Zobrazeno 1 - 10
of 50
pro vyhledávání: '"Lolitika Mandal"'
Autor:
Rakesh Mishra, Rohit Kunar, Lolitika Mandal, Debasmita Pankaj Alone, Shanti Chandrasekharan, Anand Krishna Tiwari, Madhu Gwaldas Tapadia, Ashim Mukherjee, Jagat Kumar Roy
Publikováno v:
G3: Genes, Genomes, Genetics, Vol 10, Iss 8, Pp 2601-2618 (2020)
The use of transposons to create mutations has been the cornerstone of Drosophila genetics in the past few decades. Second-site mutations caused by transpositions are often devoid of transposons and thereby affect subsequent analyses. In a P-element
Externí odkaz:
https://doaj.org/article/4957e7a258df44c09ddd5ee9e4ba5f39
Publikováno v:
Bio-Protocol, Vol 12, Iss 2 (2022)
The Drosophila larval haematopoietic organ or lymph gland consists of multiple cell types arranged in zones. The smallest stem cell compartment consists of 40-45 cells that constitute the haematopoietic niche. In order to analyse the haematopoietic n
Externí odkaz:
https://doaj.org/article/412da4e284174502b1bd3ada86238d23
Publikováno v:
PLoS Genetics, Vol 17, Iss 8, p e1009709 (2021)
Drosophila larval hematopoiesis occurs in a specialized multi-lobed organ called the lymph gland. Extensive characterization of the organ has provided mechanistic insights into events related to developmental hematopoiesis. Spanning from the thoracic
Externí odkaz:
https://doaj.org/article/bd23c4eeb9cb466cb7a5b711417014ad
Publikováno v:
eLife, Vol 10 (2021)
Immune challenges demand the gearing up of basal hematopoiesis to combat infection. Little is known about how during development, this switch is achieved to take care of the insult. Here, we show that the hematopoietic niche of the larval lymph gland
Externí odkaz:
https://doaj.org/article/3c205440e7da4803bb434c631083fa62
Publikováno v:
eLife, Vol 9 (2020)
Cell-intrinsic and extrinsic signals regulate the state and fate of stem and progenitor cells. Recent advances in metabolomics illustrate that various metabolic pathways are also important in regulating stem cell fate. However, our understanding of t
Externí odkaz:
https://doaj.org/article/50f5c8aace014b13a470292d6d0e7228
Publikováno v:
eLife, Vol 8 (2019)
Externí odkaz:
https://doaj.org/article/cb3670dcba2a4b2fa872a296973591f8
Publikováno v:
Wellcome Open Research, Vol 3 (2018)
Drosophila and mammalian hematopoiesis share several similarities that range from primitive and definitive phases of hematopoiesis to the battery of transcription factors and signaling molecules that execute this process. The similarities in blood ce
Externí odkaz:
https://doaj.org/article/52248ab4fbce463c981460fd8d6857ab
Publikováno v:
Wellcome Open Research, Vol 3 (2018)
Drosophila and mammalian hematopoiesis share several similarities that ranges from phases to the battery of transcription factors and signaling molecules that execute this process. These resounding similarities along with the rich genetic tools avail
Externí odkaz:
https://doaj.org/article/b15ed953bd6d4d31be30218a60b78105
Publikováno v:
eLife, Vol 5 (2016)
Drosophila hematopoiesis bears striking resemblance with that of vertebrates, both in the context of distinct phases and the signaling molecules. Even though, there has been no evidence of Hematopoietic stem cells (HSCs) in Drosophila, the larval lym
Externí odkaz:
https://doaj.org/article/d1217983dfb44336aa4308efd16a3e64
Autor:
Madhu G. Tapadia, Debasmita Pankaj Alone, Rohit Kunar, Rakesh Mishra, Ashim Mukherjee, Shanti Chandrasekharan, Jagat Kumar Roy, Anand K. Tiwari, Lolitika Mandal
Publikováno v:
G3: Genes, Genomes, Genetics, Vol 10, Iss 8, Pp 2601-2618 (2020)
G3: Genes|Genomes|Genetics
G3: Genes|Genomes|Genetics
The use of transposons to create mutations has been the cornerstone of Drosophila genetics in the past few decades. Second-site mutations caused by transpositions are often devoid of transposons and thereby affect subsequent analyses. In a P-element
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::14fe0722e25bab5b18b058e2e8318b14
http://g3journal.org/lookup/doi/10.1534/g3.120.401501
http://g3journal.org/lookup/doi/10.1534/g3.120.401501