Zobrazeno 1 - 5
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pro vyhledávání: '"Logan B. Smith"'
Publikováno v:
Journal of Animal Science. 84:2942-2949
Normal fibroblast growth factor receptor 3 (FGFR3) acts as a negative bone growth regulator by restricting chondrocyte proliferation and endochondral bone elongation. In sheep, a heritable mutation that inactivates FGFR3 produces skeletal overgrowth
Autor:
Caroline Anderson, Teresa J. Wright, Logan B. Smith, Michael C. Leo, Lisa Wood, Kristianna B. Weymann
Publikováno v:
Brain, behavior, and immunity. 38
Cytotoxic chemotherapeutic agents often induce a cluster of cancer treatment related symptoms (CTRS). The purpose of this study was to develop a mouse model of CTRS to examine the role of IL-1β and TNF-α signaling in the genesis of these symptoms.
Autor:
Lisa Wood, Logan B. Smith, John B. Wong, Cheleste M. Thorpe, Dakshina M. Jandhyala, Bruce E. Magun, Kirsten Kelley-Howard, Thomas Engstrom, Eli A. Magun
Publikováno v:
Cancer Biology & Therapy
The adverse side effects of doxorubicin, including cardiotoxicity and cancer treatment-related fatigue, have been associated with inflammatory cytokines, many of which are regulated by mitogen-activated protein kinases (MAPKs). ZAK is an upstream kin
Publikováno v:
Journal of Animal Science and Biotechnology
Journal of Animal Science and Biotechnology, Vol 3, Iss 1, p 39 (2012)
Smith, Logan B; Belanger, Janelle M; & Oberbauer, Anita M. (2012). Fibroblast growth factor receptor 3 effects on proliferation and telomerase activity in sheep growth plate chondrocytes. Journal of Animal Science and Biotechnology, 3(1), 39. doi: http://dx.doi.org/10.1186/2049-1891-3-39. Retrieved from: http://www.escholarship.org/uc/item/91x6p0t1
Journal of Animal Science and Biotechnology, Vol 3, Iss 1, p 39 (2012)
Smith, Logan B; Belanger, Janelle M; & Oberbauer, Anita M. (2012). Fibroblast growth factor receptor 3 effects on proliferation and telomerase activity in sheep growth plate chondrocytes. Journal of Animal Science and Biotechnology, 3(1), 39. doi: http://dx.doi.org/10.1186/2049-1891-3-39. Retrieved from: http://www.escholarship.org/uc/item/91x6p0t1
Background Fibroblast growth factor receptor 3 (FGFR3) inhibits growth-plate chondrocyte proliferation and limits bone elongation. Gain-of-function FGFR3 mutations cause dwarfism, reduced telomerase activity and shorter telomeres in growth plate chon