Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Luisa Escobar-Hoyos"'
Autor:
Danielle J. Fassler, Shahira Abousamra, Rajarsi Gupta, Chao Chen, Maozheng Zhao, David Paredes, Syeda Areeha Batool, Beatrice S. Knudsen, Luisa Escobar-Hoyos, Kenneth R. Shroyer, Dimitris Samaras, Tahsin Kurc, Joel Saltz
Publikováno v:
Diagnostic Pathology, Vol 15, Iss 1, Pp 1-7 (2020)
An amendment to this paper has been published and can be accessed via the original article.
Externí odkaz:
https://doaj.org/article/b1284fda257a4de3ab41908619895d68
Autor:
Danielle J. Fassler, Shahira Abousamra, Rajarsi Gupta, Chao Chen, Maozheng Zhao, David Paredes, Syeda Areeha Batool, Beatrice S. Knudsen, Luisa Escobar-Hoyos, Kenneth R. Shroyer, Dimitris Samaras, Tahsin Kurc, Joel Saltz
Publikováno v:
Diagnostic Pathology, Vol 15, Iss 1, Pp 1-11 (2020)
Abstract Background Multiplex immunohistochemistry (mIHC) permits the labeling of six or more distinct cell types within a single histologic tissue section. The classification of each cell type requires detection of the unique colored chromogens loca
Externí odkaz:
https://doaj.org/article/a8dfefeb428f4e0894cb68134bf3b1c4
Autor:
Yushan Zhang, Md Afjalus Siraj, Prabir Chakraborty, Robert Tseng, Li-Ting Ku, Shamik Das, Anindya Dey, Shailendra Kumar Dhar Dwivedi, Geeta Rao, Min Zhang, Da Yang, Md Nazir Hossen, Wei-Qun Ding, Kar-Ming Fung, Resham Bhattacharya, Luisa Escobar-Hoyos, Priyabrata Mukherjee
Many cancers carry change-of-function mutations affecting RNA splicing factors, however, less is known about the functional consequences of upregulated RNA splicing factors in cancer. Here, we demonstrate that SMNDC1, a poorly studied splicing factor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7e99ba625b426de1662b2f6546ce0540
https://doi.org/10.1101/2022.08.31.505957
https://doi.org/10.1101/2022.08.31.505957
Publikováno v:
The FASEB Journal. 36
Publikováno v:
Biophysical Journal. 122:27a
Autor:
Yushan Zhang, Md Afjalus Siraj, Prabir Chakrabarty, Robert Tseng, Shamik Das, Resham Bhattacharya, Luisa Escobar-Hoyos, Priyabrata Mukherjee
Publikováno v:
Cancer Research. 82:B062-B062
Many cancers carry recurrent, change-of-function mutations affecting RNA splicing factors, however, less is known about the consequences of upregulated RNA splicing factors (SFs) in cancer. Here, we describe a role of survival motor neuron domain con
Abstract A055: Altered RNA splicing causes pancreatic cancer and exposes a therapeutic vulnerability
Autor:
Natasha Pinto Medici, Diana Martinez Saucedo, Danny Lee, Li-Ting Ku, Robert Tseng, Vincent Cannataro, Deanne Yugawa, Sumedha Chowdhury, Jeffrey Townsend, Christine A. Iacobuzio-Donahue, Omar Abdel-Wahab, Steven D. Leach, Luisa Escobar-Hoyos
Publikováno v:
Cancer Research. 82:A055-A055
With the aim to identify new second-hit mutations that cause pancreatic ductal adenocarcinoma (PDAC), other than mutations in p53, we performed a genetic interaction analysis based on the concept of mutually exclusive mutations, which suggests that m
Abstract B045: Evaluating the role of altered RNA metabolism in pancreatic cancer therapy resistance
Publikováno v:
Cancer Research. 82:B045-B045
Pancreatic ductal adenocarcinomas (PDACs) are highly lethal, mostly because they quickly develop resistance to current chemotherapies: Gemcitabine (GEM)/paclitaxel or cocktail FOLFIRINOX. Both chemotherapies rely on nucleoside analogues (GEM and 5-fl
Autor:
Elias Quijano, Diana Martinez Saucedo, Minsoo Khang, Yanfeng Liu, Dale Ludwig, Bruce C. Turner, Stephen Squinto, Ranjit Bindra, W. Mark Saltzman, Luisa Escobar-Hoyos, Peter M. Glazer
Publikováno v:
Cancer Research. 82:663-663
There is intense interest in the development of nucleic acid ligands for immune stimulation of the tumor microenvironment via pattern recognition receptors (PRRs), particularly for the treatment of “cold” tumors. However, delivery of these ligand
Autor:
Luisa Escobar Hoyos, Omar Abdel-Wahab
Publikováno v:
Cancer Cell. 34:181-183
In this issue of Cancer Cell, Kahles et al. perform a comprehensive analysis of RNA splicing across cancer types and identify novel correlations between genetic alterations and splicing in cancer. In addition, they identify that tumor-specific splici