Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Luz Graciela Cervantes-Pérez"'
Autor:
Diana L. Silva-Velasco, Jesus H. Beltran-Ornelas, Jorge Tapia-Martínez, Araceli Sánchez-López, Saúl Huerta de la Cruz, Luz Graciela Cervantes-Pérez, Leonardo del Valle-Mondragón, Alicia Sánchez-Mendoza, David Centurión
Publikováno v:
Peptides. 164:171001
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::10fa570ebc615d3989ab0e2b3e8b5288
https://doi.org/10.1016/j.peptides.2023.171001
https://doi.org/10.1016/j.peptides.2023.171001
Autor:
Victoria Ramirez, Gustavo Pastelín-Hernández, Luz Ibarra-Lara, María Sánchez-Aguilar, Alicia Sánchez-Mendoza, Luz Graciela Cervantes-Pérez, David Centurión, Vicente Castrejón-Téllez, Fabiola Cortes-Lopez, Leonardo Del Valle-Mondragón, Elizabeth Soria-Castro, Wylly Ramsés García-Niño, Araceli Sánchez-López
Publikováno v:
PPAR Research, Vol 2021 (2021)
PPAR Research
PPAR Research
Lesions caused by high glucose (HG), hypoxia/reperfusion (H/R), and the coexistence of both conditions in cardiomyocytes are linked to an overproduction of reactive oxygen species (ROS), causing irreversible damage to macromolecules in the cardiomyoc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::22d0bc80cd5984bea6128b7c58cdf003
https://doi.org/10.1155/2021/8895376
https://doi.org/10.1155/2021/8895376
Autor:
Elizabeth Soria-Castro, María Sánchez-Aguilar, Alicia Sánchez-Mendoza, Luz Graciela Cervantes-Pérez, Luz Ibarra-Lara, L. del Valle-Mondragón, Gustavo Pastelín-Hernández
Publikováno v:
Journal of Pharmacological Sciences, Vol 144, Iss 4, Pp 218-228 (2020)
Myocardial ischemia initiates a chain of pathological conditions leading to cardiomyocyte death. Therefore, pharmacological treatment to stop ischemia-induced damage is necessary. Fibrates, have been reported to decrease inflammatory markers and to m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f289b7c6a922792b4292f8b766a3d559
http://www.sciencedirect.com/science/article/pii/S1347861320300955
http://www.sciencedirect.com/science/article/pii/S1347861320300955
Autor:
Ashline Paul Manoucheca Amilcar, Lorena Rosas‐Martínez, Juan Carlos Torres‐Narváez, Araceli Sánchez‐López, Luz Graciela Cervantes‐Pérez, David Centurión, Alicia Sánchez‐Mendoza
Publikováno v:
The FASEB Journal. 36
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e68647b432cd0ab5a1593b32b9b9ab5d
https://doi.org/10.1096/fasebj.2022.36.s1.r3132
https://doi.org/10.1096/fasebj.2022.36.s1.r3132
Autor:
Rafael Rodríguez-Muñoz, Alicia Sánchez-Mendoza, Luz Graciela Cervantes-Pérez, Maria del Carmen Namorado-Tónix, Lorena Rosas-Martínez, Fanis Missirlis, Leonardo Del Valle-Mondragón, José Luis Reyes-Sánchez
Publikováno v:
Life Sciences. 268:119003
Aims This study attempts to elicit whether the level of hyperglycemia in an early stage of diabetic nephropathy changes the renal expression of claudins-2 and -5 and to determine the involvement of glucose-induced oxidative stress. Main methods Strep
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67381efda041607a49a16038421acd9e
https://doi.org/10.1016/j.lfs.2020.119003
https://doi.org/10.1016/j.lfs.2020.119003
Autor:
María Sánchez-Aguilar, Elizabeth Soria-Castro, Víctor Hugo Oidor-Chan, Alicia Sánchez-Mendoza, Gustavo Pastelín-Hernández, Luz Graciela Cervantes-Pérez, Juan Carlos Torres-Narváez, Leonardo Del Valle-Mondragón, Gabriela Zarco-Olvera, Margarita Ramirez-Ortega, Francisca Pérez-Severiano, Luz Ibarra-Lara
Publikováno v:
Pharmacological Reports. 68:692-702
Background Arterial high blood pressure is a risk factor for target organ damage; the most susceptible organs are the arteries, brain, kidneys, and heart. The damage mechanisms include oxidative stress and renin-angiotensin system (RAS) overactivity.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7b455bfd3ba0972d4410cdd18114501f
https://doi.org/10.1016/j.pharep.2016.03.002
https://doi.org/10.1016/j.pharep.2016.03.002
Autor:
Alicia Sánchez-Mendoza, Víctor Hugo Oidor-Chan, Luz Graciela Cervantes-Pérez, Elizabeth Soria, Gustavo Pastelín-Hernández, Juan Carlos Torres-Narváez, Leonardo Del Valle-Mondragón, María de la Luz Ibarra-Lara, Margarita Ramirez-Ortega, Francisca Pérez-Severiano, María Sánchez-Aguilar
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 94:634-642
Myocardial infarction (MI) has been associated with an inflammatory response and a rise in TNF-α, interleukin (IL)-1β, and IL-6. Peroxisome proliferator-activated receptors (PPARs) promote a decreased expression of inflammatory molecules. We aimed
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6816781d9a0544cf899230f49a51515a
https://doi.org/10.1139/cjpp-2015-0356
https://doi.org/10.1139/cjpp-2015-0356
Autor:
Alicia Sánchez-Mendoza, Luz Graciela Cervantes-Pérez, Elizabeth Soria-Castro, María Sánchez-Aguilar, Luz Ibarra-Lara, Francisco Javier Roldán, Jesús Vargas-Barrón, Gustavo Pastelín-Hernández, Juan Carlos Torres-Narváez, Natalia Pavón
Publikováno v:
Molecules
Volume 24
Issue 2
Molecules, Vol 24, Iss 2, p 270 (2019)
Volume 24
Issue 2
Molecules, Vol 24, Iss 2, p 270 (2019)
Myocardial infarction (MI) initiates an inflammatory response that promotes both beneficial and deleterious effects. The early response helps the myocardium to remove damaged tissue
however, a prolonged later response brings cardiac remodeling c
however, a prolonged later response brings cardiac remodeling c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::25a813dc085e17b8df6a6edfc63ea3ff
https://doi.org/10.3390/molecules24020270
https://doi.org/10.3390/molecules24020270
Autor:
Alicia Sánchez-Mendoza, Luz Graciela Cervantes-Pérez, Elizabeth Soria-Castro, Gustavo Pastelín-Hernández, Juan Carlos Torres-Narváez, Margarita Ramirez-Ortega, Francisca Pérez-Severiano, Enrique Hong, Leonardo Del Valle-Mondragón, Luz Ibarra-Lara
Publikováno v:
Journal of Cardiovascular Pharmacology. 60:323-334
Peroxisome proliferator-activated receptors (PPAR) play a critical physiological role in energy homeostasis, in inflammation, and a protective role in cardiovascular function. We assessed the antioxidant effect of clofibrate-induced Peroxisome prolif
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bdd1e90e126511796755b098ef376981
https://doi.org/10.1097/fjc.0b013e31826216ed
https://doi.org/10.1097/fjc.0b013e31826216ed
Autor:
Francisca Pérez-Severiano, María de la Luz Ibarra-Lara, María Alicia Sánchez-Mendoza, Hilda Vargas-Robles, Luz Graciela Cervantes-Pérez, Gustavo Pastelín, Bruno Escalante, Leonardo Del Valle-Mondragón
Publikováno v:
European Journal of Pharmacology. 685:108-115
Adequate production of nitric oxide (NO) by endothelial nitric oxide synthase (eNOS) requires eNOS coupling promoted by tetrahydrobiopterin (BH(4)). Under pathological conditions such as hypertension, BH(4) is diminished, avoiding eNOS coupling. When
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ad6dcb1f8e76c20dcf64f745ae86820
https://doi.org/10.1016/j.ejphar.2012.04.006
https://doi.org/10.1016/j.ejphar.2012.04.006