Zobrazeno 1 - 10
of 126
pro vyhledávání: '"M, Panesar"'
Akademický článek
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Akademický článek
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Autor:
Pia Isomäki, M Panesar, Joanna M. Clark, Andrew P. Cope, Alexander Annenkov, Yuti Chernajovsky
Publikováno v:
Proceedings of the National Academy of Sciences. 101:1696-1701
Expression and function of the antigen T cell receptor (TCR) play a central role in regulating immune responsiveness. Accordingly, targeting the expression of TCRalphabeta or its associated CD3 subunits profoundly influences T cell development and ad
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::afb7e53f95d448973945c1bd1d781f90
https://doi.org/10.1073/pnas.0308231100
https://doi.org/10.1073/pnas.0308231100
Autor:
Brian M. J. Foxwell, Alex Annenkov, Yuti Chernajovsky, Andrew P. Cope, Joanna M. Clark, M Panesar, Pia Isomäki
Publikováno v:
The Journal of Immunology. 166:5495-5507
A role for TNF-α in the pathogenesis of chronic inflammatory disease is now firmly established. Paradoxically, TNF also has potent immunomodulatory effects on CD4+ T lymphocytes, because Ag-specific proliferative and cytokine responses are suppresse
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c170e56f21656698bc5044c0f10f596
https://doi.org/10.4049/jimmunol.166.9.5495
https://doi.org/10.4049/jimmunol.166.9.5495
Publikováno v:
The Journal of Immunology. 159:3584-3594
In rat membranous nephropathy, C5b-9 induces glomerular epithelial cell (GEC) injury and proteinuria, which is partially mediated by eicosanoids. In cultured rat GEC, sublytic C5b-9 injures plasma membranes and releases arachidonic acid (AA) and eico
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6438940a843e4cf187bb74954eae6e55
https://doi.org/10.4049/jimmunol.159.7.3584
https://doi.org/10.4049/jimmunol.159.7.3584
Autor:
D. J. Wilson, Deven Patel, M. Panesar, Nicolas A. Chronos, Kim M. Fox, Alison H. Goodall, Charles Knight
Publikováno v:
Circulation. 95:125-132
Background The three major classes of antianginal drug all inhibit platelet aggregation at high concentrations in vitro, but detecting clinically relevant effects has proved to be more difficult. We used whole-blood flow cytometry, a sensitive method
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::98a85844df30bfbbdc1100312e2c99ea
https://doi.org/10.1161/01.cir.95.1.125
https://doi.org/10.1161/01.cir.95.1.125
Publikováno v:
Annals of the rheumatic diseases. 64
Members of the tumour necrosis factor (TNF)/TNF-receptor (TNF-R) superfamily coordinate the immune response at multiple levels. For example, TNF, LTalpha, LTbeta and RANKL provide signals required for lymphoid neogenesis, CD27, OX-40, 4-1BB and CD30
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::097e052e6c342b948e113533648b7104
https://pubmed.ncbi.nlm.nih.gov/16239393
https://pubmed.ncbi.nlm.nih.gov/16239393
Publikováno v:
Current drug targets. Inflammation and allergy. 4(3)
Immune and inflammatory responses are governed by antigen-specific T cells, whose activation, differentiation and effector function are induced by signals delivered via the T cell antigen receptor (TCR) and by costimulatory and cytokine receptors. Th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e059f5fb7870fe80533cadc3fed2ba7f
https://pubmed.ncbi.nlm.nih.gov/16101535
https://pubmed.ncbi.nlm.nih.gov/16101535
Akademický článek
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Autor:
K, Walker, M, Bowes, M, Panesar, A, Davis, C, Gentry, A, Kesingland, F, Gasparini, W, Spooren, N, Stoehr, A, Pagano, P J, Flor, I, Vranesic, K, Lingenhoehl, E C, Johnson, M, Varney, L, Urban, R, Kuhn
Publikováno v:
Neuropharmacology. 40(1)
The excitatory neurotransmitter, glutamate, is particularly important in the transmission of pain information in the nervous system through the activation of ionotropic and metabotropic glutamate receptors. A potent, subtype-selective antagonist of t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::dfff50a6dc69a60b0455381926bdc841
https://pubmed.ncbi.nlm.nih.gov/11077065
https://pubmed.ncbi.nlm.nih.gov/11077065