
M D, Perova, M G, Shubich
Publikováno v:
Morfologiia (Saint Petersburg, Russia). 139(3)
The purpose of the present review was to analyze the accumulating evidence regarding recently discovered novel defense mechanism of neutrophils - capacity to form neutrophil extracellular traps (NETs). Contact with pathogenic microbes and/or exposure
Publikováno v:
Arkhiv patologii. 72(2)
Publikováno v:
Stomatologiia. 86(5)
Publikováno v:
Stomatologiia. 86(3)
Publikováno v:
Morfologiia (Saint Petersburg, Russia). 131(3)
This review paper describes the evolution of the ideas of periodontal tissue regeneration, starting with the concept of selective cellular repopulation, stimulation of resident precursor cells in the regeneration focus, up to the application of versa
Autor:
M D, Perova, M G, Shubich
Publikováno v:
Arkhiv patologii. 68(5)
The paper gives new information on the molecular mechanisms of pathogen reception in the human organism. The leading role is ascribed to the toll-like receptors recognizing the molecular structures of pathogens for bringing into operation natural res
Autor:
M G, Avdeeva, M G, Shubich
Publikováno v:
Klinicheskaia laboratornaia diagnostika. (6)
Autor:
V B, Karpiuk, M G, Shubich
Publikováno v:
Zhurnal voprosy neirokhirurgii imeni N. N. Burdenko. (2)
Subarachnoidal hemorrhage (SAH) causes cerebrovascular endothelial dysfunction that leads to impaired nitrosidergic vasorelaxation, delayed vasospasm, and cerebral ischemia. The purpose of the present review is to study the current potentialities of
Autor:
B G, Ermoshenko, M G, Shubich
Publikováno v:
Morfologiia (Saint Petersburg, Russia). 117(2)
Preparation for labour and its induction are controlled by an integrative but not single process. The primary stimulus that comprises this integrative hormonal prostaglandin-leukocytic mechanism is a suppression of local progesterone action with furt
Publikováno v:
Zhurnal voprosy neirokhirurgii imeni N. N. Burdenko. (3)
Subarachnoidal hemorrhage leads to impaired endothelium-dependent relaxation by inhibiting the synthesis of major vascular relaxation agent nitric oxide (NO). Nitrite, a closest product of the oxidation of NO, is responsible for the synthesis of the