Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Lori J. Manzel"'
Autor:
Cecilia L. Chin, Katherine J. D. A. Excoffon, Alicia L. Humlicek, Dwight C. Look, Lori J. Manzel, Lei Shi, D. Michael Shasby, Michael C. Winter
Publikováno v:
The Journal of Immunology. 178:6395-6403
Respiratory pathogens and toxins often assault the lung from the airway lumen. Airway epithelia may initiate and amplify inflammation in response to these attacks, but under certain conditions confinement of inflammation to the airway lumen may be be
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bddc34b432247732d08f9a853a24f433
https://doi.org/10.4049/jimmunol.178.10.6395
https://doi.org/10.4049/jimmunol.178.10.6395
Publikováno v:
American journal of respiratory cell and molecular biology. 44(2)
Although individuals exposed to cigarette smoke are more susceptible to respiratory infection, the effects of cigarette smoke on pulmonary defense are incompletely understood. Based on the observation that interactions between bacteria and host cells
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0d51191452e0de839da93b75da1150a7
https://pubmed.ncbi.nlm.nih.gov/20348206
https://pubmed.ncbi.nlm.nih.gov/20348206
Autor:
Lori J. Manzel, Paul B. McCray, Dwight C. Look, Christine L. Wohlford-Lenane, Brie N Nardy, Audra A Winder, Todd E. Scheetz
Publikováno v:
Respiratory Research, Vol 10, Iss 1, p 96 (2009)
Respiratory Research
Respiratory Research
Background The recognition of microbial molecular patterns via Toll-like receptors (TLRs) is critical for mucosal defenses. Methods Using well-differentiated primary cultures of human airway epithelia, we investigated the effects of exposure of the c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9b7b0cf86aea2a297f5e2d6322ad4ad4
http://respiratory-research.com/content/10/1/96
http://respiratory-research.com/content/10/1/96
Direct interaction between bacteria and epithelial cells may initiate or amplify the airway response through induction of epithelial defense gene expression by nuclear factor-kappaB (NF-kappaB). However, multiple signaling pathways modify NF-kappaB e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::461cff05bd213d834178dd2102f16349
https://europepmc.org/articles/PMC2633142/
https://europepmc.org/articles/PMC2633142/
Autor:
Timothy F. Murphy, Timothy D. Starner, Dwight C. Look, Lori J. Manzel, Cecilia L. Chin, Alicia L. Humlicek, Lei Shi, E. E. Lehman, G. M. Denning, S. Sethi
Publikováno v:
Journal of Investigative Medicine. 53:S361.6-362
Infection by nontypable Haemophilus influenzae in the airway causes inflammation, and newly isolated strains of these bacteria are associated with an increased risk of disease exacerbation in patients with chronic obstructive pulmonary disease (COPD)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::20d6e508f7d13844704a708e5a311481
https://doi.org/10.2310/6650.2005.00206.29
https://doi.org/10.2310/6650.2005.00206.29
Publikováno v:
Respiratory Research, Vol 11, Iss 1, p 64 (2010)
Respiratory Research
Respiratory Research
Background Although individuals exposed to cigarette smoke are more susceptible to respiratory infection, the effects of cigarette smoke on lung defense are incompletely understood. Because airway epithelial cell responses to type II interferon (IFN)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6b12979759f2d721940840a90bf5f864