Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Lvshuang Liao"'
Autor:
Shuchao Wang, Lvshuang Liao, Yanxia Huang, Mi Wang, Hongkang Zhou, Dan Chen, Fengxia Liu, Dan Ji, Xiaobo Xia, Bing Jiang, Jufang Huang, Kun Xiong
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 13 (2019)
In our previous study, we reported that peptidyl-prolyl isomerase 1 (Pin1)-modulated regulated necrosis (RN) occurred in cultured retinal neurons after glutamate injury. In the current study, we investigated the role of calcium/calmodulin-dependent p
Externí odkaz:
https://doaj.org/article/67eeb5bd8abc4a78adccbd75e67dbbc5
Autor:
Xue Yang, Yong Wang, Qiyan Li, Yaxian Zhong, Liangpei Chen, Yajun Du, Jing He, Lvshuang Liao, Kun Xiong, Chun-xia Yi, Jie Yan
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 11 (2018)
Methamphetamine (METH) is a popular new-type psychostimulant drug with complicated neurotoxicity. In spite of mounting evidence on METH-induced damage of neural cell, the accurate mechanism of toxic effect of the drug on central nervous system (CNS)
Externí odkaz:
https://doaj.org/article/de5f5150f2874230a911346c9fd8b7b4
Autor:
Shuchao Wang, Lvshuang Liao, Mi Wang, Hongkang Zhou, Yanxia Huang, Zhen Wang, Dan Chen, Dan Ji, Xiaobo Xia, Yong Wang, Fengxia Liu, Jufang Huang, Kun Xiong
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 11 (2018)
The purpose of the current study was to investigate whether peptidyl-prolyl cis/trans isomerase NIMA-interacting 1 (Pin1) can interact with calpastatin (CAST) and regulate CAST/calpain2, under excessive glutamate conditions, and subsequently regulate
Externí odkaz:
https://doaj.org/article/4737ddfd3bc6480ebab2d243884a3fda
Autor:
Shuchao Wang, Tu Hu, Zhen Wang, Na Li, Lihong Zhou, Lvshuang Liao, Mi Wang, Libin Liao, Hui Wang, Leping Zeng, Chunling Fan, Hongkang Zhou, Kun Xiong, Jufang Huang, Dan Chen
Publikováno v:
PLoS ONE, Vol 12, Iss 9, p e0185388 (2017)
Many studies on retinal injury and repair following elevated intraocular pressure suggest that the survival ratio of retinal neurons has been improved by various measures. However, the visual function recovery is far lower than expected. The homeosta
Externí odkaz:
https://doaj.org/article/e9c7ab7214b844b5b6b074c490863ae0
Publikováno v:
Acta Biochimica et Biophysica Sinica. 53:636-639
Autor:
Shuchao Wang, Kun Xiong, Bin Jiang, Yanxia Huang, Lvshuang Liao, Xiaobo Xia, Li-min Guo, Lei Shang, Fengxia Liu, Dan Ji, Jufang Huang, Mi Wang, Dan Chen, Hao Wan
Publikováno v:
J Anat
Receptor‐interacting protein 3 (RIP3) plays an important role in the necroptosis signaling pathway. Our previous studies have shown that the RIP3/mixed lineage kinase domain‐like protein (MLKL)‐mediated necroptosis occurs in retinal ganglion ce
Publikováno v:
SSRN Electronic Journal.
Autor:
Dan Chen, Xiaobo Xia, Mi Wang, Hao Wan, Bing Jiang, Dan Ji, Bo Qin, Kun Xiong, Weitao Yan, Lvshuang Liao, Yanxia Huang, Qi Zhang, Fengxia Liu, Shuchao Wang, Fei Huang, Jufang Huang
Publikováno v:
Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft. 235
Cellular FLICE-inhibitory protein (c-FLIP), an anti-apoptotic regulator, shows remarkable similarities to caspase-8, which plays a key role in the cleavage of gasdermin D (GSDMD). It has been reported that the oxygen-glucose deprivation/recovery (OGD
Publikováno v:
Acta Biochimica et Biophysica Sinica. 49:128-137
Necroptosis is a type of regulated cell death that has been implicated in various diseases. Receptor-interacting protein 3 (RIP3), a member of the RIP family, is an important mediator of the necroptotic pathway. Cleavage of RIP3 at Asp328 by caspase-
Autor:
Shuchao Wang, Lvshuang Liao, Yanxia Huang, Mi Wang, Hongkang Zhou, Dan Chen, Fengxia Liu, Dan Ji, Xiaobo Xia, Bing Jiang, Jufang Huang, Kun Xiong
Publikováno v:
Frontiers in Cellular Neuroscience
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Frontiers in Cellular Neuroscience, Vol 13 (2019)
In our previous study, we reported that peptidyl-prolyl isomerase 1 (Pin1)-modulated regulated necrosis (RN) occurred in cultured retinal neurons after glutamate injury. In the current study, we investigated the role of calcium/calmodulin-dependent p