Zobrazeno 1 - 10
of 232
pro vyhledávání: '"M, Seccia"'
Autor:
Gian Paolo Rossi, Giacomo Rossitto, Chiarastella Maifredini, Agata Barchitta, Andrea Bettella, Raffaele Latella, Luisa Ruzza, Beatrice Sabini, Teresa M. Seccia
Publikováno v:
Blood Pressure, Vol 30, Iss 4, Pp 208-219 (2021)
Background: Acute increases of high blood pressure values, usually described as ‘hypertensive crises’, ‘hypertensive urgencies’ or ‘hypertensive emergencies’, are common causes of patients’ presentation to emergency departments. Owing t
Externí odkaz:
https://doaj.org/article/848344c27579479084a1c957dc2e6ec5
Publikováno v:
Experimental and Clinical Endocrinology & Diabetes.
Drug-resistant arterial hypertension (RH) is a major risk factor for cardiovascular disease, often due to overlooked underlying causes. Identification of such causes poses significant clinical challenges. In this setting, primary aldosteronism (PA) i
Autor:
M. Piazza, N. M. J. Hanssen, J. L. J. M. Scheijen, M. vd Waarenburg, B. Caroccia, T. M. Seccia, C. D. A. Stehouwer, G. P. Rossi, C. G. Schalkwijk
Publikováno v:
Journal of Human Hypertension. Nature Publishing Group
Journal of human hypertension. Nature Publishing Group
Journal of human hypertension. Nature Publishing Group
Patients with an aldosterone-producing adenoma (APA) carry a higher risk of cardiovascular disease and commonly have high levels of autoantibodies (AT1AA) that may activate the angiotensin II type 1 receptor (AT1R). AT1R activation is linked to an in
Autor:
Teresa M. Seccia, Brasilina Caroccia, Francesca Gioco, Maria Piazza, Valentina Buccella, Diego Guidolin, Eugenia Guerzoni, Barbara Montini, Lucia Petrelli, Elisa Pagnin, Verdiana Ravarotto, Anna S. Belloni, Lorenzo A. Calò, Gian Paolo Rossi
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 5, Iss 7 (2016)
BackgroundTubulointerstitial fibrosis, the final outcome of most kidney diseases, involves activation of epithelial mesenchymal transition (EMT). Endothelin‐1 (ET‐1) activates EMT in cancer cells, but it is not known whether it drives EMT in the
Externí odkaz:
https://doaj.org/article/ff41e15239f54c0faafbd589989fa82f
Adrenal venous sampling is the gold standard test to identify surgically curable primary aldosteronism, but it is markedly underused in clinical practice being perceived as a technically challenging and invasive procedure and, moreover, as difficult
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::cbfce0b548776cb537e95722ab95c23f
http://hdl.handle.net/11577/3323854
http://hdl.handle.net/11577/3323854
Publikováno v:
Frontiers in Integrative Neuroscience
Autor:
Robert D. Madoff, Alan G. Thorson, M. Seccia, Ann C. Lowry, N. S. Williams, J. Christiansen, R. J. Nicholls, K. E. Matzel, P. A. Lehur, C. G.M.I. Baeten, W. D. Wong, H. R. Rosen, Steven D. Wexner, David Z. Lubowski, J. A. Heine
Publikováno v:
Diseases of the Colon & Rectum. 43:135-141
PURPOSE: Anal sphincter replacement offers a new treatment option for patients with severe refractory fecal incontinence or for those who require abdominoperineal resection for localized malignancy. The purpose of this study was to review the current
Publikováno v:
Free Radical Biology and Medicine. 25:519-528
The kinetics of Cu(II) reduction and its relationship to the process of low density lipoprotein (LDL) oxidation were investigated in isolated human LDL incubated with CuSO4 by using the Cu(I) chelator and indicator dye bathocuproine disulfonate (BC).
Publikováno v:
Clinical Chemistry. 43:1436-1441
Isolated human LDL, used in the native form or supplemented with α-tocopherol (αT), were oxidized with Cu2+, 2,2′-azobis-(2-amidino propane) hydrochloride (AAPH), and H2O2 plus horseradish peroxidase (HRP). The oxidation kinetics were measured sp
Publikováno v:
Biochemical and Biophysical Research Communications. 232:613-617
NO and NO-donors are able to inhibit the peroxidation of polyunsaturated fatty acids in human low-density lipoproteins (LDL) exposed to Cu+2. Here we report that 1-hydroxy-2-oxo-3,3-bis(3-aminoethyl)-1-triazene (NOC-18), a compound which releases NO