Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Lourdes A. Vega Rasgado"'
Publikováno v:
AIMS Neuroscience, Vol 7, Iss 3, Pp 344-359 (2020)
Senile plaques, a hallmark of Alzheimer's disease, are composed by Amyloid-Beta (Aβ). Aβ 25-35 toxicity is caused mainly by increasing reactive oxygen species (ROS), which is reversed by albumin preventing Aβ internalization. In addition, key cell
Externí odkaz:
https://doaj.org/article/14830f3772ce4c82a1806ed80e4d311a
Autor:
Lourdes A. Vega Rasgado, Eva Ramón-Gallegos, Lorena Rodríguez-Páez, Verónica Alcántara-Farfán
Publikováno v:
Acta Pharmaceutica
Volume 73
Issue 1
Volume 73
Issue 1
Nitric oxide (NO) participates in processes such as endothelium-dependent vasodilation and neurotransmission/neuromodulation. The role of NO in epilepsy is controversial, attributing it to anticonvulsant but also proconvulsant properties. Clarificati
Publikováno v:
Acta Phlebologica. 23
Publikováno v:
AIMS Neuroscience, Vol 7, Iss 3, Pp 344-359 (2020)
AIMS Neuroscience
AIMS Neuroscience
Senile plaques, a hallmark of Alzheimer's disease, are composed by Amyloid-Beta (Aβ). Aβ 25-35 toxicity is caused mainly by increasing reactive oxygen species (ROS), which is reversed by albumin preventing Aβ internalization. In addition, key cell
Publikováno v:
Neurochemical Journal. 14:90-100
Amyloid-β (Aβ) is the main component of senile plaques, one of the hallmarks of Alzheimer,s disease. Is been shown that Aβ25–35 decreased neuronal viability while it increased generation of reactive oxygen species (ROS), and albumin (BSA) preven
Autor:
Héctor Carrizosa, Lourdes A. Vega-Rasgado, Eugenio Jiménez, Felipe Rendon, Blanca V. Pastrana, Fernando Vega-Rasgado, Carlos Arreola, Fernando Contreras, J. Fernando Torres
Publikováno v:
Acta Phlebologica. 20
Publikováno v:
Acta Pharmaceutica, Vol 67, Iss 2, Pp 215-226 (2017)
Acta Pharmaceutica
Volume 67
Issue 2
Acta Pharmaceutica
Volume 67
Issue 2
γ-Ethyl-γ-phenyl-butyrolactone (EFBL) is a structural combination of the anticonvulsant γ-hydroxy-γ-ethyl-γ-phenylbutyramide (HEPB) and the hypnotic γ-butyrolactone (GBL), which inherits both properties. To clarify its mechanism of action, the
Publikováno v:
Acta Pharmaceutica, Vol 65, Iss 4, Pp 443-452 (2015)
Glutamate (Glu) is a major excitatory neurotransmitter involved in epilepsy. Glu is synthesized by glutamate dehydrogenase (GDH, E.C. 1.4.1.3) and dysfunction of the enzymatic activity of GDH is associated with brain pathologies. The main goal of thi
Publikováno v:
ISRN Pharmacology
Glutamate dehydrogenase (GDH, E.C. 1.4.1.3.) is a key enzyme for the biosynthesis and modulation of glutamate (GLU) metabolism and an indirectγ-aminobutyric acid (GABA) source, here we studied the effect of anticonvulsants such as pyridoxal phosphat
Publikováno v:
Acta Pharmaceutica
Volume 65
Issue 4
Volume 65
Issue 4
Glutamate (Glu) is a major excitatory neurotransmitter involved in epilepsy. Glu is synthesized by glutamate dehydrogenase (GDH, E.C. 1.4.1.3) and dysfunction of the enzymatic activity of GDH is associated with brain pathologies. The main goal of thi