Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Luca Tommasini"'
Autor:
Davide Maria Cammisuli, Cristina Pagni, Giovanni Palermo, Daniela Frosini, Joyce Bonaccorsi, Claudia Radicchi, Simona Cintoli, Luca Tommasini, Gloria Tognoni, Roberto Ceravolo, Ubaldo Bonuccelli
Publikováno v:
Frontiers in Psychology, Vol 12 (2021)
Background: In the present study, we aimed to better investigate attention system profile of Parkinson's disease-Mild Cognitive Impairment (PD-MCI) patients and to determine if specific attentional deficits are associated with 123I-FP-CIT SPECT.Metho
Externí odkaz:
https://doaj.org/article/f942c1f992614ddbae14934268e5e0a6
Autor:
Gabriele Siciliano, Luca Tommasini, Ubaldo Bonuccelli, Filippo Baldacci, Gloria Tognoni, Elisabetta Belli, Giovanni Palermo, Valentina Nicoletti, Riccardo Morganti, Daniela Frosini, Roberto Ceravolo
Publikováno v:
Journal of Alzheimer's Disease. 84:757-766
Background: Dementia with Lewy bodies (DLB) and Parkinson’s disease dementia (PDD) are differentiated by the time of onset of cognitive and motor symptoms (‘1-year rule’). We explored the neuropsychological continuum of DLB and PDD subjects wit
Autor:
Sonia Mazzucchi, Gloria Tognoni, Roberto Ceravolo, Riccardo Morganti, Eleonora Del Prete, Daniela Frosini, Giovanni Palermo, Luca Tommasini, Cristina Pagni, Ubaldo Bonuccelli
Publikováno v:
Neurological Sciences. 42:1483-1490
Progressive supranuclear palsy (PSP) is an atypical Parkinsonism characterized by motor and neuropsycological disorders. Language could be impaired in PSP patients, also in Richardson variant (PSP-RS). The analysis of connected speech is used in neur
Autor:
Gabriele Siciliano, Eleonora Del Prete, Luca Tommasini, Roberto Ceravolo, Filippo Baldacci, Giovanni Palermo
Publikováno v:
Movement Disorders
Autor:
Giovanni Palermo, Luca Tommasini, Martina Giuntini, Gayane Aghakhanyan, Gloria Tognoni, Ubaldo Bonuccelli, Duccio Volterrani, Daniela Frosini, Roberto Ceravolo
Publikováno v:
Journal of Alzheimer's disease : JAD. 70(2)
BACKGROUND Dementia in Parkinson's disease (PDD) is common presumably due to combined neuropathological substrates. Amyloid-β (Aβ) plaques are well described in PDD but their contribution in synucleinopathies is still controversial. OBJECTIVE To in