Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Lujun Dai"'
Autor:
Wei Jiang, Chun Gan, Xindi Zhou, Qing Yang, Dan Chen, Han Xiao, Lujun Dai, Yaxi Chen, Mo Wang, Haiping Yang, Qiu Li
Publikováno v:
Cardiovascular Diabetology, Vol 22, Iss 1, Pp 1-18 (2023)
Abstract Objective Diabetic kidney disease (DKD) is characterized by the abnormal deposition of oxidized low-density lipoprotein (ox-LDL), which contributes to podocyte damage. Klotho, an aging suppressor that plays a critical role in protecting podo
Externí odkaz:
https://doaj.org/article/c542de13aa634a7cb5723d32a589f1dc
Autor:
Lifen Xu, Yuxia Zhou, Guifang Wang, Li Bo, Bangming Jin, Lujun Dai, Qinli Lu, Xueni Cai, Laying Hu, Lu Liu, Yixuan Wu, Xuebing Chang, Yali Huang, Lingyu Song, Tian Zhang, Yuanyuan Wang, Ying Xiao, Fan Zhang, Lingling Liu, Mingjun Shi, Tuanlao Wang, Bing Guo
Publikováno v:
Cell Death and Disease, Vol 14, Iss 2, Pp 1-15 (2023)
Abstract Impaired protein N-glycosylation leads to the endoplasmic reticulum (ER) stress, which triggers adaptive survival or maladaptive apoptosis in renal tubules in diabetic kidney disease (DKD). Therapeutic strategies targeting ER stress are prom
Externí odkaz:
https://doaj.org/article/6669613ac7a74b6394f92ab4f6be98bb
Autor:
Lifen Xu, Yuxia Zhou, Guifang Wang, Li Bo, Bangming Jin, Lujun Dai, Qinli Lu, Xueni Cai, Laying Hu, Lu Liu, Yixuan Wu, Xuebing Chang, Yali Huang, Lingyu Song, Tian Zhang, Yuanyuan Wang, Ying Xiao, Fan Zhang, Lingling Liu, Mingjun Shi, Tuanlao Wang, Bing Guo
Impaired protein N-glycosylation leads to the endoplasmic reticulum (ER) stress, which triggers adaptive survival or maladaptive apoptosis in renal tubules in diabetic kidney disease (DKD). Therapeutic strategies targeting ER stress are promising for
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d7783ef186fa5e57190bcdadbb403aa5
https://doi.org/10.21203/rs.3.rs-2016503/v1
https://doi.org/10.21203/rs.3.rs-2016503/v1
Autor:
Bo Wei, Hanbing Zhao, Bailong Hu, Lujun Dai, Guoning Zhang, Lili Mo, Niwen Huang, Changchao Zou, Bei Zhang, Haiyan Zhou, Wei Li, Xingde Liu
Publikováno v:
BioMed Research International.
Purpose. To investigate the detailed mechanism of 3-iodothyronamine (T1AM) in cell apoptosis and programmed necrosis of hypoxia/reoxygenation- (H/R-) induced H9C2 injury. Materials and Methods. Cardiomyocyte H9C2 cells were cultured in vitro for the
Publikováno v:
Journal of Clinical Laboratory Analysis
Background Current therapeutic drugs show positive effects on non–small‐cell lung cancer (NSCLC) patients with mutant epidermal growth factor receptor (EGFR) expression, whereas a lesser beneficial effect is generally noted on NSCLC patients with
Publikováno v:
Chemico-biological interactions. 316
The incidence and mortality of lung cancer are the highest among cancer-related deaths. However, the long-term use of currently available cytotoxic drugs can increase genetic alterations in cancer cells and cause drug-resistance, which significantly
Autor:
Lingfei Wu, Qingqing Wu, Guanyou Huang, Wei Liu, Xiaoling Zeng, Yongli Huang, Congrong Zhou, Shuyun Zhao, Hua Zhou, Lujun Dai
Publikováno v:
Experimental and Therapeutic Medicine
We aimed to investigate the expression of suppressors cytokine signaling (SOCS)-3, transforming growth factor (TGF)-β and indoleamine 2,3-dioxygense (IDO) and to analyse the relationship of SOCS3 and TGF-β with IDO expression in early pregnancy cho
Autor:
WEI LIU, YONGLI HUANG, GUANYOU HUANG, CONGRONG ZHOU, XIAOLING ZENG, SHUYUN ZHAO, LINGFEI WU, HUA ZHOU, QINGQING WU, LUJUN DAI
Publikováno v:
Experimental & Therapeutic Medicine; Nov2017, Vol. 14 Issue 5, p4817-4824, 8p