Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Lulu K Callies"'
Autor:
Claire J Watson, W Joyce Tang, Maria F Rojas, Imke A K Fiedler, Ernesto Morfin Montes de Oca, Andrea R Cronrath, Lulu K Callies, Avery Angell Swearer, Ali R Ahmed, Visali Sethuraman, Sumaya Addish, Gist H Farr, Arianna Ericka Gómez, Jyoti Rai, Adrian T Monstad-Rios, Edith M Gardiner, David Karasik, Lisa Maves, Bjorn Busse, Yi-Hsiang Hsu, Ronald Young Kwon
Publikováno v:
PLoS Genetics, Vol 18, Iss 11, p e1010496 (2022)
Bone and muscle are coupled through developmental, mechanical, paracrine, and autocrine signals. Genetic variants at the CPED1-WNT16 locus are dually associated with bone- and muscle-related traits. While Wnt16 is necessary for bone mass and strength
Externí odkaz:
https://doaj.org/article/5c1e0f2009e24bea8e2128b9e43705bc
Publikováno v:
Cell reports. 41(4)
A fundamental step in regeneration is rapid growth to replace lost tissue. Cells must generate sufficient lipids, nucleotides, and proteins to fuel rapid cell division. To define metabolic pathways underlying regenerative growth, we undertake a multi
Publikováno v:
SSRN Electronic Journal.
Autor:
Claire J. Watson, W. Joyce Tang, Maria F. Rojas, Imke A.K. Fiedler, Ernesto Morfin Montes de Oca, Andrea R. Cronrath, Lulu K. Callies, Avery Angell Swearer, Ali R. Ahmed, Visali Sethuraman, Sumaya Addish, Gist H. Farr, Arianna E. Gomez, Jyoti Rai, Adrian T. Monstad-Rios, Edith M. Gardiner, David Karasik, Lisa Maves, Bjorn Busse, Yi-Hsiang Hsu, Ronald Young Kwon
Bone and muscle interact through developmental, mechanical, paracrine, and autocrine signals. Genetic variants at the CPED1-WNT16 locus are dually associated with bone- and muscle-related traits. While Wnt16 is necessary for bone mass and strength, t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::58cef41a6044d6d8325b1fc36c87fad2
https://doi.org/10.1101/2021.08.12.456120
https://doi.org/10.1101/2021.08.12.456120
Publikováno v:
J Biol Chem
Elucidation of gene function by reverse genetics in animal models frequently is complicated by the functional redundancy of homologous genes. This obstacle often is compounded by the tight clustering of homologous genes, which precludes the generatio