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pro vyhledávání: '"M C, Herzig"'
Publikováno v:
ResearcherID
A number of pathological changes have been reported in relation to CA1 pyramidal cells in Alzheimer's disease (AD), among them hyperphosphorylation of tau protein followed by the formation of filamentous tau lesions, granulovacuolar degeneration (GVD
Publikováno v:
Advances in experimental medicine and biology. 487
Autor:
Martina Stalder, Christine Sturchler-Pierrat, Sonia Boncristiano, Michelle Pfeifer, M C Herzig, Luca Bondolfi, A. Probst, Matthias Staufenbiel, M. E. Calhoun, David T. Winkler, Thomas Deller, A. Phinney, M Tolnay, Mathias Jucker, Karl-Heinz Wiederhold, Bernd Sommer, Dorothee Abramowski
Publikováno v:
Neurodegenerative Disorders: Loss of Function Through Gain of Function ISBN: 9783642074486
Transgenic mice that overexpress mutant human amyloid precursor protein (APP) exhibit one hallmark of Alzheimer’s disease pathology, namely the extracellular deposition of amyloid in plaques and vessels. In an effort to study the impact of cerebral
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::02d55ca4998561003987bec07afd1276
https://doi.org/10.1007/978-3-662-04399-8_6
https://doi.org/10.1007/978-3-662-04399-8_6
Publikováno v:
The Journal of biological chemistry. 269(42)
Several B2 bradykinin (BK) receptor-specific antagonists including HOE140, NPC17731, and NPC567 exhibited negative intrinsic activity, which was observed as a decrease in basal phosphoinositide hydrolysis in primary cultures of rat myometrial cells,
Publikováno v:
Europe PubMed Central
Second messenger calcium responses to the neuropeptide neurotensin and its non-peptide antagonist SR 48692 were studied in relation to the proliferation of pancreatic cancer cells. Neurotensin caused a transient increase in intracellular calcium in t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::a0ef7f95558252c2ac940849b710d776
http://europepmc.org/abstract/med/10226545
http://europepmc.org/abstract/med/10226545