Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Luc Forichon"'
Autor:
Malik Lutzmann, Florence Bernex, Cindy da Costa de Jesus, Dana Hodroj, Caroline Marty, Isabelle Plo, William Vainchenker, Marie Tosolini, Luc Forichon, Caroline Bret, Sophie Queille, Candice Marchive, Jean-Sébastien Hoffmann, Marcel Méchali
Publikováno v:
Cell Reports, Vol 28, Iss 11, Pp 2851-2865.e4 (2019)
Summary: Hematopoiesis is particularly sensitive to DNA damage. Myeloid tumor incidence increases in patients with DNA repair defects and after chemotherapy. It is not known why hematopoietic cells are highly vulnerable to DNA damage. Addressing this
Externí odkaz:
https://doaj.org/article/1b5c45032647429590e52fb8e732a408
Autor:
Jean-Sébastien Hoffmann, Marie Tosolini, Cindy da Costa de Jesus, Malik Lutzmann, Caroline Marty, Dana Hodroj, Candice Marchive, Marcel Méchali, Caroline Bret, William Vainchenker, Isabelle Plo, Luc Forichon, Florence Bernex, Sophie Queille
Publikováno v:
Cell Reports
Cell Reports, 2019, 28 (11), pp.2851-2865. ⟨10.1016/j.celrep.2019.07.095⟩
Cell Reports, Elsevier Inc, 2019, 28 (11), pp.2851-2865. ⟨10.1016/j.celrep.2019.07.095⟩
Cell Reports, Vol 28, Iss 11, Pp 2851-2865.e4 (2019)
Cell Reports, 2019, 28 (11), pp.2851-2865. ⟨10.1016/j.celrep.2019.07.095⟩
Cell Reports, Elsevier Inc, 2019, 28 (11), pp.2851-2865. ⟨10.1016/j.celrep.2019.07.095⟩
Cell Reports, Vol 28, Iss 11, Pp 2851-2865.e4 (2019)
Summary: Hematopoiesis is particularly sensitive to DNA damage. Myeloid tumor incidence increases in patients with DNA repair defects and after chemotherapy. It is not known why hematopoietic cells are highly vulnerable to DNA damage. Addressing this
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::366bfa15cc97041ac2ba3ff5538324ad
https://doi.org/10.1016/j.celrep.2019.07.095
https://doi.org/10.1016/j.celrep.2019.07.095
Autor:
Giuseppe Gangarossa, Dominique Françon, Benoit Lerat, Philippe Lory, Jean Chemin, Sophie Sakkaki, Luc Forichon, Emmanuel Valjent, Mireille Lerner-Natoli
Publikováno v:
Neuropharmacology
Neuropharmacology, Elsevier, 2016, 101, pp.320--9. ⟨10.1016/j.neuropharm.2015.09.032⟩
Neuropharmacology, Elsevier, 2016, 101, pp.320--9. ⟨10.1016/j.neuropharm.2015.09.032⟩
T-type (Cav3) calcium channels play important roles in neuronal excitability, both in normal and pathological activities of the brain. In particular, they contribute to hyper-excitability disorders such as epilepsy. Here we have characterized the ant
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::42b3e02c69c4d16b58f6c9beacbad3cf
https://hal.archives-ouvertes.fr/hal-01936765
https://hal.archives-ouvertes.fr/hal-01936765
Autor:
Malik Lutzmann, Apolinar Maya-Mendoza, Noemie Ranisavljevic, Marcel Méchali, Luc Forichon, Olivier Ganier, Sabine Traver, Elodie Gavois, Nathalie Montel, Florence Bernex, Bernard de Massy, Atsuya Nishiyama, Corinne Grey
Publikováno v:
Molecular Cell
Molecular Cell, Elsevier, 2012, 47 (4), pp.523-534. ⟨10.1016/j.molcel.2012.05.048⟩
Molecular Cell, Elsevier, 2012, 47 (4), pp.523-534. ⟨10.1016/j.molcel.2012.05.048⟩
Summary We generated knockout mice for MCM8 and MCM9 and show that deficiency for these genes impairs homologous recombination (HR)-mediated DNA repair during gametogenesis and somatic cells cycles. MCM8 −/− mice are sterile because spermatocytes
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dd87469702dc9654deb869f5408c666b
https://hal.archives-ouvertes.fr/hal-00733290
https://hal.archives-ouvertes.fr/hal-00733290