Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Lupus Erythematosus, Systemic/genetics/immunology"'
Autor:
Liza Ho, Frédéric Lajaunias, Carolin Dix, Eduardo Martinez-Soria, Lars Nitschke, Shozo Izui, Shuichi Kikuchi, Thomas Moll, R. Michael E. Parkhouse, Marie-Laure Santiago-Raber
Publikováno v:
International Immunology, Vol. 18, No 1 (2006) pp. 59-68
CD22 functions primarily as a negative regulator of B-cell receptor signaling. The Cd22a allele has been proposed as a candidate allele for murine systemic lupus erythematosus. In this study, we explored the possible expression of aberrant forms of C
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::89cf9c09265b624ad0fea203752ae567
http://doc.rero.ch/record/290776/files/dxh349.pdf
http://doc.rero.ch/record/290776/files/dxh349.pdf
Autor:
Shizuo Akira, Marie-Laure Santiago-Raber, Satoshi Uematsu, Shozo Izui, Brian L. Kotzin, Paula Borel, Shuichi Kikuchi
Publikováno v:
Journal of Immunology, Vol. 181, No 2 (2008) pp. 1556-1562
The accelerated development of systemic lupus erythematosus (SLE) in male BXSB mice is associated with the genetic abnormality in its Y chromosome, designated Yaa (Y-linked autoimmune acceleration). Recently, the Yaa mutation was identified to be a t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1cb8f6b4516a95761da0fbf49192e149
https://pubmed.ncbi.nlm.nih.gov/18606711
https://pubmed.ncbi.nlm.nih.gov/18606711
Autor:
L Gabriel, Shozo Izui, S. Jane Rose, Bernard J Morley, Nicola J. Rogers, Michelle E. K. Haywood
Publikováno v:
Journal of Immunology, Vol. 179, No 4 (2007) pp. 2428-2434
The BXSB strain of recombinant inbred mice develops a spontaneous pathology that closely resembles the human disease systemic lupus erythematosus. Six non-MHC loci, Yaa, Bxs1–4, and Bxs6, have been linked to the development of aspects of the diseas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d45afda2323fb51a5c0b9fdf55fb005
https://archive-ouverte.unige.ch/unige:11294
https://archive-ouverte.unige.ch/unige:11294
Autor:
Liliane Fossati-Jimack, Marie-Laure Santiago-Raber, Shozo Izui, Hirofumi Amano, Stephen J. Rozzo, Brian L. Kotzin, Nabila Ibnou-Zekri, Shuichi Kikuchi, Catherine Laporte, Akinori Ida, Thomas Moll, Eri Amano
Publikováno v:
Journal of Immunology, Vol. 174, No 2 (2005) pp. 1111-1117
By assessing the development of Y-linked autoimmune acceleration (Yaa) gene-induced systemic lupus erythematosus in C57BL/6 (B6) × (New Zealand Black (NZB) × B6.Yaa)F1 backcross male mice, we mapped three major susceptibility loci derived from the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3d1f70313579a5d8d372a105519d1d75
https://archive-ouverte.unige.ch/unige:11375
https://archive-ouverte.unige.ch/unige:11375
Autor:
Ramón Merino, Masahiro Iwamoto, Pedro Muniesa, Satoru Takahashi, Kimi Araki, Shozo Izui, Joaquin Huarte, Jean-Dominique Vassalli, Ken-ichi Yamamura, Liliane Fossati
Publikováno v:
Journal of Experimental Medicine, Vol. 178, No 4 (1993) pp. 1189-1197
The Journal of Experimental Medicine
The Journal of Experimental Medicine
Males from the BXSB murine strain (H-2b) spontaneously develop an autoimmune syndrome with features of systemic lupus erythematosus (SLE), which results in part from the action of a mutant gene (Yaa) located on the Y chromosome. Like other H-2b mice,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::db18e721aed206e41d8b530e6558528f
https://archive-ouverte.unige.ch/unige:8895
https://archive-ouverte.unige.ch/unige:8895
Publikováno v:
Journal of Immunology, Vol. 138, No 12 (1987) pp. 4222-4228
We have treated autoimmune-prone (NZW x BXSB)F1 hybrid mice with polyclonal rabbit anti-mouse IgM antibodies starting from birth to define conditions leading to quantitative and functional elimination of the B cell compartment and to determine the ef
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b0ab4a821fff52bed225ea5ca3a0790b
https://pubmed.ncbi.nlm.nih.gov/3495583
https://pubmed.ncbi.nlm.nih.gov/3495583