Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Luisa, Vernizzi"'
Autor:
Stefania Santarelli, Chiara Londero, Alessia Soldano, Carlotta Candelaresi, Leonardo Todeschini, Luisa Vernizzi, Paola Bellosta
Publikováno v:
Frontiers in Neuroscience, Vol 17 (2023)
Proteinopathies are a large group of neurodegenerative diseases caused by both genetic and sporadic mutations in particular genes which can lead to alterations of the protein structure and to the formation of aggregates, especially toxic for neurons.
Externí odkaz:
https://doaj.org/article/0ca74545f77645b299efe7a2110cd2b4
Autor:
Zeynep Kabakci, Hiro Yamada, Luisa Vernizzi, Samir Gupta, Joe Weber, Michael Shoujie Sun, Christian F Lehner
Publikováno v:
PLoS Genetics, Vol 18, Iss 10, p e1010469 (2022)
Meiosis in males of higher dipterans is achiasmate. In their spermatocytes, pairing of homologs into bivalent chromosomes does not include synaptonemal complex and crossover formation. While crossovers preserve homolog conjunction until anaphase I du
Externí odkaz:
https://doaj.org/article/84f3425d83454884854f8bb06d842dbb
Autor:
Luisa Vernizzi, Christian F Lehner
Publikováno v:
PLoS Genetics, Vol 18, Iss 7, p e1010327 (2022)
The bivalent chromosomes that are generated during prophase of meiosis I comprise a pair of homologous chromosomes. Homolog pairing during prophase I must include mechanisms that avoid or eliminate entanglements between non-homologous chromosomes. In
Externí odkaz:
https://doaj.org/article/131b6a9746334ba8b18c9998cb1aea5b
Autor:
Luisa Vernizzi, Christian F Lehner
Publikováno v:
PLoS Genetics, Vol 17, Iss 10, p e1009870 (2021)
Reduction of genome ploidy from diploid to haploid necessitates stable pairing of homologous chromosomes into bivalents before the start of the first meiotic division. Importantly, this chromosome pairing must avoid interlocking of non-homologous chr
Externí odkaz:
https://doaj.org/article/a8d1b51ede404cec8e80e3e624b6a942
Autor:
Luisa Vernizzi, Chiara Paiardi, Giusimaria Licata, Teresa Vitali, Stefania Santarelli, Martino Raneli, Vera Manelli, Manuela Rizzetto, Mariarosa Gioria, Maria E. Pasini, Daniela Grifoni, Maria A. Vanoni, Cinzia Gellera, Franco Taroni, Paola Bellosta
Publikováno v:
Cells, Vol 9, Iss 1, p 196 (2020)
Glutamine Synthetase 1 (GS1) is a key enzyme that catalyzes the ATP-dependent synthesis of l-glutamine from l-glutamate and is also member of the Glutamate Glutamine Cycle, a complex physiological process between glia and neurons that controls glutam
Externí odkaz:
https://doaj.org/article/b0c23f7ef2ed4f918a4713db0e37249d
Autor:
Martino Raneli, Stefania Santarelli, Paola Bellosta, G. Licata, Maria A. Vanoni, Chiara Paiardi, Cinzia Gellera, Daniela Grifoni, Maria Enrica Pasini, Luisa Vernizzi, Mariarosa Gioria, Vera Manelli, Manuela Rizzetto, Teresa Vitali, Franco Taroni
Publikováno v:
Cells
Volume 9
Issue 1
Cells, Vol 9, Iss 1, p 196 (2020)
Volume 9
Issue 1
Cells, Vol 9, Iss 1, p 196 (2020)
Glutamine Synthetase 1 (GS1) is a key enzyme that catalyzes the ATP-dependent synthesis of l-glutamine from l-glutamate and is also member of the Glutamate Glutamine Cycle, a complex physiological process between glia and neurons that controls glutam
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b5eee7ae8eea859b611655067a904b6
http://hdl.handle.net/11572/248388
http://hdl.handle.net/11572/248388
Autor:
Luisa, Vernizzi, Chiara, Paiardi, Giusimaria, Licata, Teresa, Vitali, Stefania, Santarelli, Martino, Raneli, Vera, Manelli, Manuela, Rizzetto, Mariarosa, Gioria, Maria E, Pasini, Daniela, Grifoni, Maria A, Vanoni, Cinzia, Gellera, Franco, Taroni, Paola, Bellosta
Publikováno v:
Cells
Glutamine Synthetase 1 (GS1) is a key enzyme that catalyzes the ATP-dependent synthesis of l-glutamine from l-glutamate and is also member of the Glutamate Glutamine Cycle, a complex physiological process between glia and neurons that controls glutam
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::6f5edb913bd3b686ef59318718367887
https://pubmed.ncbi.nlm.nih.gov/32098205
https://pubmed.ncbi.nlm.nih.gov/32098205
Autor:
Manuela Rizzetto, Teresa Vitali, Paola Bellosta, Alice Valenza, Maria A. Vanoni, Chiara Paiardi, Franco Taroni, Maria Enrica Pasini, G. Licata, Luisa Vernizzi, Cinzia Gellera, Caterina Mariotti
Publikováno v:
Journal of Neurology, Neurosurgery & Psychiatry. 87:A21.2-A21
Background Neuronal death is often caused by an excess of glutamate that is maintained at physiological level by a non-autonomous cycle between glia and neurons called the glutamate-glutamine cycle. Aims It is possible to manipulate the activity of t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::536f12c70a6d9cfa8617272ded53bdf5
https://doi.org/10.1136/jnnp-2016-314597.66
https://doi.org/10.1136/jnnp-2016-314597.66