Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Lucila Cassino"'
Autor:
Rita Reynoso, Jorge Quarleri, Natalia Laufer, Lucila Cassino, Carolina Torres, Federico Bolcic
Publikováno v:
Antiviral Research. 95:72-81
The rate of non-response to pegylated interferon plus ribavirin (peg-IFN+RBV) in HCV/HIV coinfected patients is higher than in HCV-monoinfected patients. In this sense, the contribution of HCV genetic variability is unknown. The 5' untranslated (5'UT
Autor:
Natalia Laufer, Carolina Torres, Jorge Quarleri, Lucila Cassino, Viviana Andrea Mbayed, Roberto Campos
Publikováno v:
Journal of medical virology. 84(4)
HIV infection has a significant impact on the natural progression of liver disease caused by infection with hepatitis B virus (HBV), but its role in the molecular evolution of HBV is unknown. It is difficult to study the molecular evolution of HBV lo
Autor:
Martin P. Vazquez, H. Tanno, Gaston Westergaard, Mariano Miguel Sede, Jorge Quarleri, F. Fay, S. Benetti, Diego Sebastian Ojeda, Lucila Cassino
Publikováno v:
Antiviral research. 94(2)
The aim of this study was to analyze the spectrum and dynamics of low-prevalent HBV mutations in the reverse transcriptase (rt) and S antigen by ultra-deep pyrosequencing (UDPS). Samples were obtained from a chronically infected patient who was follo
Publikováno v:
CONICET Digital (CONICET)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
BMC Infectious Diseases
BMC Infectious Diseases, Vol 11, Iss 1, p 178 (2011)
Consejo Nacional de Investigaciones Científicas y Técnicas
instacron:CONICET
BMC Infectious Diseases
BMC Infectious Diseases, Vol 11, Iss 1, p 178 (2011)
Background Complex mutants can be selected under sequential selective pressure by HBV therapy. To determine hepatitis B virus genomic evolution during antiviral therapy we characterized the HBV quasi-species in a patient who did no respond to therapy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4c520757eaf56da9ac1a1bec597e8df8
http://bmcinfectdis.biomedcentral.com/articles/10.1186/1471-2334-11-178
http://bmcinfectdis.biomedcentral.com/articles/10.1186/1471-2334-11-178
Publikováno v:
AIDS research and human retroviruses. 27(5)
The presence of HBV genomes with deletions at the basal core promoter (BCP) is associated with more aggressive liver disease. This 3-year longitudinal analysis of two HIV-HBV-coinfected patients allowed identification of three deletions with dissimil
Publikováno v:
Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology. 46(4)
Background The course of chronic HBV infection is modified by HIV-coexistence. Objective To analyze the role of HBV genomic heterogeneity in basal core promoter (BCP) and precore (Pc) genomic regions. Study design In a 3-yr prospective study, 39 HBV
Autor:
Natalia Laufer, Lucila Cassino, Federico Bolcic, Franco Moretti, Rita Reynoso, Maria Belen Bouzas, Pedro Cahn, Horacio Salomón, Jorge Quarleri
Publikováno v:
Journal of acquired immune deficiency syndromes (1999). 49(2)