Zobrazeno 1 - 5
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pro vyhledávání: '"Lucas Jagemann"'
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-8 (2024)
Abstract Aquaporins (AQPs) are a family of water permeable channels expressed on the plasma membrane with AQP5 being the major channel expressed in several human tissues including salivary and lacrimal glands. Anti-AQP5 autoantibodies have been obser
Externí odkaz:
https://doaj.org/article/409131c0b540484e9187b5e2b8fe2020
Autor:
Stefan Lang, Isabelle V. Leefa, Isabella Artner, Sara Nolbrant, Nils Offen, Marcus Winkler, Sebastian Braun, Ulrike A. Nuber, Falk Hertwig, Lucas Jagemann, Gaëlle Prost, Kenichi Miharada
Publikováno v:
Oncotarget
Bmi1 was originally identified as a gene that contributes to the development of mouse lymphoma by inhibiting MYC-induced apoptosis through repression of Ink4a and Arf. It codes for the Polycomb group protein BMI-1 and acts primarily as a transcriptio
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1845
Ectopic lymphoid structure (ELS) can form in the target tissues of patients with chronic inflammatory autoimmune diseases such as rheumatoid arthritis (RA) and Sjögren's syndrome (SS). Although it is still not clear why ELS form only in a subset of
Autor:
Elisa, Corsiero, Lucas, Jagemann, Mauro, Perretti, Costantino, Pitzalis, Michele, Bombardieri
Publikováno v:
The Journal of Immunology Author Choice
Rheumatoid arthritis (RA) is characterized by formation of synovial ectopic lymphoid structures (ELS) supporting B cell autoreactivity toward locally generated citrullinated (cit) antigens, including those contained in neutrophil extracellular traps
Autor:
Emanuela Monni, Olle Lindvall, Vladimer Darsalia, James Wood, Karthikeyan Devaraju, Zaal Kokaia, Henrik Ahlenius, Lucas Jagemann, Robert E. Iosif, Olof Torper, Somsak Wattananit, Sebastian Braun, Sten Eirik W. Jacobsen, Ulrike A. Nuber, Elisabet Englund, Koichi Oki
Publikováno v:
Journal of Neuroscience
Journal of Neuroscience; Vol 32
The Journal of Neuroscience
Journal of Neuroscience; Vol 32
The Journal of Neuroscience
Ischemic stroke causes transient increase of neural stem and progenitor cell (NSPC) proliferation in the subventricular zone (SVZ), and migration of newly formed neuroblasts toward the damaged area where they mature to striatal neurons. The molecular