Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Lucas C, Olson"'
Autor:
Kohei Togami, Whitney Wolf, Lucas C. Olson, Madison Card, Limei Shen, Alison Schaefer, Kenichi Okuda, Larry Zeitlin, Michael Pauly, Kevin Whaley, Raymond J. Pickles, Samuel K. Lai
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
Effective treatment and immunoprophylaxis of viral respiratory infections with neutralizing monoclonal antibodies (mAbs) require maintaining inhibitory concentrations of mAbs at the airway surface. While engineered mAbs with increased affinity to the
Externí odkaz:
https://doaj.org/article/1ce0586831914fc7af05bae7e4c1ab5f
Autor:
Kayla M. Scott, D. Joshua Cohen, Dane W. Nielson, Gloria Kim, Lucas C. Olson, Michael J. McClure, Mark W. Grinstaff, Barbara D. Boyan, Zvi Schwartz
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-10 (2022)
Abstract Transfection of chondrocytes with microRNA-451(miR-451), present in growth zone cartilage of the growth plate, upregulates production of enzymes association with extracellular matrix degradation. miR-451 is also present in articular cartilag
Externí odkaz:
https://doaj.org/article/608830619bb44b6d9f9ec9afd83cba45
Autor:
Lucas C. Olson, James T. Redden, LaStar Gilliam, Tri M. Nguyen, Josephina A. Vossen, David J. Cohen, Zvi Schwartz, Michael J. McClure
Publikováno v:
Bioengineering, Vol 9, Iss 9, p 426 (2022)
Volumetric muscle loss (VML) is the acute loss of muscle mass due to trauma. Such injuries occur primarily in the extremities and are debilitating, as there is no clinical treatment to restore muscle function. Pro-inflammatory advanced glycation end-
Externí odkaz:
https://doaj.org/article/3ad6c3b77a304d0abca733b9aec8a4fd
Publikováno v:
Bioengineering, Vol 8, Iss 11, p 168 (2021)
Advanced age causes skeletal muscle to undergo deleterious changes including muscle atrophy, fast-to-slow muscle fiber transition, and an increase in collagenous material that culminates in the age-dependent muscle wasting disease known as sarcopenia
Externí odkaz:
https://doaj.org/article/e2d4a466b8d940f4bebd22c16eed01a3
Autor:
D Joshua, Cohen, Christoph H, Lohmann, Kayla M, Scott, Lucas C, Olson, Barbara D, Boyan, Zvi, Schwartz
Publikováno v:
J Bone Joint Surg Am
Bisphosphonates limit resorption by inhibiting osteoclast formation and activation. They are removed during preparation of demineralized bone matrix (DBM) particles, but it is not known if osteogenesis and incorporation of mineralized bone allografts
Autor:
Michael J. McClure, Lucas C. Olson, David J. Cohen, Yen Chen Huang, Shirley Zhang, Tri Nguyen, Barbara D. Boyan, Zvi Schwartz
Publikováno v:
Bioengineering, Vol 8, Iss 1, p 12 (2021)
Products developed for skeletal muscle regeneration frequently incorporate allogeneic and xenogeneic materials to elicit a regenerative response to heal skeletal muscle wounds. To avoid graft rejection in preclinical studies, immunodeficient rodents
Externí odkaz:
https://doaj.org/article/a5f8f4ed99b7466cbdb2fcd4334c5f50
Autor:
Lucas C. Olson, Zvi Schwartz, Barbara D. Boyan, Michael J. McClure, Tri Nguyen, Rebecca L. Heise
Publikováno v:
International Journal of Molecular Sciences
Volume 22
Issue 16
International Journal of Molecular Sciences, Vol 22, Iss 8832, p 8832 (2021)
Volume 22
Issue 16
International Journal of Molecular Sciences, Vol 22, Iss 8832, p 8832 (2021)
Decellularized tissues are biocompatible materials that engraft well, but the age of their source has not been explored for clinical translation. Advanced glycation end products (AGEs) are chemical cross-links that accrue on skeletal muscle collagen
Autor:
Zvi Schwartz, Shirley Zhang, Michael J. McClure, Tri Nguyen, Yen Chen Huang, Lucas C. Olson, David J. Cohen, Barbara D. Boyan
Publikováno v:
Bioengineering
Volume 8
Issue 1
Bioengineering, Vol 8, Iss 12, p 12 (2021)
Volume 8
Issue 1
Bioengineering, Vol 8, Iss 12, p 12 (2021)
Products developed for skeletal muscle regeneration frequently incorporate allogeneic and xenogeneic materials to elicit a regenerative response to heal skeletal muscle wounds. To avoid graft rejection in preclinical studies, immunodeficient rodents